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Journal Abstract Search

200 related items for PubMed ID: 15069015

  • 1. G-CSF receptor truncations found in SCN/AML relieve SOCS3-controlled inhibition of STAT5 but leave suppression of STAT3 intact.
    van de Geijn GJ, Gits J, Aarts LH, Heijmans-Antonissen C, Touw IP.
    Blood; 2004 Aug 01; 104(3):667-74. PubMed ID: 15069015
    [Abstract] [Full Text] [Related]

  • 2. Loss of SHIP and CIS recruitment to the granulocyte colony-stimulating factor receptor contribute to hyperproliferative responses in severe congenital neutropenia/acute myelogenous leukemia.
    Hunter MG, Jacob A, O'donnell LC, Agler A, Druhan LJ, Coggeshall KM, Avalos BR.
    J Immunol; 2004 Oct 15; 173(8):5036-45. PubMed ID: 15470047
    [Abstract] [Full Text] [Related]

  • 3. Tyrosine 729 of the G-CSF receptor controls the duration of receptor signaling: involvement of SOCS3 and SOCS1.
    Zhuang D, Qiu Y, Haque SJ, Dong F.
    J Leukoc Biol; 2005 Oct 15; 78(4):1008-15. PubMed ID: 16033816
    [Abstract] [Full Text] [Related]

  • 4. Sustained receptor activation and hyperproliferation in response to granulocyte colony-stimulating factor (G-CSF) in mice with a severe congenital neutropenia/acute myeloid leukemia-derived mutation in the G-CSF receptor gene.
    Hermans MH, Antonissen C, Ward AC, Mayen AE, Ploemacher RE, Touw IP.
    J Exp Med; 1999 Feb 15; 189(4):683-92. PubMed ID: 9989983
    [Abstract] [Full Text] [Related]

  • 5. Distinct activities of suppressor of cytokine signaling (SOCS) proteins and involvement of the SOCS box in controlling G-CSF signaling.
    van de Geijn GJ, Gits J, Touw IP.
    J Leukoc Biol; 2004 Jul 15; 76(1):237-44. PubMed ID: 15107455
    [Abstract] [Full Text] [Related]

  • 6. Defective internalization and sustained activation of truncated granulocyte colony-stimulating factor receptor found in severe congenital neutropenia/acute myeloid leukemia.
    Ward AC, van Aesch YM, Schelen AM, Touw IP.
    Blood; 1999 Jan 15; 93(2):447-58. PubMed ID: 9885206
    [Abstract] [Full Text] [Related]

  • 7. The carboxyl terminus of the granulocyte colony-stimulating factor receptor, truncated in patients with severe congenital neutropenia/acute myeloid leukemia, is required for SH2-containing phosphatase-1 suppression of Stat activation.
    Dong F, Qiu Y, Yi T, Touw IP, Larner AC.
    J Immunol; 2001 Dec 01; 167(11):6447-52. PubMed ID: 11714811
    [Abstract] [Full Text] [Related]

  • 8. Signaling mechanisms coupled to tyrosines in the granulocyte colony-stimulating factor receptor orchestrate G-CSF-induced expansion of myeloid progenitor cells.
    Hermans MH, van de Geijn GJ, Antonissen C, Gits J, van Leeuwen D, Ward AC, Touw IP.
    Blood; 2003 Apr 01; 101(7):2584-90. PubMed ID: 12468431
    [Abstract] [Full Text] [Related]

  • 9. The G-CSF receptor carboxyl terminus, truncated in AML/SCN, is required for induction of a Stat5 protease activity.
    Qiu Y, Zhuang D, MacRae A, Dong F.
    Leuk Res; 2005 Oct 01; 29(10):1153-62. PubMed ID: 16111533
    [Abstract] [Full Text] [Related]

  • 10. Tyrosine-dependent and -independent mechanisms of STAT3 activation by the human granulocyte colony-stimulating factor (G-CSF) receptor are differentially utilized depending on G-CSF concentration.
    Ward AC, Hermans MH, Smith L, van Aesch YM, Schelen AM, Antonissen C, Touw IP.
    Blood; 1999 Jan 01; 93(1):113-24. PubMed ID: 9864153
    [Abstract] [Full Text] [Related]

