These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
Pubmed for Handhelds
PUBMED FOR HANDHELDS
Journal Abstract Search
155 related items for PubMed ID: 14623233
1. In vivo mutagenesis of the Hoxb8 hexapeptide domain leads to dominant homeotic transformations that mimic the loss-of-function mutations in genes of the Hoxb cluster. Medina-Martínez O, Ramírez-Solis R. Dev Biol; 2003 Dec 01; 264(1):77-90. PubMed ID: 14623233 [Abstract] [Full Text] [Related]
2. Loss of function but no gain of function caused by amino acid substitutions in the hexapeptide of Hoxa1 in vivo. Remacle S, Abbas L, De Backer O, Pacico N, Gavalas A, Gofflot F, Picard JJ, Rezsöhazy R. Mol Cell Biol; 2004 Oct 01; 24(19):8567-75. PubMed ID: 15367676 [Abstract] [Full Text] [Related]
3. An extradenticle-induced conformational change in a HOX protein overcomes an inhibitory function of the conserved hexapeptide motif. Chan SK, Pöpperl H, Krumlauf R, Mann RS. EMBO J; 1996 May 15; 15(10):2476-87. PubMed ID: 8665855 [Abstract] [Full Text] [Related]
6. Mutations in paralogous Hox genes result in overlapping homeotic transformations of the axial skeleton: evidence for unique and redundant function. Horan GS, Kovàcs EN, Behringer RR, Featherstone MS. Dev Biol; 1995 May 15; 169(1):359-72. PubMed ID: 7750651 [Abstract] [Full Text] [Related]
7. Compound mutants for the paralogous hoxa-4, hoxb-4, and hoxd-4 genes show more complete homeotic transformations and a dose-dependent increase in the number of vertebrae transformed. Horan GS, Ramírez-Solis R, Featherstone MS, Wolgemuth DJ, Bradley A, Behringer RR. Genes Dev; 1995 Jul 01; 9(13):1667-77. PubMed ID: 7628700 [Abstract] [Full Text] [Related]
8. The hexapeptide LFPWMR in Hoxb-8 is required for cooperative DNA binding with Pbx1 and Pbx2 proteins. Neuteboom ST, Peltenburg LT, van Dijk MA, Murre C. Proc Natl Acad Sci U S A; 1995 Sep 26; 92(20):9166-70. PubMed ID: 7568094 [Abstract] [Full Text] [Related]
12. HoxB8 requires its Pbx-interaction motif to block differentiation of primary myeloid progenitors and of most cell line models of myeloid differentiation. Knoepfler PS, Sykes DB, Pasillas M, Kamps MP. Oncogene; 2001 Sep 06; 20(39):5440-8. PubMed ID: 11571641 [Abstract] [Full Text] [Related]
16. Genetic interaction between hoxb-5 and hoxb-6 is revealed by nonallelic noncomplementation. Rancourt DE, Tsuzuki T, Capecchi MR. Genes Dev; 1995 Jan 01; 9(1):108-22. PubMed ID: 7828847 [Abstract] [Full Text] [Related]
17. Retinoic acid regulates a subset of Cdx1 function in vivo. Houle M, Sylvestre JR, Lohnes D. Development; 2003 Dec 01; 130(26):6555-67. PubMed ID: 14660544 [Abstract] [Full Text] [Related]
18. Selection of distinct Hox-Extradenticle interaction modes fine-tunes Hox protein activity. Saadaoui M, Merabet S, Litim-Mecheri I, Arbeille E, Sambrani N, Damen W, Brena C, Pradel J, Graba Y. Proc Natl Acad Sci U S A; 2011 Feb 08; 108(6):2276-81. PubMed ID: 21262810 [Abstract] [Full Text] [Related]
19. Defective neural tube closure and anteroposterior patterning in mice lacking the LIM protein LMO4 or its interacting partner Deaf-1. Hahm K, Sum EY, Fujiwara Y, Lindeman GJ, Visvader JE, Orkin SH. Mol Cell Biol; 2004 Mar 08; 24(5):2074-82. PubMed ID: 14966286 [Abstract] [Full Text] [Related]