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Journal Abstract Search

367 related items for PubMed ID: 15096042

  • 1. Lys691 and Asp714 of the Na+/K+-ATPase alpha subunit are essential for phosphorylation, dephosphorylation, and enzyme turnover.
    Su P, Scheiner-Bobis G.
    Biochemistry; 2004 Apr 27; 43(16):4731-40. PubMed ID: 15096042
    [Abstract] [Full Text] [Related]

  • 2. Palytoxin-induced channel formation within the Na+/K+-ATPase does not require a catalytically active enzyme.
    Scheiner-Bobis G, Schneider H.
    Eur J Biochem; 1997 Sep 15; 248(3):717-23. PubMed ID: 9342222
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  • 3. Glutamic acid 472 and lysine 480 of the sodium pump alpha 1 subunit are essential for activity. Their conservation in pyrophosphatases suggests their involvement in recognition of ATP phosphates.
    Scheiner-Bobis G, Schreiber S.
    Biochemistry; 1999 Jul 20; 38(29):9198-208. PubMed ID: 10413494
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  • 4. Palytoxin-induced Na+ influx into yeast cells expressing the mammalian sodium pump is due to the formation of a channel within the enzyme.
    Redondo J, Fiedler B, Scheiner-Bobis G.
    Mol Pharmacol; 1996 Jan 20; 49(1):49-57. PubMed ID: 8569711
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  • 5. Amino acid replacement of Asp369 in the sheep alpha 1 isoform eliminates ATP and phosphate stimulation of [3H]ouabain binding to the Na+, K(+)-ATPase without altering the cation binding properties of the enzyme.
    Kuntzweiler TA, Wallick ET, Johnson CL, Lingrel JB.
    J Biol Chem; 1995 Jul 07; 270(27):16206-12. PubMed ID: 7608186
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  • 6. Comparison of the effects of potassium on ouabain binding to native and site-directed mutants of Na,K-ATPase.
    Johnson CL, Schultheis PJ, Lingrel JB, Johnson CG, Wallick ET.
    Arch Biochem Biophys; 1995 Feb 20; 317(1):133-41. PubMed ID: 7872775
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  • 7. Palytoxin induces K+ efflux from yeast cells expressing the mammalian sodium pump.
    Scheiner-Bobis G, Meyer zu Heringdorf D, Christ M, Habermann E.
    Mol Pharmacol; 1994 Jun 20; 45(6):1132-6. PubMed ID: 7912814
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  • 9. Consequences of mutations to the phosphorylation site of the alpha-subunit of Na, K-ATPase for ATP binding and E1-E2 conformational equilibrium.
    Pedersen PA, Rasmussen JH, Jørgensen PL.
    Biochemistry; 1996 Dec 17; 35(50):16085-93. PubMed ID: 8973179
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  • 13. Glutamic acid 327 in the sheep alpha 1 isoform of Na+,K(+)-ATPase stabilizes a K(+)-induced conformational change.
    Kuntzweiler TA, Wallick ET, Johnson CL, Lingrel JB.
    J Biol Chem; 1995 Feb 17; 270(7):2993-3000. PubMed ID: 7852379
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  • 14. Ouabain affinity determining residues lie close to the Na/K pump ion pathway.
    Artigas P, Gadsby DC.
    Proc Natl Acad Sci U S A; 2006 Aug 15; 103(33):12613-8. PubMed ID: 16894161
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  • 15. Site-directed mutagenesis of amino acids in the cytoplasmic loop 6/7 of Na,K-ATPase.
    Xu G, Farley RA, Kane DJ, Faller LD.
    Ann N Y Acad Sci; 2003 Apr 15; 986():96-100. PubMed ID: 12763780
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  • 16. 18O-exchange evidence that mutations of arginine in a signature sequence for P-type pumps affect inorganic phosphate binding.
    Farley RA, Elquza E, Müller-Ehmsen J, Kane DJ, Nagy AK, Kasho VN, Faller LD.
    Biochemistry; 2001 May 29; 40(21):6361-70. PubMed ID: 11371198
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  • 19. D443 of the N domain of Na+,K+-ATPase interacts with the ATP-Mg2+ complex, possibly via a second Mg2+ ion.
    Strugatsky D, Gottschalk KE, Goldshleger R, Karlish SJ.
    Biochemistry; 2005 Dec 13; 44(49):15961-9. PubMed ID: 16331955
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  • 20. Significance of the glutamic acid residues Glu334, Glu959, and Glu960 of the alpha subunits of Torpedo Na+, K+ pumps for transport activity and ouabain binding.
    Vasilets LA, Takeda K, Kawamura M, Schwarz W.
    Biochim Biophys Acta; 1998 Jan 05; 1368(1):137-49. PubMed ID: 9459592
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