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205 related items for PubMed ID: 15126344

  • 1. Role of the retinoblastoma pathway in senescence triggered by repression of the human papillomavirus E7 protein in cervical carcinoma cells.
    Psyrri A, DeFilippis RA, Edwards AP, Yates KE, Manuelidis L, DiMaio D.
    Cancer Res; 2004 May 01; 64(9):3079-86. PubMed ID: 15126344
    [Abstract] [Full Text] [Related]

  • 2. Human papillomavirus E7 repression in cervical carcinoma cells initiates a transcriptional cascade driven by the retinoblastoma family, resulting in senescence.
    Johung K, Goodwin EC, DiMaio D.
    J Virol; 2007 Mar 01; 81(5):2102-16. PubMed ID: 17182682
    [Abstract] [Full Text] [Related]

  • 3. Repression of human papillomavirus oncogenes in HeLa cervical carcinoma cells causes the orderly reactivation of dormant tumor suppressor pathways.
    Goodwin EC, DiMaio D.
    Proc Natl Acad Sci U S A; 2000 Nov 07; 97(23):12513-8. PubMed ID: 11070078
    [Abstract] [Full Text] [Related]

  • 4. Effect of BPV1 E2-mediated inhibition of E6/E7 expression in HPV16-positive cervical carcinoma cells.
    Moon MS, Lee CJ, Um SJ, Park JS, Yang JM, Hwang ES.
    Gynecol Oncol; 2001 Feb 07; 80(2):168-75. PubMed ID: 11161855
    [Abstract] [Full Text] [Related]

  • 5. Suppression of tumorigenesis by transcription units expressing the antisense E6 and E7 messenger RNA (mRNA) for the transforming proteins of the human papilloma virus and the sense mRNA for the retinoblastoma gene in cervical carcinoma cells.
    Hu G, Liu W, Hanania EG, Fu S, Wang T, Deisseroth AB.
    Cancer Gene Ther; 1995 Mar 07; 2(1):19-32. PubMed ID: 7621252
    [Abstract] [Full Text] [Related]

  • 6. Repression of the human papillomavirus E6 gene initiates p53-dependent, telomerase-independent senescence and apoptosis in HeLa cervical carcinoma cells.
    Horner SM, DeFilippis RA, Manuelidis L, DiMaio D.
    J Virol; 2004 Apr 07; 78(8):4063-73. PubMed ID: 15047823
    [Abstract] [Full Text] [Related]

  • 7. E2F-Rb complexes assemble and inhibit cdc25A transcription in cervical carcinoma cells following repression of human papillomavirus oncogene expression.
    Wu L, Goodwin EC, Naeger LK, Vigo E, Galaktionov K, Helin K, DiMaio D.
    Mol Cell Biol; 2000 Oct 07; 20(19):7059-67. PubMed ID: 10982822
    [Abstract] [Full Text] [Related]

  • 8. Endogenous human papillomavirus E6 and E7 proteins differentially regulate proliferation, senescence, and apoptosis in HeLa cervical carcinoma cells.
    DeFilippis RA, Goodwin EC, Wu L, DiMaio D.
    J Virol; 2003 Jan 07; 77(2):1551-63. PubMed ID: 12502868
    [Abstract] [Full Text] [Related]

  • 9. Induction of senescence-like state and suppression of telomerase activity through inhibition of HPV E6/E7 gene expression in cells immortalized by HPV16 DNA.
    Lee CJ, Suh EJ, Kang HT, Im JS, Um SJ, Park JS, Hwang ES.
    Exp Cell Res; 2002 Jul 15; 277(2):173-82. PubMed ID: 12083799
    [Abstract] [Full Text] [Related]

  • 10. Antisense targeting human papillomavirus type 16 E6 and E7 genes contributes to apoptosis and senescence in SiHa cervical carcinoma cells.
    Sima N, Wang S, Wang W, Kong D, Xu Q, Tian X, Luo A, Zhou J, Xu G, Meng L, Lu Y, Ma D.
    Gynecol Oncol; 2007 Aug 15; 106(2):299-304. PubMed ID: 17586029
    [Abstract] [Full Text] [Related]

  • 11. Papillomavirus E2 induces senescence in HPV-positive cells via pRB- and p21(CIP)-dependent pathways.
    Wells SI, Francis DA, Karpova AY, Dowhanick JJ, Benson JD, Howley PM.
    EMBO J; 2000 Nov 01; 19(21):5762-71. PubMed ID: 11060027
    [Abstract] [Full Text] [Related]

  • 12. CRISPR/Cas9-based inactivation of human papillomavirus oncogenes E6 or E7 induces senescence in cervical cancer cells.
    Inturi R, Jemth P.
    Virology; 2021 Oct 01; 562():92-102. PubMed ID: 34280810
    [Abstract] [Full Text] [Related]

  • 13. A novel MLL5 isoform that is essential to activate E6 and E7 transcription in HPV16/18-associated cervical cancers.
    Yew CW, Lee P, Chan WK, Lim VK, Tay SK, Tan TM, Deng LW.
    Cancer Res; 2011 Nov 01; 71(21):6696-707. PubMed ID: 21908553
    [Abstract] [Full Text] [Related]

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  • 15. The role of HPV oncoproteins and cellular factors in maintenance of hTERT expression in cervical carcinoma cells.
    Jeong Seo E, Jung Kim H, Jae Lee C, Tae Kang H, Seong Hwang E.
    Gynecol Oncol; 2004 Jul 01; 94(1):40-7. PubMed ID: 15262117
    [Abstract] [Full Text] [Related]

  • 16. Primary human cervical carcinoma cells require human papillomavirus E6 and E7 expression for ongoing proliferation.
    Magaldi TG, Almstead LL, Bellone S, Prevatt EG, Santin AD, DiMaio D.
    Virology; 2012 Jan 05; 422(1):114-24. PubMed ID: 22056390
    [Abstract] [Full Text] [Related]

  • 17. Induction of dormancy in hypoxic human papillomavirus-positive cancer cells.
    Hoppe-Seyler K, Bossler F, Lohrey C, Bulkescher J, Rösl F, Jansen L, Mayer A, Vaupel P, Dürst M, Hoppe-Seyler F.
    Proc Natl Acad Sci U S A; 2017 Feb 07; 114(6):E990-E998. PubMed ID: 28115701
    [Abstract] [Full Text] [Related]

  • 18. Activation of the retinoblastoma tumor suppressor mediates cell cycle inhibition and cell death in specific cervical cancer cell lines.
    Bourgo RJ, Braden WA, Wells SI, Knudsen ES.
    Mol Carcinog; 2009 Jan 07; 48(1):45-55. PubMed ID: 18506774
    [Abstract] [Full Text] [Related]

  • 19. Reversible repression of papillomavirus oncogene expression in cervical carcinoma cells: consequences for the phenotype and E6-p53 and E7-pRB interactions.
    von Knebel Doeberitz M, Rittmüller C, Aengeneyndt F, Jansen-Dürr P, Spitkovsky D.
    J Virol; 1994 May 07; 68(5):2811-21. PubMed ID: 8151752
    [Abstract] [Full Text] [Related]

  • 20. Repression of the integrated papillomavirus E6/E7 promoter is required for growth suppression of cervical cancer cells.
    Francis DA, Schmid SI, Howley PM.
    J Virol; 2000 Mar 07; 74(6):2679-86. PubMed ID: 10684283
    [Abstract] [Full Text] [Related]

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