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192 related items for PubMed ID: 1354613

  • 1. Transcriptional repression of band 3 and CAII in v-erbA transformed erythroblasts accounts for an important part of the leukaemic phenotype.
    Fuerstenberg S, Leitner I, Schroeder C, Schwarz H, Vennström B, Beug H.
    EMBO J; 1992 Sep; 11(9):3355-65. PubMed ID: 1354613
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  • 2. v-erbA specifically suppresses transcription of the avian erythrocyte anion transporter (band 3) gene.
    Zenke M, Kahn P, Disela C, Vennström B, Leutz A, Keegan K, Hayman MJ, Choi HR, Yew N, Engel JD.
    Cell; 1988 Jan 15; 52(1):107-19. PubMed ID: 2830979
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  • 3. v-erbA overexpression is required to extinguish c-erbA function in erythroid cell differentiation and regulation of the erbA target gene CAII.
    Disela C, Glineur C, Bugge T, Sap J, Stengl G, Dodgson J, Stunnenberg H, Beug H, Zenke M.
    Genes Dev; 1991 Nov 15; 5(11):2033-47. PubMed ID: 1682217
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  • 5. Phosphorylation of the v-erbA protein is required for its function as an oncogene.
    Glineur C, Zenke M, Beug H, Ghysdael J.
    Genes Dev; 1990 Oct 15; 4(10):1663-76. PubMed ID: 1979040
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  • 6. The v-erb A oncogene causes repression of erythrocyte-specific genes and an immature, aberrant differentiation phenotype in normal erythroid progenitors.
    Schroeder C, Raynoschek C, Fuhrmann U, Damm K, Vennström B, Beug H.
    Oncogene; 1990 Oct 15; 5(10):1445-53. PubMed ID: 1979159
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  • 7. c-ErbA, but not v-ErbA, competes with a putative erythroid repressor for binding to the carbonic anhydrase II promoter.
    Rascle A, Ghysdael J, Samarut J.
    Oncogene; 1994 Oct 15; 9(10):2853-67. PubMed ID: 7916146
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  • 13. The v-ski oncogene cooperates with the v-sea oncogene in erythroid transformation by blocking erythroid differentiation.
    Larsen J, Beug H, Hayman MJ.
    Oncogene; 1992 Oct 15; 7(10):1903-11. PubMed ID: 1408132
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  • 14. The leukaemia oncogene v-erbA: a dominant negative version of ligand dependent transcription factors that regulates red cell differentiation?
    Ghysdael J, Beug H.
    Cancer Surv; 1992 Oct 15; 14():169-80. PubMed ID: 1358441
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  • 15. Mechanism of transformation by v-ErbA: substitution for steroid hormone receptor function in self renewal induction.
    Bauer A, Ulrich E, Andersson M, Beug H, von Lindern M.
    Oncogene; 1997 Aug 07; 15(6):701-15. PubMed ID: 9264411
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  • 19. A single point mutation in erbA restores the erythroid transforming potential of a mutant avian erythroblastosis virus (AEV) defective in both erbA and erbB oncogenes.
    Damm K, Beug H, Graf T, Vennström B.
    EMBO J; 1987 Feb 07; 6(2):375-82. PubMed ID: 2884103
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