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188 related items for PubMed ID: 14514962

  • 1. Lack of expression of EGF and TGF-alpha in the fetal mouse alters formation of prostatic epithelial buds and influences the response to TCDD.
    Abbott BD, Lin TM, Rasmussen NT, Albrecht RM, Schmid JE, Peterson RE.
    Toxicol Sci; 2003 Dec; 76(2):427-36. PubMed ID: 14514962
    [Abstract] [Full Text] [Related]

  • 2. EGF and TGF-alpha expression influence the developmental toxicity of TCDD: dose response and AhR phenotype in EGF, TGF-alpha, and EGF + TGF-alpha knockout mice.
    Abbott BD, Buckalew AR, DeVito MJ, Ross D, Bryant PL, Schmid JE.
    Toxicol Sci; 2003 Jan; 71(1):84-95. PubMed ID: 12520078
    [Abstract] [Full Text] [Related]

  • 3. Region-specific inhibition of prostatic epithelial bud formation in the urogenital sinus of C57BL/6 mice exposed in utero to 2,3,7,8-tetrachlorodibenzo-p-dioxin.
    Lin TM, Rasmussen NT, Moore RW, Albrecht RM, Peterson RE.
    Toxicol Sci; 2003 Nov; 76(1):171-81. PubMed ID: 12944588
    [Abstract] [Full Text] [Related]

  • 4. Teratogenicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) in mice lacking the expression of EGF and/or TGF-alpha.
    Bryant PL, Schmid JE, Fenton SE, Buckalew AR, Abbott BD.
    Toxicol Sci; 2001 Jul; 62(1):103-14. PubMed ID: 11399798
    [Abstract] [Full Text] [Related]

  • 5. Aryl hydrocarbon receptors in urogenital sinus mesenchyme mediate the inhibition of prostatic epithelial bud formation by 2,3,7,8-tetrachlorodibenzo-p-dioxin.
    Ko K, Moore RW, Peterson RE.
    Toxicol Appl Pharmacol; 2004 Apr 01; 196(1):149-55. PubMed ID: 15050416
    [Abstract] [Full Text] [Related]

  • 6. Effects of TCDD on Ah receptor, ARNT, EGF, and TGF-alpha expression in embryonic mouse urinary tract.
    Bryant PL, Clark GC, Probst MR, Abbott BD.
    Teratology; 1997 May 01; 55(5):326-37. PubMed ID: 9261927
    [Abstract] [Full Text] [Related]

  • 7. 2,3,7,8-Tetrachlorodibenzo-p-dioxin inhibits fibroblast growth factor 10-induced prostatic bud formation in mouse urogenital sinus.
    Vezina CM, Hardin HA, Moore RW, Allgeier SH, Peterson RE.
    Toxicol Sci; 2010 Jan 01; 113(1):198-206. PubMed ID: 19805408
    [Abstract] [Full Text] [Related]

  • 8. Evidence that inhibited prostatic epithelial bud formation in 2,3,7,8-tetrachlorodibenzo-p-dioxin-exposed C57BL/6J fetal mice is not due to interruption of androgen signaling in the urogenital sinus.
    Ko K, Theobald HM, Moore RW, Peterson RE.
    Toxicol Sci; 2004 Jun 01; 79(2):360-9. PubMed ID: 15056816
    [Abstract] [Full Text] [Related]

  • 9. 2,3,7,8-tetrachlorodibenzo-p-dioxin inhibits prostatic epithelial bud formation by acting directly on the urogenital sinus.
    Lin TM, Rasmussen NT, Moore RW, Albrecht RM, Peterson RE.
    J Urol; 2004 Jul 01; 172(1):365-8. PubMed ID: 15201812
    [Abstract] [Full Text] [Related]

  • 10. TCDD-induced altered expression of growth factors may have a role in producing cleft palate and enhancing the incidence of clefts after coadministration of retinoic acid and TCDD.
    Abbott BD, Birnbaum LS.
    Toxicol Appl Pharmacol; 1990 Dec 01; 106(3):418-32. PubMed ID: 2260090
    [Abstract] [Full Text] [Related]

