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Journal Abstract Search

110 related items for PubMed ID: 15034483

  • 1. [Physiopathology of ALS: therapeutic approach].
    Bruneteau G, Demeret S, Meininger V.
    Rev Neurol (Paris); 2004 Feb; 160(2):235-41. PubMed ID: 15034483
    [Abstract] [Full Text] [Related]

  • 2. Mitochondria in amyotrophic lateral sclerosis: a trigger and a target.
    Dupuis L, Gonzalez de Aguilar JL, Oudart H, de Tapia M, Barbeito L, Loeffler JP.
    Neurodegener Dis; 2004 Feb; 1(6):245-54. PubMed ID: 16908975
    [Abstract] [Full Text] [Related]

  • 3. Current hypotheses for the underlying biology of amyotrophic lateral sclerosis.
    Rothstein JD.
    Ann Neurol; 2009 Jan; 65 Suppl 1():S3-9. PubMed ID: 19191304
    [Abstract] [Full Text] [Related]

  • 4. Transgenic mouse models of amyotrophic lateral sclerosis.
    Julien JP, Kriz J.
    Biochim Biophys Acta; 2006 Jan; 1762(11-12):1013-24. PubMed ID: 16675207
    [Abstract] [Full Text] [Related]

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  • 6. Pathophysiology of neurodegeneration in familial amyotrophic lateral sclerosis.
    Vucic S, Kiernan MC.
    Curr Mol Med; 2009 Apr; 9(3):255-72. PubMed ID: 19355908
    [Abstract] [Full Text] [Related]

  • 7. Managing amyotrophic lateral sclerosis: slowing disease progression and improving patient quality of life.
    Brooks BR.
    Ann Neurol; 2009 Jan; 65 Suppl 1():S17-23. PubMed ID: 19191306
    [Abstract] [Full Text] [Related]

  • 8. Mitochondrial dysfunction and its role in motor neuron degeneration in ALS.
    Manfredi G, Xu Z.
    Mitochondrion; 2005 Apr; 5(2):77-87. PubMed ID: 16050975
    [Abstract] [Full Text] [Related]

  • 9. Neurofilaments and orthograde transport are reduced in ventral root axons of transgenic mice that express human SOD1 with a G93A mutation.
    Zhang B, Tu P, Abtahian F, Trojanowski JQ, Lee VM.
    J Cell Biol; 1997 Dec 01; 139(5):1307-15. PubMed ID: 9382875
    [Abstract] [Full Text] [Related]

  • 10. GLT1 overexpression in SOD1(G93A) mouse cervical spinal cord does not preserve diaphragm function or extend disease.
    Li K, Hala TJ, Seetharam S, Poulsen DJ, Wright MC, Lepore AC.
    Neurobiol Dis; 2015 Jun 01; 78():12-23. PubMed ID: 25818008
    [Abstract] [Full Text] [Related]

  • 11. Human superoxide dismutase 1 overexpression in motor neurons of Caenorhabditis elegans causes axon guidance defect and neurodegeneration.
    Li J, Li T, Zhang X, Tang Y, Yang J, Le W.
    Neurobiol Aging; 2014 Apr 01; 35(4):837-46. PubMed ID: 24126158
    [Abstract] [Full Text] [Related]

  • 12. Oxidative stress in ALS: a mechanism of neurodegeneration and a therapeutic target.
    Barber SC, Mead RJ, Shaw PJ.
    Biochim Biophys Acta; 2006 Apr 01; 1762(11-12):1051-67. PubMed ID: 16713195
    [Abstract] [Full Text] [Related]

  • 13. Excitotoxicity and amyotrophic lateral sclerosis.
    Van Damme P, Dewil M, Robberecht W, Van Den Bosch L.
    Neurodegener Dis; 2005 Apr 01; 2(3-4):147-59. PubMed ID: 16909020
    [Abstract] [Full Text] [Related]

  • 14. Knocking down metabotropic glutamate receptor 1 improves survival and disease progression in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
    Milanese M, Giribaldi F, Melone M, Bonifacino T, Musante I, Carminati E, Rossi PI, Vergani L, Voci A, Conti F, Puliti A, Bonanno G.
    Neurobiol Dis; 2014 Apr 01; 64():48-59. PubMed ID: 24361555
    [Abstract] [Full Text] [Related]

  • 15. Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1.
    Jaarsma D, Haasdijk ED, Grashorn JA, Hawkins R, van Duijn W, Verspaget HW, London J, Holstege JC.
    Neurobiol Dis; 2000 Dec 01; 7(6 Pt B):623-43. PubMed ID: 11114261
    [Abstract] [Full Text] [Related]

  • 16. Recent advances in amyotrophic lateral sclerosis research.
    Przedborski S, Mitsumoto H, Rowland LP.
    Curr Neurol Neurosci Rep; 2003 Jan 01; 3(1):70-7. PubMed ID: 12507415
    [Abstract] [Full Text] [Related]

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  • 18. Early abnormalities in transgenic mouse models of amyotrophic lateral sclerosis.
    Durand J, Amendola J, Bories C, Lamotte d'Incamps B.
    J Physiol Paris; 2006 Jan 01; 99(2-3):211-20. PubMed ID: 16448809
    [Abstract] [Full Text] [Related]

  • 19. Overexpression of metallothionein-I, a copper-regulating protein, attenuates intracellular copper dyshomeostasis and extends lifespan in a mouse model of amyotrophic lateral sclerosis caused by mutant superoxide dismutase-1.
    Tokuda E, Okawa E, Watanabe S, Ono S.
    Hum Mol Genet; 2014 Mar 01; 23(5):1271-85. PubMed ID: 24163136
    [Abstract] [Full Text] [Related]

  • 20. Mechanism and treatment of motoneuron degeneration in ALS: what have SOD1 mutants told us?
    Xu Z.
    Amyotroph Lateral Scler Other Motor Neuron Disord; 2000 Sep 01; 1(4):225-34. PubMed ID: 11465016
    [Abstract] [Full Text] [Related]

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