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101 related items for PubMed ID: 1408132

  • 1. The v-ski oncogene cooperates with the v-sea oncogene in erythroid transformation by blocking erythroid differentiation.
    Larsen J, Beug H, Hayman MJ.
    Oncogene; 1992 Oct; 7(10):1903-11. PubMed ID: 1408132
    [Abstract] [Full Text] [Related]

  • 2. The v-erb A oncogene causes repression of erythrocyte-specific genes and an immature, aberrant differentiation phenotype in normal erythroid progenitors.
    Schroeder C, Raynoschek C, Fuhrmann U, Damm K, Vennström B, Beug H.
    Oncogene; 1990 Oct; 5(10):1445-53. PubMed ID: 1979159
    [Abstract] [Full Text] [Related]

  • 3. Transformation of chicken bone marrow cells by the v-ski oncogene.
    Larsen J, Meyer S, Steinlein P, Beug H, Hayman MJ.
    Oncogene; 1993 Dec; 8(12):3221-8. PubMed ID: 7504231
    [Abstract] [Full Text] [Related]

  • 4. The v-erbA oncogene requires cooperation with tyrosine kinases to arrest erythroid differentiation induced by ligand-activated endogenous c-erbA and retinoic acid receptor.
    Schroeder C, Gibson L, Beug H.
    Oncogene; 1992 Feb; 7(2):203-16. PubMed ID: 1347913
    [Abstract] [Full Text] [Related]

  • 5. The nuclear oncogenes v-erbA and v-ets cooperate in the induction of avian erythroleukemia.
    Metz T, Graf T.
    Oncogene; 1992 Mar; 7(3):597-605. PubMed ID: 1347919
    [Abstract] [Full Text] [Related]

  • 6. Defective v-erbB genes can be complemented by v-erbA in erythroblast and fibroblast transformation.
    Jansson M, Beug H, Gray C, Graf T, Vennström B.
    Oncogene; 1987 May; 1(2):167-73. PubMed ID: 2830578
    [Abstract] [Full Text] [Related]

  • 7. v-erbA cooperates with bFGF in neuroretina cell transformation.
    Garrido C, Li RP, Samarut J, Gospodarowicz D, Saule S.
    Virology; 1993 Feb; 192(2):578-86. PubMed ID: 7678474
    [Abstract] [Full Text] [Related]

  • 8. Modulation of normal erythroid differentiation by the endogenous thyroid hormone and retinoic acid receptors: a possible target for v-erbA oncogene action.
    Schroeder C, Gibson L, Zenke M, Beug H.
    Oncogene; 1992 Feb; 7(2):217-27. PubMed ID: 1347914
    [Abstract] [Full Text] [Related]

  • 9. Mutational analysis of the role of the carboxy-terminal region of the v-erbB protein in erythroid cell transformation.
    Lee EB, Beug H, Hayman MJ.
    Oncogene; 1993 May; 8(5):1317-27. PubMed ID: 8097581
    [Abstract] [Full Text] [Related]

  • 10. Mechanism of transformation by v-ErbA: substitution for steroid hormone receptor function in self renewal induction.
    Bauer A, Ulrich E, Andersson M, Beug H, von Lindern M.
    Oncogene; 1997 Aug 07; 15(6):701-15. PubMed ID: 9264411
    [Abstract] [Full Text] [Related]

  • 11. Induction of differentiation of avian erythroblastosis virus-transformed erythroblasts by the protein kinase inhibitor H7: analysis of the transcription factor EF1.
    Nicolas RH, Partington G, Major GN, Smith B, Carne AF, Huskisson N, Goodwin G.
    Cell Growth Differ; 1991 Mar 07; 2(3):129-35. PubMed ID: 1676294
    [Abstract] [Full Text] [Related]

  • 12. Expression of v-rel in a replication competent virus: transformation and biochemical characterization.
    Morrison LE, Boehmelt G, Beug H, Enrietto PJ.
    Oncogene; 1991 Sep 07; 6(9):1657-66. PubMed ID: 1656369
    [Abstract] [Full Text] [Related]

  • 13. Requirement for the C-terminal domain of the v-erbA oncogene protein for biological function and transcriptional repression.
    Forrest D, Muñoz A, Raynoschek C, Vennström B, Beug H.
    Oncogene; 1990 Mar 07; 5(3):309-16. PubMed ID: 1969136
    [Abstract] [Full Text] [Related]

  • 14. The leukaemia oncogene v-erbA: a dominant negative version of ligand dependent transcription factors that regulates red cell differentiation?
    Ghysdael J, Beug H.
    Cancer Surv; 1992 Mar 07; 14():169-80. PubMed ID: 1358441
    [Abstract] [Full Text] [Related]

  • 15. Abnormal glycosylation of the env-sea oncogene product inhibits its proteolytic cleavage and blocks its transforming ability.
    Knight J, Beug H, Marshall J, Hayman MJ.
    Oncogene; 1988 Apr 07; 2(4):317-26. PubMed ID: 2834683
    [Abstract] [Full Text] [Related]

  • 16. In vitro transformation of murine pro-B and pre-B cells by v-mpl, a truncated form of a cytokine receptor.
    Fichelson S, Vigon I, Dusanter I, Charon M, Velu T, Baillou C, Gisselbrecht S, Lemoine FM.
    J Immunol; 1995 Feb 15; 154(4):1577-86. PubMed ID: 7836743
    [Abstract] [Full Text] [Related]

  • 17. Tumorigenic effects mediated in the avian embryo by one or more oncogenes associated with v-myc.
    al Moustafa AE, Quatannens B, Dieterlen-Lièvre F, Saule S.
    Oncogene; 1992 Aug 15; 7(8):1667-70. PubMed ID: 1630827
    [Abstract] [Full Text] [Related]

  • 18. Phosphorylation of the v-erbA protein is required for its function as an oncogene.
    Glineur C, Zenke M, Beug H, Ghysdael J.
    Genes Dev; 1990 Oct 15; 4(10):1663-76. PubMed ID: 1979040
    [Abstract] [Full Text] [Related]

  • 19. Transcriptional repression of band 3 and CAII in v-erbA transformed erythroblasts accounts for an important part of the leukaemic phenotype.
    Fuerstenberg S, Leitner I, Schroeder C, Schwarz H, Vennström B, Beug H.
    EMBO J; 1992 Sep 15; 11(9):3355-65. PubMed ID: 1354613
    [Abstract] [Full Text] [Related]

  • 20. Raf revertant cells resist transformation by non-nuclear oncogenes and are deficient in the induction of early response genes by TPA and serum.
    Kolch W, Heidecker G, Troppmair J, Yanagihara K, Bassin RH, Rapp UR.
    Oncogene; 1993 Feb 15; 8(2):361-70. PubMed ID: 8426742
    [Abstract] [Full Text] [Related]

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