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274 related items for PubMed ID: 1347914

  • 1. Modulation of normal erythroid differentiation by the endogenous thyroid hormone and retinoic acid receptors: a possible target for v-erbA oncogene action.
    Schroeder C, Gibson L, Zenke M, Beug H.
    Oncogene; 1992 Feb; 7(2):217-27. PubMed ID: 1347914
    [Abstract] [Full Text] [Related]

  • 2. The v-erbA oncogene requires cooperation with tyrosine kinases to arrest erythroid differentiation induced by ligand-activated endogenous c-erbA and retinoic acid receptor.
    Schroeder C, Gibson L, Beug H.
    Oncogene; 1992 Feb; 7(2):203-16. PubMed ID: 1347913
    [Abstract] [Full Text] [Related]

  • 3. The thyroid hormone receptor functions as a ligand-operated developmental switch between proliferation and differentiation of erythroid progenitors.
    Bauer A, Mikulits W, Lagger G, Stengl G, Brosch G, Beug H.
    EMBO J; 1998 Aug 03; 17(15):4291-303. PubMed ID: 9687498
    [Abstract] [Full Text] [Related]

  • 4. Mechanism of transformation by v-ErbA: substitution for steroid hormone receptor function in self renewal induction.
    Bauer A, Ulrich E, Andersson M, Beug H, von Lindern M.
    Oncogene; 1997 Aug 07; 15(6):701-15. PubMed ID: 9264411
    [Abstract] [Full Text] [Related]

  • 5. c-erbA alpha/T3R and RARs control commitment of hematopoietic self-renewing progenitor cells to apoptosis or differentiation and are antagonized by the v-erbA oncogene.
    Gandrillon O, Ferrand N, Michaille JJ, Roze L, Zile MH, Samarut J.
    Oncogene; 1994 Mar 07; 9(3):749-58. PubMed ID: 7906409
    [Abstract] [Full Text] [Related]

  • 6. V-erbA requires auxiliary proteins for dominant negative activity.
    Hermann T, Hoffmann B, Piedrafita FJ, Zhang XK, Pfahl M.
    Oncogene; 1993 Jan 07; 8(1):55-65. PubMed ID: 8093812
    [Abstract] [Full Text] [Related]

  • 7. c-ErbA, but not v-ErbA, competes with a putative erythroid repressor for binding to the carbonic anhydrase II promoter.
    Rascle A, Ghysdael J, Samarut J.
    Oncogene; 1994 Oct 07; 9(10):2853-67. PubMed ID: 7916146
    [Abstract] [Full Text] [Related]

  • 8. The leukaemia oncogene v-erbA: a dominant negative version of ligand dependent transcription factors that regulates red cell differentiation?
    Ghysdael J, Beug H.
    Cancer Surv; 1992 Oct 07; 14():169-80. PubMed ID: 1358441
    [Abstract] [Full Text] [Related]

  • 9. The v-erb A oncogene causes repression of erythrocyte-specific genes and an immature, aberrant differentiation phenotype in normal erythroid progenitors.
    Schroeder C, Raynoschek C, Fuhrmann U, Damm K, Vennström B, Beug H.
    Oncogene; 1990 Oct 07; 5(10):1445-53. PubMed ID: 1979159
    [Abstract] [Full Text] [Related]

  • 10. v-erbA stimulates quail myoblast differentiation in a T3 independent, cell-specific manner.
    Cassar-Malek I, Marchal S, Altabef M, Wrutniak C, Samarut J, Cabello G.
    Oncogene; 1994 Aug 07; 9(8):2197-206. PubMed ID: 7913543
    [Abstract] [Full Text] [Related]

  • 11. Overexpression of c-erbA proto-oncogene enhances myogenic differentiation.
    Carnac G, Albagli-Curiel O, Desclozeaux M, Vandromme M, Glineur C, Bègue A, Laudet V, Bonnieu A.
    Oncogene; 1993 Nov 07; 8(11):3103-10. PubMed ID: 8414512
    [Abstract] [Full Text] [Related]

  • 12. c-erbA and v-erbA modulate growth and gene expression of a mouse glial precursor cell line.
    Iglesias T, Llanos S, López-Barahona M, Pérez-Aranda A, Rodríguez-Peña A, Bernal J, Höhne A, Seliger B, Muñoz A.
    Cell Growth Differ; 1994 Jul 07; 5(7):697-704. PubMed ID: 7947384
    [Abstract] [Full Text] [Related]

  • 13. v-erbA overexpression is required to extinguish c-erbA function in erythroid cell differentiation and regulation of the erbA target gene CAII.
    Disela C, Glineur C, Bugge T, Sap J, Stengl G, Dodgson J, Stunnenberg H, Beug H, Zenke M.
    Genes Dev; 1991 Nov 07; 5(11):2033-47. PubMed ID: 1682217
    [Abstract] [Full Text] [Related]

  • 14. Functional domains of the v-erbA protein necessary for oncogenesis are required for transcriptional activation in Saccharomyces cerevisiae.
    Smit-McBride Z, Privalsky ML.
    Oncogene; 1993 Jun 07; 8(6):1465-75. PubMed ID: 8099219
    [Abstract] [Full Text] [Related]

  • 15. V-erbA and c-erbA proteins enhance transcriptional activation by c-jun.
    Sharif M, Privalsky ML.
    Oncogene; 1992 May 07; 7(5):953-60. PubMed ID: 1349165
    [Abstract] [Full Text] [Related]

  • 16. v-erbA oncogene activation entails the loss of hormone-dependent regulator activity of c-erbA.
    Zenke M, Muñoz A, Sap J, Vennström B, Beug H.
    Cell; 1990 Jun 15; 61(6):1035-49. PubMed ID: 1972036
    [Abstract] [Full Text] [Related]

  • 17. A conserved C-terminal sequence that is deleted in v-ErbA is essential for the biological activities of c-ErbA (the thyroid hormone receptor).
    Saatcioglu F, Bartunek P, Deng T, Zenke M, Karin M.
    Mol Cell Biol; 1993 Jun 15; 13(6):3675-85. PubMed ID: 8098843
    [Abstract] [Full Text] [Related]

  • 18. Inhibition of thyroid hormone action by a non-hormone binding c-erbA protein generated by alternative mRNA splicing.
    Koenig RJ, Lazar MA, Hodin RA, Brent GA, Larsen PR, Chin WW, Moore DD.
    Nature; 1989 Feb 16; 337(6208):659-61. PubMed ID: 2537467
    [Abstract] [Full Text] [Related]

  • 19. Differential and tissue-specific regulation of the multiple rat c-erbA messenger RNA species by thyroid hormone.
    Hodin RA, Lazar MA, Chin WW.
    J Clin Invest; 1990 Jan 16; 85(1):101-5. PubMed ID: 2153150
    [Abstract] [Full Text] [Related]

  • 20. Retinoid X receptor and c-cerbA/thyroid hormone receptor regulate erythroid cell growth and differentiation.
    Bartůnĕk P, Zenke M.
    Mol Endocrinol; 1998 Sep 16; 12(9):1269-79. PubMed ID: 9731697
    [Abstract] [Full Text] [Related]

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