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Journal Abstract Search

205 related items for PubMed ID: 14518238

  • 21. [Studies on the temporal profile of expression and release of major pro-inflammatory cytokines in vital organs following hemorrhagic shock].
    Liu YJ, Mao EQ, Li L, Tang YQ, Zhang SD.
    Zhongguo Wei Zhong Bing Ji Jiu Yi Xue; 2007 May; 19(5):290-4. PubMed ID: 17490570
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  • 22. How relevant are in vivo and in vitro studies for clinical sepsis? A mathematical model of LPS signaling based on endotoxin tolerance.
    Vasilescu C, Olteanu M, Flondor P.
    Chirurgia (Bucur); 2009 May; 104(2):195-201. PubMed ID: 19499663
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  • 23. [Protective effect of the cholinergic anti-inflammatory pathway against hemorrhagic shock in rats].
    Li JG, Hu ZF, Du ZH, Zhou Q, Jia BH, Peng ZQ, Ye XF, Li B.
    Zhongguo Wei Zhong Bing Ji Jiu Yi Xue; 2005 Jan; 17(1):24-7. PubMed ID: 15636707
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  • 32. Endotoxin tolerance attenuates LPS-induced TLR4 mobilization to lipid rafts: a condition reversed by PKC activation.
    Cuschieri J, Billigren J, Maier RV.
    J Leukoc Biol; 2006 Dec; 80(6):1289-97. PubMed ID: 16959900
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  • 33. [Induction of early endotoxin tolerance with atoxic endotoxin--a new method for preventing sepsis syndrome].
    Staubach KH, Weber H, Brade H, Bruch HP.
    Langenbecks Arch Chir Suppl Kongressbd; 1998 Dec; 115():1067-70. PubMed ID: 9931792
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  • 35. Reconstituted high-density lipoprotein attenuates organ injury and adhesion molecule expression in a rodent model of endotoxic shock.
    McDonald MC, Dhadly P, Cockerill GW, Cuzzocrea S, Mota-Filipe H, Hinds CJ, Miller NE, Thiemermann C.
    Shock; 2003 Dec; 20(6):551-7. PubMed ID: 14625480
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