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269 related items for PubMed ID: 15081310

  • 1. Selective upregulation of cardiac brain natriuretic peptide at the transcriptional and translational levels by pro-inflammatory cytokines and by conditioned medium derived from mixed lymphocyte reactions via p38 MAP kinase.
    Ma KK, Ogawa T, de Bold AJ.
    J Mol Cell Cardiol; 2004 Apr; 36(4):505-13. PubMed ID: 15081310
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  • 2. Relationship between natriuretic peptides and inflammation: proteomic evidence obtained during acute cellular cardiac allograft rejection in humans.
    Meirovich YF, Veinot JP, de Bold ML, Haddad H, Davies RA, Masters RG, Hendry PJ, de Bold AJ.
    J Heart Lung Transplant; 2008 Jan; 27(1):31-7. PubMed ID: 18187084
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  • 3. Phorbol ester stimulates the synthesis and secretion of brain natriuretic peptide from neonatal rat ventricular cardiocytes: a comparison with the regulation of atrial natriuretic factor.
    LaPointe MC, Sitkins JR.
    Mol Endocrinol; 1993 Oct; 7(10):1284-96. PubMed ID: 8264660
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  • 4. Uncoordinated regulation of atrial natriuretic factor and brain natriuretic peptide in lipopolysaccharide-treated rats.
    Ogawa T, de Bold AJ.
    Biomarkers; 2012 Mar; 17(2):140-9. PubMed ID: 22224641
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  • 5. Transcriptional activation of the BNP gene by lipopolysaccharide is mediated through GATA elements in neonatal rat cardiac myocytes.
    Tomaru Ki K, Arai M, Yokoyama T, Aihara Y, Sekiguchi Ki K, Tanaka T, Nagai R, Kurabayashi M.
    J Mol Cell Cardiol; 2002 Jun; 34(6):649-59. PubMed ID: 12054852
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  • 7. Induction of VEGF gene transcription by IL-1 beta is mediated through stress-activated MAP kinases and Sp1 sites in cardiac myocytes.
    Tanaka T, Kanai H, Sekiguchi K, Aihara Y, Yokoyama T, Arai M, Kanda T, Nagai R, Kurabayashi M.
    J Mol Cell Cardiol; 2000 Nov; 32(11):1955-67. PubMed ID: 11040101
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  • 11. Pro-inflammatory cytokines stimulate mitogen-activated protein kinase subfamilies, increase phosphorylation of c-Jun and ATF2 and upregulate c-Jun protein in neonatal rat ventricular myocytes.
    Clerk A, Harrison JG, Long CS, Sugden PH.
    J Mol Cell Cardiol; 1999 Dec; 31(12):2087-99. PubMed ID: 10640438
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  • 12. Mechanical strain activates BNP gene transcription through a p38/NF-kappaB-dependent mechanism.
    Liang F, Gardner DG.
    J Clin Invest; 1999 Dec; 104(11):1603-12. PubMed ID: 10587524
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  • 13. P38 mitogen-activated protein kinase inhibition attenuates pulmonary inflammatory response in a rat cardiopulmonary bypass model.
    Dong X, Liu Y, Du M, Wang Q, Yu CT, Fan X.
    Eur J Cardiothorac Surg; 2006 Jul; 30(1):77-84. PubMed ID: 16723252
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  • 16. Predominant activation of MAP kinases and pro-destructive/pro-inflammatory features by TNF alpha in early-passage synovial fibroblasts via TNF receptor-1: failure of p38 inhibition to suppress matrix metalloproteinase-1 in rheumatoid arthritis.
    Kunisch E, Gandesiri M, Fuhrmann R, Roth A, Winter R, Kinne RW.
    Ann Rheum Dis; 2007 Aug; 66(8):1043-51. PubMed ID: 17223661
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  • 17. The physiological and pathophysiological modulation of the endocrine function of the heart.
    de Bold AJ, Ma KK, Zhang Y, de Bold ML, Bensimon M, Khoshbaten A.
    Can J Physiol Pharmacol; 2001 Aug; 79(8):705-14. PubMed ID: 11558679
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  • 18. M-CAT element mediates mechanical stretch-activated transcription of B-type natriuretic peptide via ERK activation.
    Koivisto E, Karkkola L, Majalahti T, Aro J, Tokola H, Kerkelä R, Ruskoaho H.
    Can J Physiol Pharmacol; 2011 Aug; 89(8):539-50. PubMed ID: 21812548
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  • 19. Interleukin-18 is a pro-hypertrophic cytokine that acts through a phosphatidylinositol 3-kinase-phosphoinositide-dependent kinase-1-Akt-GATA4 signaling pathway in cardiomyocytes.
    Chandrasekar B, Mummidi S, Claycomb WC, Mestril R, Nemer M.
    J Biol Chem; 2005 Feb 11; 280(6):4553-67. PubMed ID: 15574430
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