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158 related items for PubMed ID: 1357585

  • 1. 5-(N-ethyl-N-isopropyl)amiloride and mild acidosis protect cultured cerebellar granule cells against glutamate-induced delayed neuronal death.
    Andreeva N, Khodorov B, Stelmashook E, Sokolova S, Cragoe E, Victorov I.
    Neuroscience; 1992 Jul; 49(1):175-81. PubMed ID: 1357585
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  • 2. Inhibition of Na+/Ca2+ exchange enhances delayed neuronal death elicited by glutamate in cerebellar granule cell cultures.
    Andreeva N, Khodorov B, Stelmashook E, Cragoe E, Victorov I.
    Brain Res; 1991 May 10; 548(1-2):322-5. PubMed ID: 1678303
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  • 3. Acidosis and 5-(N-ethyl-N-isopropyl)amiloride (EIPA) Attenuate Zinc/Kainate Toxicity in Cultured Cerebellar Granule Neurons.
    Stelmashook EV, Novikova SV, Amelkina GA, Ivashkin EG, Genrikhs EE, Khaspekov LG, Isaev NK.
    Biochemistry (Mosc); 2015 Aug 10; 80(8):1065-72. PubMed ID: 26547075
    [Abstract] [Full Text] [Related]

  • 4. Amiloride blocks glutamate-operated cationic channels and protects neurons in culture from glutamate-induced death.
    Manev H, Bertolino M, DeErausquin G.
    Neuropharmacology; 1990 Dec 10; 29(12):1103-10. PubMed ID: 1963475
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  • 5. N-methyl-D-aspartate exposure blocks glutamate toxicity in cultured cerebellar granule cells.
    Chuang DM, Gao XM, Paul SM.
    Mol Pharmacol; 1992 Aug 10; 42(2):210-6. PubMed ID: 1355259
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  • 6. Noise and single channels activated by excitatory amino acids in rat cerebellar granule neurones.
    Cull-Candy SG, Howe JR, Ogden DC.
    J Physiol; 1988 Jun 10; 400():189-222. PubMed ID: 2458453
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  • 7. Excitotoxic death induced by released glutamate in depolarized primary cultures of mouse cerebellar granule cells is dependent on GABAA receptors and niflumic acid-sensitive chloride channels.
    Babot Z, Cristòfol R, Suñol C.
    Eur J Neurosci; 2005 Jan 10; 21(1):103-12. PubMed ID: 15654847
    [Abstract] [Full Text] [Related]

  • 8. Intracellular acidification by inhibition of the Na+/H+-exchanger leads to caspase-independent death of cerebellar granule neurons resembling paraptosis.
    Schneider D, Gerhardt E, Bock J, Müller MM, Wolburg H, Lang F, Schulz JB.
    Cell Death Differ; 2004 Jul 10; 11(7):760-70. PubMed ID: 15017383
    [Abstract] [Full Text] [Related]

  • 9. Failure of glycine site NMDA receptor antagonists to protect against L-2-chloropropionic acid-induced neurotoxicity highlights the uniqueness of cerebellar NMDA receptors.
    Widdowson PS, Gyte AJ, Upton R, Wyatt I.
    Brain Res; 1996 Nov 04; 738(2):236-42. PubMed ID: 8955518
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  • 11. Glutamate receptor subtypes in cultured cerebellar neurons: modulation of glutamate and gamma-aminobutyric acid release.
    Gallo V, Suergiu R, Giovannini C, Levi G.
    J Neurochem; 1987 Dec 04; 49(6):1801-9. PubMed ID: 2890714
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  • 12. Glutamate-mediated neuroprotection against N-methyl-D-aspartate toxicity: a role for metabotropic glutamate receptors.
    Adamchik Y, Baskys A.
    Neuroscience; 2000 Dec 04; 99(4):731-6. PubMed ID: 10974436
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  • 16. L-2-chloropropionic acid inhibits glutamate and aspartate release from rat cerebellar slices but does not activate cerebellar NMDA receptors: implications for L-2-chloropropionic acid-induced neurotoxicity.
    Widdowson PS, Briggs I, BoSmith RE, Sturgess NC, Rosbottom A, Smith JC, Wyatt I.
    Neurotoxicology; 1997 Dec 04; 18(1):169-77. PubMed ID: 9215999
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  • 17. Chronic mild acidosis specifically reduces functional expression of N-methyl-D-aspartate receptors and increases long-term survival in primary cultures of cerebellar granule cells.
    Leahy JC, Chen Q, Vallano ML.
    Neuroscience; 1994 Nov 04; 63(2):457-70. PubMed ID: 7891858
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  • 18. Intracellular sodium concentration in cultured cerebellar granule cells challenged with glutamate.
    Kiedrowski L, Wroblewski JT, Costa E.
    Mol Pharmacol; 1994 May 04; 45(5):1050-4. PubMed ID: 7910657
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  • 19. Neurotoxicity of polyamines and pharmacological neuroprotection in cultures of rat cerebellar granule cells.
    Sparapani M, Dall'Olio R, Gandolfi O, Ciani E, Contestabile A.
    Exp Neurol; 1997 Nov 04; 148(1):157-66. PubMed ID: 9398458
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  • 20. Rat cerebellar granule cells are protected from glutamate-induced excitotoxicity by S-nitrosoglutathione but not glutathione.
    Li CY, Chin TY, Chueh SH.
    Am J Physiol Cell Physiol; 2004 Apr 04; 286(4):C893-904. PubMed ID: 15001426
    [Abstract] [Full Text] [Related]

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