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92 related items for PubMed ID: 1338278

  • 1. The effect of n-6 fatty acids on normal and v-Ki ras transformed NIH-3T3 cells.
    Cantrill RC, Ells GW, de Antueno RJ, Elliot M, Raha SK, Horrobin DF.
    Anticancer Res; 1992; 12(6B):2197-201. PubMed ID: 1338278
    [Abstract] [Full Text] [Related]

  • 2. Metabolism of radiolabelled 18:2n-6 and 18:3n-6 by NIH-3T3 cells and the DT subclone.
    De Antueno RJ, Cantrill RC, Ells GW, Elliot M, Huang YS, Horrobin DF.
    Anticancer Res; 1993; 13(4):973-7. PubMed ID: 8394680
    [Abstract] [Full Text] [Related]

  • 3. Effect of omega 3 and omega 6 fatty acids on transformation of cultured cells by irradiation and transfection.
    Takahashi M, Przetakiewicz M, Ong A, Borek C, Lowenstein JM.
    Cancer Res; 1992 Jan 01; 52(1):154-62. PubMed ID: 1530767
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  • 4. Thy-1 as a negative growth regulator in ras-transformed mouse fibroblasts.
    Sugimoto Y, Ikawa Y, Nakauchi H.
    Cancer Res; 1991 Jan 01; 51(1):99-104. PubMed ID: 1670996
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  • 5. Resistance to apoptosis induced by alkylating agents in v-Ha-ras-transformed cells due to defect in p53 function.
    Kuo ML, Chou YW, Chau YP, Huang TS.
    Mol Carcinog; 1997 Apr 01; 18(4):221-31. PubMed ID: 9142217
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  • 6. Transfection of EK-3, a subline of NIH 3T3, with the oncogene Ha-ras does not abolish its anchorage dependence.
    Katz E, Samid D.
    Eur J Cell Biol; 1989 Aug 01; 49(2):221-4. PubMed ID: 2673787
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  • 7. Decreased retinoylation in NIH 3T3 cells transformed with activated Ha-ras.
    Takahashi N, De Luca LM, Breitman TR.
    Biochem Biophys Res Commun; 1997 Oct 09; 239(1):80-4. PubMed ID: 9345273
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  • 8. Recessive (mediator-) revertants from c-H-ras oncogene-transformed NIH 3T3 cells: tumorigenicity in nude mice and transient anchorage and serum independence of the recovered tumor cells in culture.
    Omata-Yamada T, Yamada H, Lengyel P.
    J Cell Physiol; 1991 Nov 09; 149(2):214-21. PubMed ID: 1748716
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  • 12. Comparison of the metabolism of alpha-linolenic acid and its delta 6 desaturation product, stearidonic acid, in cultured NIH-3T3 cells.
    Cantrill RC, Huang YS, Ells GW, Horrobin DF.
    Lipids; 1993 Mar 09; 28(3):163-6. PubMed ID: 8096621
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  • 13. Increased cell-substrate adhesion accompanies conditional reversion to the normal phenotype in ras-oncogene-transformed NIH-3T3 cells.
    Shumaker DK, Sklar MD, Prochownik EV, Varani J.
    Exp Cell Res; 1994 Oct 09; 214(2):440-6. PubMed ID: 7925639
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  • 14. Transformation of NIH/3T3 to anchorage independence by H-ras is accompanied by loss of suppressor activity.
    Tolsma SS, Cohen JD, Ehrlich LS, Bouck NP.
    Exp Cell Res; 1993 Apr 09; 205(2):232-9. PubMed ID: 8482334
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  • 15. Cytoskeletal reorganization in NIH 3T3 fibroblasts expressing the ras oncogene.
    Dartsch PC, Ritter M, Häussinger D, Lang F.
    Eur J Cell Biol; 1994 Apr 09; 63(2):316-25. PubMed ID: 8082656
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  • 17. Retinoic acid downregulates growth, fibronectin and RAR alpha in 3T3 cells: Ha-ras blocks this response and RA metabolism.
    De Luca LM, Scita G, Takatsuka J.
    J Cell Physiol; 1997 Nov 09; 173(2):297-300. PubMed ID: 9365540
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  • 18. Effect of diets enriched in Delta6 desaturated fatty acids (18:3n-6 and 18:4n-3), on growth, fatty acid composition and highly unsaturated fatty acid synthesis in two populations of Arctic charr (Salvelinus alpinus L.).
    Tocher DR, Dick JR, MacGlaughlin P, Bell JG.
    Comp Biochem Physiol B Biochem Mol Biol; 2006 Jun 09; 144(2):245-53. PubMed ID: 16630735
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  • 19. Ras-interacting domain of Ral GDP dissociation stimulator like (RGL) reverses v-Ras-induced transformation and Raf-1 activation in NIH3T3 cells.
    Okazaki M, Kishida S, Murai H, Hinoi T, Kikuchi A.
    Cancer Res; 1996 May 15; 56(10):2387-92. PubMed ID: 8625316
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  • 20. SCH 51344 inhibits ras transformation by a novel mechanism.
    Kumar CC, Prorock-Rogers C, Kelly J, Dong Z, Lin JJ, Armstrong L, Kung HF, Weber MJ, Afonso A.
    Cancer Res; 1995 Nov 01; 55(21):5106-17. PubMed ID: 7585559
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