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Journal Abstract Search

201 related items for PubMed ID: 14604841

  • 1.
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  • 2. Destruction of serotonergic nerve terminals prevents fluoxetine-induced desensitization of hypothalamic 5-HT(1A) receptors.
    D'Souza DN, Zhang Y, Garcia F, Battaglia G, Van De Kar LD.
    Psychopharmacology (Berl); 2002 Dec; 164(4):392-400. PubMed ID: 12457269
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  • 3. Daily injections of fluoxetine induce dose-dependent desensitization of hypothalamic 5-HT1A receptors: reductions in neuroendocrine responses to 8-OH-DPAT and in levels of Gz and Gi proteins.
    Raap DK, Evans S, Garcia F, Li Q, Muma NA, Wolf WA, Battaglia G, Van De Kar LD.
    J Pharmacol Exp Ther; 1999 Jan; 288(1):98-106. PubMed ID: 9862759
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  • 4. Sustained desensitization of hypothalamic 5-Hydroxytryptamine1A receptors after discontinuation of fluoxetine: inhibited neuroendocrine responses to 8-hydroxy-2-(Dipropylamino)Tetralin in the absence of changes in Gi/o/z proteins.
    Raap DK, Garcia F, Muma NA, Wolf WA, Battaglia G, van de Kar LD.
    J Pharmacol Exp Ther; 1999 Feb; 288(2):561-7. PubMed ID: 9918559
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  • 5. Fluoxetine treatment of prepubescent rats produces a selective functional reduction in the 5-HT2A receptor-mediated stimulation of oxytocin.
    Landry M, Frasier M, Chen Z, Van De Kar LD, Zhang Y, Garcia F, Battaglia G.
    Synapse; 2005 Nov; 58(2):102-9. PubMed ID: 16088947
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  • 6. Treatment of cycling female rats with fluoxetine induces desensitization of hypothalamic 5-HT(1A) receptors with no change in 5-HT(2A) receptors.
    Van de Kar LD, Raap DK, Battaglia G, Muma NA, Garcia F, DonCarlos LL.
    Neuropharmacology; 2002 Jul; 43(1):45-54. PubMed ID: 12213258
    [Abstract] [Full Text] [Related]

  • 7. Chronic fluoxetine induces a gradual desensitization of 5-HT1A receptors: reductions in hypothalamic and midbrain Gi and G(o) proteins and in neuroendocrine responses to a 5-HT1A agonist.
    Li Q, Muma NA, van de Kar LD.
    J Pharmacol Exp Ther; 1996 Nov; 279(2):1035-42. PubMed ID: 8930214
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  • 10. WAY-100635 inhibits 8-OH-DPAT-stimulated oxytocin, ACTH and corticosterone, but not prolactin secretion.
    Vicentic A, Li Q, Battaglia G, Van de Kar LD.
    Eur J Pharmacol; 1998 Apr 10; 346(2-3):261-6. PubMed ID: 9652368
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  • 13. Chronic treatment with fluoxetine decreases cerebral metabolic responses to the 5-HT1A agonist 8-hydroxy-2(di-N-propylamino)tetralin and increases those to the 5-HT2A/2C agonist 1-(2,5-dimethoxy-4-iodophenyl)-2-aminopropane and to the dopaminergic agonist apomorphine.
    Freo U, Merico A, Ermani M, Ori C.
    Brain Res; 2010 Jun 04; 1335():24-34. PubMed ID: 20381465
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  • 16. Repeated cocaine exposure inhibits the adrenocorticotropic hormone response to the serotonin releaser d-fenfluramine and the 5-HT1A agonist, 8-OH-DPAT.
    Levy AD, Li Q, Van de Kar LD.
    Neuropharmacology; 1994 Jun 04; 33(3-4):335-42. PubMed ID: 7984271
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  • 18. Reduction of 5-hydroxytryptamine (5-HT)(1A)-mediated temperature and neuroendocrine responses and 5-HT(1A) binding sites in 5-HT transporter knockout mice.
    Li Q, Wichems C, Heils A, Van De Kar LD, Lesch KP, Murphy DL.
    J Pharmacol Exp Ther; 1999 Dec 04; 291(3):999-1007. PubMed ID: 10565817
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  • 19. Modulation of 5-HT1A receptor mediated response by fluoxetine in rat brain.
    Subhash MN, Srinivas BN, Vinod KY, Jagadeesh S.
    J Neural Transm (Vienna); 2000 Dec 04; 107(3):377-87. PubMed ID: 10821446
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