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Journal Abstract Search

250 related items for PubMed ID: 14966563

  • 1.
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  • 2. Genetic and pharmacologic analyses of the role of Icmt in Ras membrane association and function.
    Svensson AW, Casey PJ, Young SG, Bergo MO.
    Methods Enzymol; 2006; 407():144-59. PubMed ID: 16757321
    [Abstract] [Full Text] [Related]

  • 3. Targeted inactivation of the isoprenylcysteine carboxyl methyltransferase gene causes mislocalization of K-Ras in mammalian cells.
    Bergo MO, Leung GK, Ambroziak P, Otto JC, Casey PJ, Young SG.
    J Biol Chem; 2000 Jun 09; 275(23):17605-10. PubMed ID: 10747846
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  • 4. Rit, a non-lipid-modified Ras-related protein, transforms NIH3T3 cells without activating the ERK, JNK, p38 MAPK or PI3K/Akt pathways.
    Rusyn EV, Reynolds ER, Shao H, Grana TM, Chan TO, Andres DA, Cox AD.
    Oncogene; 2000 Sep 28; 19(41):4685-94. PubMed ID: 11032018
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  • 6. Absence of the CAAX endoprotease Rce1: effects on cell growth and transformation.
    Bergo MO, Ambroziak P, Gregory C, George A, Otto JC, Kim E, Nagase H, Casey PJ, Balmain A, Young SG.
    Mol Cell Biol; 2002 Jan 28; 22(1):171-81. PubMed ID: 11739732
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  • 7. Inactivating Icmt ameliorates K-RAS-induced myeloproliferative disease.
    Wahlstrom AM, Cutts BA, Liu M, Lindskog A, Karlsson C, Sjogren AK, Andersson KM, Young SG, Bergo MO.
    Blood; 2008 Aug 15; 112(4):1357-65. PubMed ID: 18502828
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  • 8. Isoprenylcysteine carboxyl methyltransferase activity modulates endothelial cell apoptosis.
    Kramer K, Harrington EO, Lu Q, Bellas R, Newton J, Sheahan KL, Rounds S.
    Mol Biol Cell; 2003 Mar 15; 14(3):848-57. PubMed ID: 12631708
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  • 11. Chemical genetic blockade of transformation reveals dependence on aberrant oncogenic signaling.
    Fan QW, Zhang C, Shokat KM, Weiss WA.
    Curr Biol; 2002 Aug 20; 12(16):1386-94. PubMed ID: 12194819
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  • 13. Ornithine decarboxylase induction in transformation by H-Ras and RhoA.
    Shantz LM, Pegg AE.
    Cancer Res; 1998 Jul 01; 58(13):2748-53. PubMed ID: 9661886
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  • 14. Raf and RhoA cooperate to transform intestinal epithelial cells and induce growth resistance to transforming growth factor beta.
    Du J, Jiang B, Coffey RJ, Barnard J.
    Mol Cancer Res; 2004 Apr 01; 2(4):233-41. PubMed ID: 15140945
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  • 15. Lysyl oxidase inhibits ras-mediated transformation by preventing activation of NF-kappa B.
    Jeay S, Pianetti S, Kagan HM, Sonenshein GE.
    Mol Cell Biol; 2003 Apr 01; 23(7):2251-63. PubMed ID: 12640111
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  • 16. Radicicol suppresses transformation and restores tropomyosin-2 expression in both ras- and MEK-transformed cells without inhibiting the Raf/MEK/ERK signaling cascade.
    Kim PN, Jonasch E, Mosterman BC, Mier JW, Janssen RA.
    Cell Growth Differ; 2001 Nov 01; 12(11):543-50. PubMed ID: 11714635
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  • 17. Oncogenic Ras activation of Raf/mitogen-activated protein kinase-independent pathways is sufficient to cause tumorigenic transformation.
    Khosravi-Far R, White MA, Westwick JK, Solski PA, Chrzanowska-Wodnicka M, Van Aelst L, Wigler MH, Der CJ.
    Mol Cell Biol; 1996 Jul 01; 16(7):3923-33. PubMed ID: 8668210
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  • 18. A small-molecule inhibitor of isoprenylcysteine carboxyl methyltransferase with antitumor activity in cancer cells.
    Winter-Vann AM, Baron RA, Wong W, dela Cruz J, York JD, Gooden DM, Bergo MO, Young SG, Toone EJ, Casey PJ.
    Proc Natl Acad Sci U S A; 2005 Mar 22; 102(12):4336-41. PubMed ID: 15784746
    [Abstract] [Full Text] [Related]

  • 19. The downregulation of the pro-apoptotic protein Par-4 is critical for Ras-induced survival and tumor progression.
    Barradas M, Monjas A, Diaz-Meco MT, Serrano M, Moscat J.
    EMBO J; 1999 Nov 15; 18(22):6362-9. PubMed ID: 10562548
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  • 20. Isoprenylcysteine carboxyl methyltransferase is critical for glioblastoma growth and survival by activating Ras/Raf/Mek/Erk.
    Wan W, Xiao W, Pan W, Chen L, Liu Z, Xu J.
    Cancer Chemother Pharmacol; 2022 Mar 15; 89(3):401-411. PubMed ID: 35171349
    [Abstract] [Full Text] [Related]

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