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Journal Abstract Search

214 related items for PubMed ID: 14732230

  • 1. The Bmi-1 oncoprotein is overexpressed in human colorectal cancer and correlates with the reduced p16INK4a/p14ARF proteins.
    Kim JH, Yoon SY, Kim CN, Joo JH, Moon SK, Choe IS, Choe YK, Kim JW.
    Cancer Lett; 2004 Jan 20; 203(2):217-24. PubMed ID: 14732230
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  • 2. The bmi-1 oncoprotein is differentially expressed in non-small cell lung cancer and correlates with INK4A-ARF locus expression.
    Vonlanthen S, Heighway J, Altermatt HJ, Gugger M, Kappeler A, Borner MM, van Lohuizen M, Betticher DC.
    Br J Cancer; 2001 May 18; 84(10):1372-6. PubMed ID: 11355949
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  • 3. Implication of polycomb members Bmi-1, Mel-18, and Hpc-2 in the regulation of p16INK4a, p14ARF, h-TERT, and c-Myc expression in primary breast carcinomas.
    Silva J, García JM, Peña C, García V, Domínguez G, Suárez D, Camacho FI, Espinosa R, Provencio M, España P, Bonilla F.
    Clin Cancer Res; 2006 Dec 01; 12(23):6929-36. PubMed ID: 17145810
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  • 4. USP22 acts as an oncogene by the activation of BMI-1-mediated INK4a/ARF pathway and Akt pathway.
    Liu YL, Jiang SX, Yang YM, Xu H, Liu JL, Wang XS.
    Cell Biochem Biophys; 2012 Jan 01; 62(1):229-35. PubMed ID: 21928107
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  • 5. Increased expression of ubiquitin-specific protease 22 can promote cancer progression and predict therapy failure in human colorectal cancer.
    Liu YL, Yang YM, Xu H, Dong XS.
    J Gastroenterol Hepatol; 2010 Nov 01; 25(11):1800-5. PubMed ID: 21039844
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  • 6. Concomitant expression of p16INK4a and p14ARF in primary breast cancer and analysis of inactivation mechanisms.
    Silva J, Silva JM, Domínguez G, García JM, Cantos B, Rodríguez R, Larrondo FJ, Provencio M, España P, Bonilla F.
    J Pathol; 2003 Mar 01; 199(3):289-97. PubMed ID: 12579530
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  • 7. Expression of p16INK4a/p16alpha and p19ARF/p16beta is frequently altered in non-small cell lung cancer and correlates with p53 overexpression.
    Vonlanthen S, Heighway J, Tschan MP, Borner MM, Altermatt HJ, Kappeler A, Tobler A, Fey MF, Thatcher N, Yarbrough WG, Betticher DC.
    Oncogene; 1998 Nov 26; 17(21):2779-85. PubMed ID: 9840942
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  • 15. Bmi-1 promotes neural stem cell self-renewal and neural development but not mouse growth and survival by repressing the p16Ink4a and p19Arf senescence pathways.
    Molofsky AV, He S, Bydon M, Morrison SJ, Pardal R.
    Genes Dev; 2005 Jun 15; 19(12):1432-7. PubMed ID: 15964994
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  • 16. Overexpression of p16INK4A and p14ARF in haematological malignancies.
    Lee YK, Park JY, Kang HJ, Cho HC.
    Clin Lab Haematol; 2003 Aug 15; 25(4):233-7. PubMed ID: 12890162
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  • 17. Hypermethylation-associated inactivation of p14(ARF) is independent of p16(INK4a) methylation and p53 mutational status.
    Esteller M, Tortola S, Toyota M, Capella G, Peinado MA, Baylin SB, Herman JG.
    Cancer Res; 2000 Jan 01; 60(1):129-33. PubMed ID: 10646864
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  • 19. [Relationship between alterations of p16INK4a and p14ARF genes of CDKN2A locus and gastric carcinogenesis].
    Tang SH, Yang DH, Luo HS, Yu JP, Shu JC.
    Zhonghua Liu Xing Bing Xue Za Zhi; 2004 Jun 01; 25(6):517-21. PubMed ID: 15231134
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