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Journal Abstract Search
115 related items for PubMed ID: 1427561
1. Role of asynchronous activation of the ventricular fibres by an ectopic pacemaker in the accidents, especially fibrillation, caused by Ic antiarrhythmic drugs. Timour Q, Aupetit JF, Loufoua-Moundanga J, Kioueh I, Omar S, Chevrel G, Faucon G. Fundam Clin Pharmacol; 1992; 6(4-5):159-67. PubMed ID: 1427561 [Abstract] [Full Text] [Related]
3. Disappearance with ischaemic depolarization of the antifibrillatory activity in a sodium channel blocker and appearance in calcium channel blocker. Bui-Xuan B, Aupetit JF, Freysz M, Loufoua-Moundanga J, Faucon G, Timour Q. Arch Int Pharmacodyn Ther; 1996; 331(3):246-62. PubMed ID: 9124997 [Abstract] [Full Text] [Related]
4. Opposite change with ischaemia in the antifibrillatory effects of class I and class IV antiarrhythmic drugs resulting from the alteration in ion transmembrane exchanges related to depolarization. Aupetit JF, Bui-Xuan B, Kioueh I, Loufoua J, Frassati D, Timour Q. Can J Physiol Pharmacol; 2000 Mar; 78(3):208-16. PubMed ID: 10721812 [Abstract] [Full Text] [Related]
9. Prevention by calcium antagonists of profibrillatory effects of class I antiarrhythmic drugs in acute myocardial ischemia: study in pig heart in situ. Bui-Xuan B, Aupetit JF, Freysz M, Faucon G, Timour Q. Pharmacotherapy; 1997 Mar; 17(4):737-45. PubMed ID: 9250551 [Abstract] [Full Text] [Related]
19. Characterization of an anesthetized dog model of transient cardiac ischemia and rapid pacing: a pilot study for preclinical assessment of the potential for proarrhythmic risk of novel drug candidates. Damiano BP, van der Linde HJ, Van Deuren B, Somers Y, Lubomirski M, Teisman A, Gallacher DJ. J Pharmacol Toxicol Methods; 2015 Mar; 72():72-84. PubMed ID: 25450838 [Abstract] [Full Text] [Related]