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Journal Abstract Search

261 related items for PubMed ID: 14738882

  • 1. DSCR1 gene expression is dependent on NFATc1 during cardiac valve formation and colocalizes with anomalous organ development in trisomy 16 mice.
    Lange AW, Molkentin JD, Yutzey KE.
    Dev Biol; 2004 Feb 15; 266(2):346-60. PubMed ID: 14738882
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  • 2. Restoration of DSCR1 to disomy in the trisomy 16 mouse model of Down syndrome does not correct cardiac or craniofacial development anomalies.
    Lange AW, Rothermel BA, Yutzey KE.
    Dev Dyn; 2005 Jul 15; 233(3):954-63. PubMed ID: 15906378
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  • 4. Characterization of Nfatc1 regulation identifies an enhancer required for gene expression that is specific to pro-valve endocardial cells in the developing heart.
    Zhou B, Wu B, Tompkins KL, Boyer KL, Grindley JC, Baldwin HS.
    Development; 2005 Mar 15; 132(5):1137-46. PubMed ID: 15689382
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  • 6. NFATc1 expression in the developing heart valves is responsive to the RANKL pathway and is required for endocardial expression of cathepsin K.
    Lange AW, Yutzey KE.
    Dev Biol; 2006 Apr 15; 292(2):407-17. PubMed ID: 16680826
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  • 7. Sculpting heart valves with NFATc and VEGF.
    Lambrechts D, Carmeliet P.
    Cell; 2004 Sep 03; 118(5):532-4. PubMed ID: 15339657
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  • 8. DSCR1, overexpressed in Down syndrome, is an inhibitor of calcineurin-mediated signaling pathways.
    Fuentes JJ, Genescà L, Kingsbury TJ, Cunningham KW, Pérez-Riba M, Estivill X, de la Luna S.
    Hum Mol Genet; 2000 Jul 01; 9(11):1681-90. PubMed ID: 10861295
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  • 10. Trisomy of the Dscr1 gene suppresses early progression of pancreatic intraepithelial neoplasia driven by oncogenic Kras.
    Lee JC, Shin J, Baek KH.
    Biochem Biophys Res Commun; 2013 Oct 11; 440(1):50-5. PubMed ID: 24041692
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  • 11. The secondary heart field is a new site of calcineurin/Nfatc1 signaling for semilunar valve development.
    Lin CY, Lin CJ, Chen CH, Chen RM, Zhou B, Chang CP.
    J Mol Cell Cardiol; 2012 May 11; 52(5):1096-102. PubMed ID: 22300732
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  • 13. VEGF selectively induces Down syndrome critical region 1 gene expression in endothelial cells: a mechanism for feedback regulation of angiogenesis?
    Yao YG, Duh EJ.
    Biochem Biophys Res Commun; 2004 Aug 27; 321(3):648-56. PubMed ID: 15358155
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  • 15. Depolarization of neural cells induces transcription of the Down syndrome critical region 1 isoform 4 via a calcineurin/nuclear factor of activated T cells-dependent pathway.
    Cano E, Canellada A, Minami T, Iglesias T, Redondo JM.
    J Biol Chem; 2005 Aug 19; 280(33):29435-43. PubMed ID: 15975916
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  • 16. A single extra copy of Dscr1 improves survival of mice developing spontaneous lung tumors through suppression of tumor angiogenesis.
    Shin J, Lee JC, Baek KH.
    Cancer Lett; 2014 Jan 01; 342(1):70-81. PubMed ID: 24051307
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  • 17. DSCR1 (ADAPT78) lethality: evidence for a protective effect of trisomy 21 genes?
    Kluetzman KS, Perez AV, Crawford DR.
    Biochem Biophys Res Commun; 2005 Nov 18; 337(2):595-601. PubMed ID: 16198305
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