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Journal Abstract Search

188 related items for PubMed ID: 15031800

  • 1. Compartmentalization of tolerance to endotoxin.
    Fitting C, Dhawan S, Cavaillon JM.
    J Infect Dis; 2004 Apr 01; 189(7):1295-303. PubMed ID: 15031800
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  • 2. Glucocorticoid-dependent and -independent mechanisms involved in lipopolysaccharide tolerance.
    Evans GF, Zuckerman SH.
    Eur J Immunol; 1991 Sep 01; 21(9):1973-9. PubMed ID: 1716205
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  • 3. Sublethal hemorrhagic shock reduces tumor necrosis factor-alpha-producing capacity in different cell compartments.
    Flohé S, Ackermann M, Reuter M, Nast-Kolb D, Schade FU.
    Eur Cytokine Netw; 2000 Sep 01; 11(3):420-6. PubMed ID: 11022127
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  • 5. Endotoxin tolerance: is there a clinical relevance?
    Cavaillon JM, Adrie C, Fitting C, Adib-Conquy M.
    J Endotoxin Res; 2003 Sep 01; 9(2):101-7. PubMed ID: 12803883
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  • 6. Endotoxin-macrophage interaction: post-translational regulation of tumor necrosis factor expression.
    Zuckerman SH, Evans GF, Snyder YM, Roeder WD.
    J Immunol; 1989 Aug 15; 143(4):1223-7. PubMed ID: 2501392
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  • 7. In vivo lipopolysaccharide exposure of human blood leukocytes induces cross-tolerance to multiple TLR ligands.
    de Vos AF, Pater JM, van den Pangaart PS, de Kruif MD, van 't Veer C, van der Poll T.
    J Immunol; 2009 Jul 01; 183(1):533-42. PubMed ID: 19542464
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  • 8. Staphylococcal enterotoxin B induces hepatic injury and lethal shock in endotoxin-resistant C3H/HeJ mice despite a deficient macrophage response.
    Yasuda S, Nagaki M, Moriwaki H.
    J Endotoxin Res; 2002 Jul 01; 8(4):253-61. PubMed ID: 12230915
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  • 9. Early endotoxin tolerance is associated with alterations in bone marrow-derived macrophage precursor pools.
    Madonna GS, Vogel SN.
    J Immunol; 1985 Dec 01; 135(6):3763-71. PubMed ID: 3905964
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  • 13. Differential regulation of cytokine and chemokine production in lipopolysaccharide-induced tolerance and priming.
    Peck OM, Williams DL, Breuel KF, Kalbfleisch JH, Fan H, Tempel GE, Teti G, Cook JA.
    Cytokine; 2004 Jun 07; 26(5):202-8. PubMed ID: 15157897
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  • 16. How relevant are in vivo and in vitro studies for clinical sepsis? A mathematical model of LPS signaling based on endotoxin tolerance.
    Vasilescu C, Olteanu M, Flondor P.
    Chirurgia (Bucur); 2009 Jun 07; 104(2):195-201. PubMed ID: 19499663
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  • 17. Antimalarial artesunate protects sepsis model mice against heat-killed Escherichia coli challenge by decreasing TLR4, TLR9 mRNA expressions and transcription factor NF-kappa B activation.
    Li B, Zhang R, Li J, Zhang L, Ding G, Luo P, He S, Dong Y, Jiang W, Lu Y, Cao H, Zheng J, Zhou H.
    Int Immunopharmacol; 2008 Mar 07; 8(3):379-89. PubMed ID: 18279792
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  • 18. Endotoxin tolerance attenuates LPS-induced TLR4 mobilization to lipid rafts: a condition reversed by PKC activation.
    Cuschieri J, Billigren J, Maier RV.
    J Leukoc Biol; 2006 Dec 07; 80(6):1289-97. PubMed ID: 16959900
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  • 19. Endotoxin potentiation of trichothecene-induced lymphocyte apoptosis is mediated by up-regulation of glucocorticoids.
    Islam Z, Moon YS, Zhou HR, King LE, Fraker PJ, Pestka JJ.
    Toxicol Appl Pharmacol; 2002 Apr 01; 180(1):43-55. PubMed ID: 11922776
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  • 20. Endotoxin tolerance in sepsis: concentration-dependent augmentation or inhibition of LPS-stimulated macrophage TNF secretion by LPS pretreatment.
    West MA, Koons A.
    J Trauma; 2008 Oct 01; 65(4):893-8; discussion 898-900. PubMed ID: 18849808
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