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Journal Abstract Search
270 related items for PubMed ID: 15023869
21. Myocardial triglyceride turnover and contribution to energy substrate utilization in isolated working rat hearts. Saddik M, Lopaschuk GD. J Biol Chem; 1991 May 05; 266(13):8162-70. PubMed ID: 1902472 [Abstract] [Full Text] [Related]
22. Functional and metabolic remodelling in GLUT4-deficient hearts confers hyper-responsiveness to substrate intervention. Huggins CE, Domenighetti AA, Ritchie ME, Khalil N, Favaloro JM, Proietto J, Smyth GK, Pepe S, Delbridge LM. J Mol Cell Cardiol; 2008 Feb 05; 44(2):270-80. PubMed ID: 18191142 [Abstract] [Full Text] [Related]
23. Myocardial energy provision is preserved by increased utilization of glucose and ketone bodies in CD36 knockout mice. Nakatani K, Watabe T, Masuda D, Imaizumi M, Shimosegawa E, Kobayashi T, Sairyo M, Zhu Y, Okada T, Kawase R, Nakaoka H, Naito A, Ohama T, Koseki M, Oka T, Akazawa H, Nishida M, Komuro I, Sakata Y, Hatazawa J, Yamashita S. Metabolism; 2015 Sep 05; 64(9):1165-74. PubMed ID: 26130608 [Abstract] [Full Text] [Related]
25. Accelerated glycolysis and greater postischemic dysfunction in hypertrophied rat hearts are independent of coronary flow. Wambolt RB, Grist M, Bondy GP, Allard MF. Can J Cardiol; 2001 Aug 05; 17(8):889-94. PubMed ID: 11521131 [Abstract] [Full Text] [Related]
36. Myocardial recovery from ischemia is impaired in CD36-null mice and restored by myocyte CD36 expression or medium-chain fatty acids. Irie H, Krukenkamp IB, Brinkmann JF, Gaudette GR, Saltman AE, Jou W, Glatz JF, Abumrad NA, Ibrahimi A. Proc Natl Acad Sci U S A; 2003 May 27; 100(11):6819-24. PubMed ID: 12746501 [Abstract] [Full Text] [Related]
37. Efficacy of ischaemic preconditioning in the eNOS overexpressed working mouse heart model. du Toit EF, Genade S, Carlini S, Moolman JA, Brunner F, Lochner A. Eur J Pharmacol; 2007 Feb 05; 556(1-3):115-20. PubMed ID: 17157294 [Abstract] [Full Text] [Related]
38. The antianginal drug trimetazidine shifts cardiac energy metabolism from fatty acid oxidation to glucose oxidation by inhibiting mitochondrial long-chain 3-ketoacyl coenzyme A thiolase. Kantor PF, Lucien A, Kozak R, Lopaschuk GD. Circ Res; 2000 Mar 17; 86(5):580-8. PubMed ID: 10720420 [Abstract] [Full Text] [Related]
39. A novel partial fatty acid oxidation inhibitor decreases myocardial oxygen consumption and improves cardiac efficiency in demand-induced ischemic heart. Wu L, Belardinelli L, Fraser H. J Cardiovasc Pharmacol; 2008 Apr 17; 51(4):372-9. PubMed ID: 18427280 [Abstract] [Full Text] [Related]