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Journal Abstract Search

225 related items for PubMed ID: 14658402

  • 1. [Clinical features and neuropathological findings of familial amyotrophic lateral sclerosis with an H43R mutation in Cu/Zn superoxide dismutase].
    Mochizuki Y, Mizutani T, Nakano R, Fukushima T, Honma T, Nemoto N, Takei K.
    Rinsho Shinkeigaku; 2003 Aug; 43(8):491-5. PubMed ID: 14658402
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  • 4. Clinical features and neuropathological findings of familial amyotrophic lateral sclerosis with a His46Arg mutation in Cu/Zn superoxide dismutase.
    Ohi T, Saita K, Takechi S, Nabesima K, Tashiro H, Shiomi K, Sugimoto S, Akematsu T, Nakayama T, Iwaki T, Matsukura S.
    J Neurol Sci; 2002 May 15; 197(1-2):73-8. PubMed ID: 11997070
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  • 10. Familial amyotrophic lateral sclerosis with onset in bulbar sign, benign clinical course, and Bunina bodies: a clinical, genetic, and pathological study of a Japanese family.
    Tsuchiya K, Shintani S, Nakabayashi H, Kikugawa K, Nakano R, Haga C, Nakano I, Ikeda K, Tsuji S.
    Acta Neuropathol; 2000 Dec 15; 100(6):603-7. PubMed ID: 11078211
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  • 12. Familial amyotrophic lateral sclerosis: a SOD1-unrelated Japanese family of bulbar type with Bunina bodies and ubiquitin-positive skein-like inclusions in lower motor neurons.
    Tagawa A, Tan CF, Kikugawa K, Fukase M, Nakano R, Onodera O, Nishizawa M, Takahashi H.
    Acta Neuropathol; 2007 Feb 15; 113(2):205-11. PubMed ID: 17036243
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  • 15. Familial amyotrophic lateral sclerosis with posterior column degeneration and basophilic inclusion bodies: a clinical, genetic and pathological study.
    Tsuchiya K, Matsunaga T, Aoki M, Haga C, Ooe K, Abe K, Ikeda K, Nakano I.
    Clin Neuropathol; 2001 Feb 15; 20(2):53-9. PubMed ID: 11327297
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  • 16. Absence of SOD1 gene abnormalities in familial amyotrophic lateral sclerosis with posterior column involvement without Lewy-body-like hyaline inclusions.
    Kato S, Kawata A, Oda M, Arai N, Komori T, Tanabe H.
    Acta Neuropathol; 1996 Nov 15; 92(5):528-33. PubMed ID: 8922067
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  • 17. TDP-43 immunoreactivity in neuronal inclusions in familial amyotrophic lateral sclerosis with or without SOD1 gene mutation.
    Tan CF, Eguchi H, Tagawa A, Onodera O, Iwasaki T, Tsujino A, Nishizawa M, Kakita A, Takahashi H.
    Acta Neuropathol; 2007 May 15; 113(5):535-42. PubMed ID: 17333220
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  • 18. [Different clinical phenotypes of siblings with familial amyotrophic lateral sclerosis showing Cys146Arg point mutation of superoxide dismutase 1 gene].
    Ito K, Uchiyama T, Fukutake T, Arai K, Kanesaka T, Hattori T.
    Rinsho Shinkeigaku; 2002 Feb 15; 42(2):175-7. PubMed ID: 12424972
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  • 19. [Two siblings of familial amyotrophic lateral sclerosis with multisystemic degeneration characterized by mild involvement of the middle root zone of the posterior column, Clarke's nuclei and spinocerebellar tract].
    Yoshida M, Okuda S, Murakami N, Hashizume Y, Sobue G.
    Rinsho Shinkeigaku; 1995 Jun 15; 35(6):589-99. PubMed ID: 8521632
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  • 20. Molecular analyses of the Cu/Zn superoxide dismutase gene in patients with familial amyotrophic lateral sclerosis (ALS) in Japan.
    Aoki M, Abe K, Itoyama Y.
    Cell Mol Neurobiol; 1998 Dec 15; 18(6):639-47. PubMed ID: 9876871
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