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Journal Abstract Search

213 related items for PubMed ID: 14633129

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  • 4. Sequence of events in the glomerular capillary wall at the onset of proteinuria in passive Heymann nephritis.
    Fujigaki Y, Batsford S, Yamashita F, Yonemura K, Hishida A, Kawachi H, Shimizu F, Vogt A.
    Virchows Arch; 2001 Feb; 438(2):136-45. PubMed ID: 11253115
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  • 6. Complement and monocytes are essential for provoking glomerular injury in passive Heymann nephritis in rats. Terminal complement components are not the sole mediators of proteinuria.
    Hara M, Batsford SR, Mihatsch MJ, Bitter-Suermann D, Vogt A.
    Lab Invest; 1991 Aug; 65(2):168-79. PubMed ID: 1881120
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  • 7. Basic fibroblast growth factor augments podocyte injury and induces glomerulosclerosis in rats with experimental membranous nephropathy.
    Floege J, Kriz W, Schulze M, Susani M, Kerjaschki D, Mooney A, Couser WG, Koch KM.
    J Clin Invest; 1995 Dec; 96(6):2809-19. PubMed ID: 8675651
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  • 11. Podocyte slit-diaphragm protein nephrin is linked to the actin cytoskeleton.
    Yuan H, Takeuchi E, Salant DJ.
    Am J Physiol Renal Physiol; 2002 Apr; 282(4):F585-91. PubMed ID: 11880318
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  • 13. Depletion of C6 prevents development of proteinuria in experimental membranous nephropathy in rats.
    Baker PJ, Ochi RF, Schulze M, Johnson RJ, Campbell C, Couser WG.
    Am J Pathol; 1989 Jul; 135(1):185-94. PubMed ID: 2672823
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  • 18. Rapamycin ameliorates proteinuria and restores nephrin and podocin expression in experimental membranous nephropathy.
    Stratakis S, Stylianou K, Petrakis I, Mavroeidi V, Poulidaki R, Petra C, Moisiadis D, Stratigis S, Vardaki E, Nakopoulou L, Daphnis E.
    Clin Dev Immunol; 2013 Jul; 2013():941893. PubMed ID: 24069045
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  • 19. Nephrin redistribution on podocytes is a potential mechanism for proteinuria in patients with primary acquired nephrotic syndrome.
    Doublier S, Ruotsalainen V, Salvidio G, Lupia E, Biancone L, Conaldi PG, Reponen P, Tryggvason K, Camussi G.
    Am J Pathol; 2001 May; 158(5):1723-31. PubMed ID: 11337370
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