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Journal Abstract Search

184 related items for PubMed ID: 1310762

  • 1. Functions of the sequences at the ends of the inverted repeats of pseudorabies virus.
    Rall GF, Kupershmidt S, Sugg N, Veach RA, Ben-Porat T.
    J Virol; 1992 Mar; 66(3):1506-19. PubMed ID: 1310762
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  • 2. Cleavage of concatemeric DNA at the internal junction of "translocation" mutants of pseudorabies virus and inversion of their L component appear to be linked.
    Kupershmidt S, Rall GF, Lu ZQ, Ben-Porat T.
    Virology; 1992 Mar; 187(1):223-32. PubMed ID: 1310557
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  • 3. Low-level inversion of the L component of pseudorabies virus is not dependent on sequence homology.
    Rall GF, Kupershmidt S, Lu XQ, Mettenleiter TC, Ben-Porat T.
    J Virol; 1991 Dec; 65(12):7016-9. PubMed ID: 1658393
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  • 4. Nucleotide sequences at recombinational junctions present in pseudorabies virus variants with an invertible L component.
    Lu ZQ, DeMarchi JM, Harper L, Rall GF, Ben-Porat T.
    J Virol; 1989 Jun; 63(6):2690-8. PubMed ID: 2542582
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  • 7. Equalization of the inverted repeat sequences of the pseudorabies virus genome by intermolecular recombination.
    Ben-Porat T, Deatly A, Veach RA, Blankenship ML.
    Virology; 1984 Jan 30; 132(2):303-14. PubMed ID: 6322414
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  • 8. Analysis of the equalization of inverted repeats and neurovirulence using a pseudorabies virus mutant strain altered at the Ul/Ir junction.
    Boldogköi Z, Braun A, Medveczky I, Glávits R, Gyúró B, Fodor I.
    Virus Genes; 1998 Jan 30; 17(1):89-98. PubMed ID: 9778792
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  • 9. Analysis of an origin of DNA replication located at the L terminus of the genome of pseudorabies virus.
    Kupershmidt S, DeMarchi JM, Lu ZQ, Ben-Porat T.
    J Virol; 1991 Nov 30; 65(11):6283-91. PubMed ID: 1656095
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  • 10. Site-specific inversion sequence of the herpes simplex virus genome: domain and structural features.
    Mocarski ES, Roizman B.
    Proc Natl Acad Sci U S A; 1981 Nov 30; 78(11):7047-51. PubMed ID: 6273905
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  • 11. Vaccine properties of pseudorabies virus strain 783 are not affected by a deletion of 71 base pairs in the promoter/enhancer region of the viral immediate early gene.
    Glazenburg KL, Elgersma-Hooisma M, Briaire J, Voermans J, Kimman TG, Gielkens AL, Moormann RJ.
    Vaccine; 1994 Sep 30; 12(12):1097-100. PubMed ID: 7998419
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  • 15. The structure of the pseudorabies virus genome at the end of the inverted repeat sequences proximal to the junction with the short unique region.
    Zhang G, Leader DP.
    J Gen Virol; 1990 Oct 30; 71 ( Pt 10)():2433-41. PubMed ID: 2172457
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  • 16. Requirement for double-strand breaks but not for specific DNA sequences in herpes simplex virus type 1 genome isomerization events.
    Sarisky RT, Weber PC.
    J Virol; 1994 Jan 30; 68(1):34-47. PubMed ID: 8254746
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  • 17. Characterization of the terminal inverted repeats and their neighboring tandem repeats in the Chlorella CVK1 virus genome.
    Yamada T, Higashiyama T.
    Mol Gen Genet; 1993 Dec 30; 241(5-6):554-63. PubMed ID: 8264529
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  • 18. Terminally repeated sequences on a herpesvirus genome are deleted following circularization but are reconstituted by duplication during cleavage and packaging of concatemeric DNA.
    Nixon DE, McVoy MA.
    J Virol; 2002 Feb 30; 76(4):2009-13. PubMed ID: 11799198
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  • 19. The terminal a sequence of the herpes simplex virus genome contains the promoter of a gene located in the repeat sequences of the L component.
    Chou J, Roizman B.
    J Virol; 1986 Feb 30; 57(2):629-37. PubMed ID: 3003394
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  • 20. Identification of the site of recombination in the generation of the genome of DI particles of equine herpesvirus type 1.
    Yalamanchili RR, Raengsakulrach B, Baumann RP, O'Callaghan DJ.
    Virology; 1990 Apr 30; 175(2):448-55. PubMed ID: 2158182
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