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Journal Abstract Search


528 related items for PubMed ID: 15047810

  • 1. Identification of Epstein-Barr virus (EBV) nuclear antigen 2 (EBNA2) target proteins by proteome analysis: activation of EBNA2 in conditionally immortalized B cells reflects early events after infection of primary B cells by EBV.
    Schlee M, Krug T, Gires O, Zeidler R, Hammerschmidt W, Mailhammer R, Laux G, Sauer G, Lovric J, Bornkamm GW.
    J Virol; 2004 Apr; 78(8):3941-52. PubMed ID: 15047810
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  • 2. Notch1IC partially replaces EBNA2 function in B cells immortalized by Epstein-Barr virus.
    Gordadze AV, Peng R, Tan J, Liu G, Sutton R, Kempkes B, Bornkamm GW, Ling PD.
    J Virol; 2001 Jul; 75(13):5899-912. PubMed ID: 11390591
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  • 3. c-Myc and Rel/NF-kappaB are the two master transcriptional systems activated in the latency III program of Epstein-Barr virus-immortalized B cells.
    Faumont N, Durand-Panteix S, Schlee M, Grömminger S, Schuhmacher M, Hölzel M, Laux G, Mailhammer R, Rosenwald A, Staudt LM, Bornkamm GW, Feuillard J.
    J Virol; 2009 May; 83(10):5014-27. PubMed ID: 19264782
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  • 6. c-Myc Represses Transcription of Epstein-Barr Virus Latent Membrane Protein 1 Early after Primary B Cell Infection.
    Price AM, Messinger JE, Luftig MA.
    J Virol; 2018 Jan 15; 92(2):. PubMed ID: 29118124
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  • 9. c-myc activation renders proliferation of Epstein-Barr virus (EBV)-transformed cells independent of EBV nuclear antigen 2 and latent membrane protein 1.
    Polack A, Hörtnagel K, Pajic A, Christoph B, Baier B, Falk M, Mautner J, Geltinger C, Bornkamm GW, Kempkes B.
    Proc Natl Acad Sci U S A; 1996 Sep 17; 93(19):10411-6. PubMed ID: 8816814
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  • 10. The Epstein-Barr virus EBNA-LP protein preferentially coactivates EBNA2-mediated stimulation of latent membrane proteins expressed from the viral divergent promoter.
    Peng R, Moses SC, Tan J, Kremmer E, Ling PD.
    J Virol; 2005 Apr 17; 79(7):4492-505. PubMed ID: 15767449
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  • 11. Determining the role of the Epstein-Barr virus Cp EBNA2-dependent enhancer during the establishment of latency by using mutant and wild-type viruses recovered from cottontop marmoset lymphoblastoid cell lines.
    Yoo L, Speck SH.
    J Virol; 2000 Dec 17; 74(23):11115-20. PubMed ID: 11070007
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  • 13. Inhibition of Epstein-Barr virus-induced growth proliferation by a nuclear antigen EBNA2-TAT peptide.
    Farrell CJ, Lee JM, Shin EC, Cebrat M, Cole PA, Hayward SD.
    Proc Natl Acad Sci U S A; 2004 Mar 30; 101(13):4625-30. PubMed ID: 15070768
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  • 16. EBNA2-deleted Epstein-Barr virus (EBV) isolate, P3HR1, causes Hodgkin-like lymphomas and diffuse large B cell lymphomas with type II and Wp-restricted latency types in humanized mice.
    Li C, Romero-Masters JC, Huebner S, Ohashi M, Hayes M, Bristol JA, Nelson SE, Eichelberg MR, Van Sciver N, Ranheim EA, Scott RS, Johannsen EC, Kenney SC.
    PLoS Pathog; 2020 Jun 30; 16(6):e1008590. PubMed ID: 32542010
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  • 17. Species-specific functions of Epstein-Barr virus nuclear antigen 2 (EBNA2) reveal dual roles for initiation and maintenance of B cell immortalization.
    Mühe J, Wang F.
    PLoS Pathog; 2017 Dec 30; 13(12):e1006772. PubMed ID: 29261800
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  • 19. Modulation of cellular gene expression in B lymphoma cells following in vitro infection by Epstein-Barr virus (EBV).
    Calender A, Cordier M, Billaud M, Lenoir GM.
    Int J Cancer; 1990 Oct 15; 46(4):658-63. PubMed ID: 1698730
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