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252 related items for PubMed ID: 14769389

  • 1. 5-HT2C receptors inhibit and 5-HT1A receptors activate the generation of spike-wave discharges in a genetic rat model of absence epilepsy.
    Jakus R, Graf M, Juhasz G, Gerber K, Levay G, Halasz P, Bagdy G.
    Exp Neurol; 2003 Dec; 184(2):964-72. PubMed ID: 14769389
    [Abstract] [Full Text] [Related]

  • 2. Evidence for accelerated desensitisation of 5-HT(2C) receptors following combined treatment with fluoxetine and the 5-HT(1A) receptor antagonist, WAY 100,635, in the rat.
    Bristow LJ, O'Connor D, Watts R, Duxon MS, Hutson PH.
    Neuropharmacology; 2000 Apr 27; 39(7):1222-36. PubMed ID: 10760364
    [Abstract] [Full Text] [Related]

  • 3. Mechanism of action of the bimodal antidepressant vilazodone: evidence for serotonin1A-receptor-mediated auto-augmentation of extracellular serotonin output.
    van Amsterdam C, Seyfried CA.
    Psychopharmacology (Berl); 2014 Jun 27; 231(12):2547-58. PubMed ID: 24419272
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  • 4. Activation of 5-HT2C (but not 5-HT1A) receptors in the amygdala enhances fear-induced antinociception: Blockade with local 5-HT2C antagonist or systemic fluoxetine.
    Tavares LRR, Baptista-de-Souza D, Canto-de-Souza A.
    Neuropharmacology; 2018 Jun 27; 135():376-385. PubMed ID: 29548885
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  • 5. Anxiety-like effects induced by acute fluoxetine, sertraline or m-CPP treatment are reversed by pretreatment with the 5-HT2C receptor antagonist SB-242084 but not the 5-HT1A receptor antagonist WAY-100635.
    Bagdy G, Graf M, Anheuer ZE, Modos EA, Kantor S.
    Int J Neuropsychopharmacol; 2001 Dec 27; 4(4):399-408. PubMed ID: 11806866
    [Abstract] [Full Text] [Related]

  • 6. Prelimbic cortex 5-HT1A and 5-HT2C receptors are involved in the hypophagic effects caused by fluoxetine in fasted rats.
    Stanquini LA, Resstel LB, Corrêa FM, Joca SR, Scopinho AA.
    Pharmacol Biochem Behav; 2015 Sep 27; 136():31-8. PubMed ID: 26143050
    [Abstract] [Full Text] [Related]

  • 7. The 5-HT1A agonist 8-OH-DPAT increases the number of spike-wave discharges in a genetic rat model of absence epilepsy.
    Gerber K, Filakovszky J, Halasz P, Bagdy G.
    Brain Res; 1998 Oct 05; 807(1-2):243-5. PubMed ID: 9757054
    [Abstract] [Full Text] [Related]

  • 8. Citalopram-induced hypophagia is enhanced by blockade of 5-HT(1A) receptors: role of 5-HT(2C) receptors.
    Grignaschi G, Invernizzi RW, Fanelli E, Fracasso C, Caccia S, Samanin R.
    Br J Pharmacol; 1998 Aug 05; 124(8):1781-7. PubMed ID: 9756397
    [Abstract] [Full Text] [Related]

  • 9. Chronic escitalopram treatment caused dissociative adaptation in serotonin (5-HT) 2C receptor antagonist-induced effects in REM sleep, wake and theta wave activity.
    Kostyalik D, Kátai Z, Vas S, Pap D, Petschner P, Molnár E, Gyertyán I, Kalmár L, Tóthfalusi L, Bagdy G.
    Exp Brain Res; 2014 Mar 05; 232(3):935-46. PubMed ID: 24395141
    [Abstract] [Full Text] [Related]

  • 10. Role of 5-HT(1A) receptors in fluoxetine-induced lordosis inhibition.
    Guptarak J, Sarkar J, Hiegel C, Uphouse L.
    Horm Behav; 2010 Jul 05; 58(2):290-6. PubMed ID: 20223238
    [Abstract] [Full Text] [Related]

  • 11. In vivo evidence that 5-HT(2C) receptors inhibit 5-HT neuronal activity via a GABAergic mechanism.
    Boothman L, Raley J, Denk F, Hirani E, Sharp T.
    Br J Pharmacol; 2006 Dec 05; 149(7):861-9. PubMed ID: 17043669
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  • 18. A 5-HT(2C) receptor-mediated interaction between 2,5-dimethoxy-4-methylamphetamine and citalopram in the rat.
    Eckler JR, Reissig CJ, Rabin RA, Winter JC.
    Pharmacol Biochem Behav; 2004 Sep 05; 79(1):25-30. PubMed ID: 15388280
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  • 20. Modulation of the subthalamic nucleus activity by serotonergic agents and fluoxetine administration.
    Aristieta A, Morera-Herreras T, Ruiz-Ortega JA, Miguelez C, Vidaurrazaga I, Arrue A, Zumarraga M, Ugedo L.
    Psychopharmacology (Berl); 2014 May 05; 231(9):1913-24. PubMed ID: 24271033
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