  • 11. Effects of epidermal growth factor (EGF), transforming growth factor-alpha (TGFalpha), and 2,3,7,8-tetrachlorodibenzo-p-dioxin on fusion of embryonic palates in serum-free organ culture using wild-type, EGF knockout, and TGFalpha knockout mouse strains.
    Abbott BD, Buckalew AR, Leffler KE.
    Birth Defects Res A Clin Mol Teratol; 2005 Jun 01; 73(6):447-54. PubMed ID: 15880701
    [Abstract] [Full Text] [Related]

  • 12. Dioxin causes ventral prostate agenesis by disrupting dorsoventral patterning in developing mouse prostate.
    Vezina CM, Allgeier SH, Moore RW, Lin TM, Bemis JC, Hardin HA, Gasiewicz TA, Peterson RE.
    Toxicol Sci; 2008 Dec 01; 106(2):488-96. PubMed ID: 18779384
    [Abstract] [Full Text] [Related]

  • 13. Comparisons of the effects of TCDD and hydrocortisone on growth factor expression provide insight into their interaction in the embryonic mouse palate.
    Abbott BD, Harris MW, Birnbaum LS.
    Teratology; 1992 Jan 01; 45(1):35-53. PubMed ID: 1731395
    [Abstract] [Full Text] [Related]

  • 14. Effects of aryl hydrocarbon receptor null mutation and in utero and lactational 2,3,7,8-tetrachlorodibenzo-p-dioxin exposure on prostate and seminal vesicle development in C57BL/6 mice.
    Lin TM, Ko K, Moore RW, Simanainen U, Oberley TD, Peterson RE.
    Toxicol Sci; 2002 Aug 01; 68(2):479-87. PubMed ID: 12151645
    [Abstract] [Full Text] [Related]

  • 15. AH receptor, ARNT, glucocorticoid receptor, EGF receptor, EGF, TGF alpha, TGF beta 1, TGF beta 2, and TGF beta 3 expression in human embryonic palate, and effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD).
    Abbott BD, Probst MR, Perdew GH, Buckalew AR.
    Teratology; 1998 Aug 01; 58(2):30-43. PubMed ID: 9787404
    [Abstract] [Full Text] [Related]

  • 16. In utero exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin induces amphiregulin gene expression in the developing mouse ureter.
    Choi SS, Miller MA, Harper PA.
    Toxicol Sci; 2006 Nov 01; 94(1):163-74. PubMed ID: 16929009
    [Abstract] [Full Text] [Related]

  • 17. 2,3,7,8-tetrachlorodibenzo-p-dioxin interacts with endogenous estradiol to disrupt prostate gland morphogenesis in male rat fetuses.
    Timms BG, Peterson RE, vom Saal FS.
    Toxicol Sci; 2002 Jun 01; 67(2):264-74. PubMed ID: 12011486
    [Abstract] [Full Text] [Related]

  • 18. Influence of the Ah locus on the effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin on the hepatic epidermal growth factor receptor.
    Lin FH, Clark G, Birnbaum LS, Lucier GW, Goldstein JA.
    Mol Pharmacol; 1991 Mar 01; 39(3):307-13. PubMed ID: 1848654
    [Abstract] [Full Text] [Related]

  • 19. In utero and lactational exposure of the male rat to 2,3,7,8-tetrachlorodibenzo-p-dioxin impairs prostate development. 2. Effects on growth and cytodifferentiation.
    Roman BL, Timms BG, Prins GS, Peterson RE.
    Toxicol Appl Pharmacol; 1998 Jun 01; 150(2):254-70. PubMed ID: 9653056
    [Abstract] [Full Text] [Related]

  • 20. Dioxin-induced retardation of development through a reduction in the expression of pituitary hormones and possible involvement of an aryl hydrocarbon receptor in this defect: a comparative study using two strains of mice with different sensitivities to dioxin.
    Takeda T, Taura J, Hattori Y, Ishii Y, Yamada H.
    Toxicol Appl Pharmacol; 2014 Aug 01; 278(3):220-9. PubMed ID: 24793433
    [Abstract] [Full Text] [Related]

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