  • 11. SOCS3 is a physiological negative regulator for granulopoiesis and granulocyte colony-stimulating factor receptor signaling.
    Kimura A, Kinjyo I, Matsumura Y, Mori H, Mashima R, Harada M, Chien KR, Yasukawa H, Yoshimura A.
    J Biol Chem; 2004 Feb 20; 279(8):6905-10. PubMed ID: 14699146
    [Abstract] [Full Text] [Related]

  • 12. Multiple signaling pathways induced by granulocyte colony-stimulating factor involving activation of JAKs, STAT5, and/or STAT3 are required for regulation of three distinct classes of immediate early genes.
    Tian SS, Tapley P, Sincich C, Stein RB, Rosen J, Lamb P.
    Blood; 1996 Dec 15; 88(12):4435-44. PubMed ID: 8977235
    [Abstract] [Full Text] [Related]

  • 13. The membrane-distal cytoplasmic region of human granulocyte colony-stimulating factor receptor is required for STAT3 but not STAT1 homodimer formation.
    de Koning JP, Dong F, Smith L, Schelen AM, Barge RM, van der Plas DC, Hoefsloot LH, Löwenberg B, Touw IP.
    Blood; 1996 Feb 15; 87(4):1335-42. PubMed ID: 8608222
    [Abstract] [Full Text] [Related]

  • 14. Stimulation of Stat5 by granulocyte colony-stimulating factor (G-CSF) is modulated by two distinct cytoplasmic regions of the G-CSF receptor.
    Dong F, Liu X, de Koning JP, Touw IP, Hennighausen L, Larner A, Grimley PM.
    J Immunol; 1998 Dec 15; 161(12):6503-9. PubMed ID: 9862674
    [Abstract] [Full Text] [Related]

  • 15. Delineation and mapping of Stat5 isoforms activated by granulocyte colony-stimulating factor in myeloid cells.
    Chakraborty A, Dyer KF, Tweardy DJ.
    Blood Cells Mol Dis; 2000 Aug 15; 26(4):320-30. PubMed ID: 11042034
    [Abstract] [Full Text] [Related]

  • 16. The SOCS box of suppressor of cytokine signaling-3 contributes to the control of G-CSF responsiveness in vivo.
    Boyle K, Egan P, Rakar S, Willson TA, Wicks IP, Metcalf D, Hilton DJ, Nicola NA, Alexander WS, Roberts AW, Robb L.
    Blood; 2007 Sep 01; 110(5):1466-74. PubMed ID: 17510322
    [Abstract] [Full Text] [Related]

  • 17. Pim-1 kinase inhibits STAT5-dependent transcription via its interactions with SOCS1 and SOCS3.
    Peltola KJ, Paukku K, Aho TL, Ruuska M, Silvennoinen O, Koskinen PJ.
    Blood; 2004 May 15; 103(10):3744-50. PubMed ID: 14764533
    [Abstract] [Full Text] [Related]

  • 18. Combined corticosteroid/granulocyte colony-stimulating factor (G-CSF) therapy in the treatment of severe congenital neutropenia unresponsive to G-CSF: Activated glucocorticoid receptors synergize with G-CSF signals.
    Dror Y, Ward AC, Touw IP, Freedman MH.
    Exp Hematol; 2000 Dec 15; 28(12):1381-9. PubMed ID: 11146160
    [Abstract] [Full Text] [Related]

  • 19. Interleukin-6 inhibits hepatic growth hormone signaling via upregulation of Cis and Socs-3.
    Denson LA, Held MA, Menon RK, Frank SJ, Parlow AF, Arnold DL.
    Am J Physiol Gastrointest Liver Physiol; 2003 Apr 15; 284(4):G646-54. PubMed ID: 12519742
    [Abstract] [Full Text] [Related]

  • 20. G-CSF receptor mutations in patients with severe congenital neutropenia do not abrogate Jak2 activation and stat1/stat3 translocation.
    Herbst A, Koester M, Wirth D, Hauser H, Welte K.
    Ann N Y Acad Sci; 1999 Apr 30; 872():320-5; discussion 325-7. PubMed ID: 10372134
    [Abstract] [Full Text] [Related]

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