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Journal Abstract Search

227 related items for PubMed ID: 14500338

  • 21. Host bone-marrow cells are a source of donor intimal smooth- muscle-like cells in murine aortic transplant arteriopathy.
    Shimizu K, Sugiyama S, Aikawa M, Fukumoto Y, Rabkin E, Libby P, Mitchell RN.
    Nat Med; 2001 Jun; 7(6):738-41. PubMed ID: 11385513
    [Abstract] [Full Text] [Related]

  • 22. Estrogen-mediated, endothelial nitric oxide synthase-dependent mobilization of bone marrow-derived endothelial progenitor cells contributes to reendothelialization after arterial injury.
    Iwakura A, Luedemann C, Shastry S, Hanley A, Kearney M, Aikawa R, Isner JM, Asahara T, Losordo DW.
    Circulation; 2003 Dec 23; 108(25):3115-21. PubMed ID: 14676142
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  • 23. [Detection of bone marrow-derived cells in neointimal thickening in the rat carotid artery by nested polymerase chain reaction].
    Il'inskaia OP, Antropova IuG, Kalinina NI, Mishina VA, Radiukhina NV, Tararak EM.
    Ontogenez; 2008 Dec 23; 39(4):282-8. PubMed ID: 18792640
    [Abstract] [Full Text] [Related]

  • 24. Quantitative enumeration of vascular smooth muscle cells and endothelial cells derived from bone marrow precursors in experimental choroidal neovascularization.
    Espinosa-Heidmann DG, Reinoso MA, Pina Y, Csaky KG, Caicedo A, Cousins SW.
    Exp Eye Res; 2005 Mar 23; 80(3):369-78. PubMed ID: 15721619
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  • 25. Plasminogen activator inhibitor-1 from bone marrow-derived cells suppresses neointimal formation after vascular injury in mice.
    Schäfer K, Schroeter MR, Dellas C, Puls M, Nitsche M, Weiss E, Hasenfuss G, Konstantinides SV.
    Arterioscler Thromb Vasc Biol; 2006 Jun 23; 26(6):1254-9. PubMed ID: 16514083
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  • 26. Circulating progenitor cells contribute to neointimal formation in nonirradiated chimeric mice.
    Tanaka K, Sata M, Natori T, Kim-Kaneyama JR, Nose K, Shibanuma M, Hirata Y, Nagai R.
    FASEB J; 2008 Feb 23; 22(2):428-36. PubMed ID: 17848623
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  • 27. Targeted disruption of TGF-beta-Smad3 signaling leads to enhanced neointimal hyperplasia with diminished matrix deposition in response to vascular injury.
    Kobayashi K, Yokote K, Fujimoto M, Yamashita K, Sakamoto A, Kitahara M, Kawamura H, Maezawa Y, Asaumi S, Tokuhisa T, Mori S, Saito Y.
    Circ Res; 2005 Apr 29; 96(8):904-12. PubMed ID: 15790953
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  • 29. Role of bone marrow-derived cells in the genetic control of restenosis.
    Langwieser N, Schwarz JB, Reichenbächer C, Stemmer B, Massberg S, Langwieser NN, Zohlnhöfer D.
    Arterioscler Thromb Vasc Biol; 2009 Oct 29; 29(10):1551-7. PubMed ID: 19644054
    [Abstract] [Full Text] [Related]

  • 30. Effects of G-CSF on cardiac remodeling and arterial hyperplasia in rats.
    Li Y, Fukuda N, Yokoyama S, Kusumi Y, Hagikura K, Kawano T, Takayama T, Matsumoto T, Satomi A, Honye J, Mugishima H, Mitsumata M, Saito S.
    Eur J Pharmacol; 2006 Nov 07; 549(1-3):98-106. PubMed ID: 16979158
    [Abstract] [Full Text] [Related]

  • 31. Bone marrow-derived cells contribute to vascular inflammation but do not differentiate into smooth muscle cell lineages.
    Iwata H, Manabe I, Fujiu K, Yamamoto T, Takeda N, Eguchi K, Furuya A, Kuro-o M, Sata M, Nagai R.
    Circulation; 2010 Nov 16; 122(20):2048-57. PubMed ID: 21041690
    [Abstract] [Full Text] [Related]

  • 32. Granulocyte colony-stimulating factor (G-CSF) accelerates reendothelialization and reduces neointimal formation after vascular injury in mice.
    Yoshioka T, Takahashi M, Shiba Y, Suzuki C, Morimoto H, Izawa A, Ise H, Ikeda U.
    Cardiovasc Res; 2006 Apr 01; 70(1):61-9. PubMed ID: 16448633
    [Abstract] [Full Text] [Related]

  • 33. Bone marrow-derived CX3CR1 progenitors contribute to neointimal smooth muscle cells via fractalkine CX3CR1 interaction.
    Kumar AH, Metharom P, Schmeckpeper J, Weiss S, Martin K, Caplice NM.
    FASEB J; 2010 Jan 01; 24(1):81-92. PubMed ID: 19745110
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  • 36. Comparison of various bone marrow fractions in the ability to participate in vascular remodeling after mechanical injury.
    Sahara M, Sata M, Matsuzaki Y, Tanaka K, Morita T, Hirata Y, Okano H, Nagai R.
    Stem Cells; 2005 Aug 01; 23(7):874-8. PubMed ID: 15941860
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  • 38. Erythropoietin-mobilized endothelial progenitors enhance reendothelialization via Akt-endothelial nitric oxide synthase activation and prevent neointimal hyperplasia.
    Urao N, Okigaki M, Yamada H, Aadachi Y, Matsuno K, Matsui A, Matsunaga S, Tateishi K, Nomura T, Takahashi T, Tatsumi T, Matsubara H.
    Circ Res; 2006 Jun 09; 98(11):1405-13. PubMed ID: 16645141
    [Abstract] [Full Text] [Related]

  • 39. Adrenomedullin gene transfer induces neointimal apoptosis and inhibits neointimal hyperplasia in injured rat artery.
    Rauma-Pinola T, Pääkkö P, Ilves M, Serpi R, Romppanen H, Vuolteenaho O, Ruskoaho H, Hautala T.
    J Gene Med; 2006 Apr 09; 8(4):452-8. PubMed ID: 16389603
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  • 40. Crucial role of the CCL2/CCR2 axis in neointimal hyperplasia after arterial injury in hyperlipidemic mice involves early monocyte recruitment and CCL2 presentation on platelets.
    Schober A, Zernecke A, Liehn EA, von Hundelshausen P, Knarren S, Kuziel WA, Weber C.
    Circ Res; 2004 Nov 26; 95(11):1125-33. PubMed ID: 15528472
    [Abstract] [Full Text] [Related]

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