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399 related items for PubMed ID: 15137061

  • 1. BRD7, a novel bromodomain gene, inhibits G1-S progression by transcriptionally regulating some important molecules involved in ras/MEK/ERK and Rb/E2F pathways.
    Zhou J, Ma J, Zhang BC, Li XL, Shen SR, Zhu SG, Xiong W, Liu HY, Huang H, Zhou M, Li GY.
    J Cell Physiol; 2004 Jul; 200(1):89-98. PubMed ID: 15137061
    [Abstract] [Full Text] [Related]

  • 2. Identification of nuclear localization signal that governs nuclear import of BRD7 and its essential roles in inhibiting cell cycle progression.
    Zhou M, Liu H, Xu X, Zhou H, Li X, Peng C, Shen S, Xiong W, Ma J, Zeng Z, Fang S, Nie X, Yang Y, Zhou J, Xiang J, Cao L, Peng S, Li S, Li G.
    J Cell Biochem; 2006 Jul 01; 98(4):920-30. PubMed ID: 16475162
    [Abstract] [Full Text] [Related]

  • 3. The transcriptional regulation role of BRD7 by binding to acetylated histone through bromodomain.
    Peng C, Zhou J, Liu HY, Zhou M, Wang LL, Zhang QH, Yang YX, Xiong W, Shen SR, Li XL, Li GY.
    J Cell Biochem; 2006 Mar 01; 97(4):882-92. PubMed ID: 16265664
    [Abstract] [Full Text] [Related]

  • 4. [Expression of BRD7-interacting proteins,BRD2 and BRD3, in nasopharyngeal carcinoma tissues].
    Zhou M, Peng C, Nie XM, Zhang BC, Zhu SG, Yu Y, Li XL, Li GY.
    Ai Zheng; 2003 Feb 01; 22(2):123-7. PubMed ID: 12600283
    [Abstract] [Full Text] [Related]

  • 5. [Researching a novel NPC-related candidate suppressor gene BRD7 by two-dimensional gel electrophoresis and MALDI-TOF-MS].
    Peng C, Liang SP, Tan C, Lv HB, Huang H, Zhou M, Wang R, Li XL, Li GY.
    Sheng Wu Hua Xue Yu Sheng Wu Wu Li Xue Bao (Shanghai); 2003 Sep 01; 35(9):816-22. PubMed ID: 12958654
    [Abstract] [Full Text] [Related]

  • 6. AP-1 blockade in breast cancer cells causes cell cycle arrest by suppressing G1 cyclin expression and reducing cyclin-dependent kinase activity.
    Liu Y, Lu C, Shen Q, Munoz-Medellin D, Kim H, Brown PH.
    Oncogene; 2004 Oct 28; 23(50):8238-46. PubMed ID: 15378019
    [Abstract] [Full Text] [Related]

  • 7. Identification of novel pRb binding sites using CpG microarrays suggests that E2F recruits pRb to specific genomic sites during S phase.
    Wells J, Yan PS, Cechvala M, Huang T, Farnham PJ.
    Oncogene; 2003 Mar 13; 22(10):1445-60. PubMed ID: 12629508
    [Abstract] [Full Text] [Related]

  • 8. Cloning and characterization of the BRD7 gene promoter.
    Liu H, Peng C, Zhou M, Zhou J, Shen S, Zhou H, Xiong W, Luo X, Peng S, Niu Z, Ouyang J, Li X, Li G.
    DNA Cell Biol; 2006 Jun 13; 25(6):346-58. PubMed ID: 16792505
    [Abstract] [Full Text] [Related]

  • 9. pRB phosphorylation mutants reveal role of pRB in regulating S phase completion by a mechanism independent of E2F.
    Chew YP, Ellis M, Wilkie S, Mittnacht S.
    Oncogene; 1998 Oct 29; 17(17):2177-86. PubMed ID: 9811449
    [Abstract] [Full Text] [Related]

  • 10. E2Fs up-regulate expression of genes involved in DNA replication, DNA repair and mitosis.
    Polager S, Kalma Y, Berkovich E, Ginsberg D.
    Oncogene; 2002 Jan 17; 21(3):437-46. PubMed ID: 11821956
    [Abstract] [Full Text] [Related]

  • 11. Unbiased analysis of RB-mediated transcriptional repression identifies novel targets and distinctions from E2F action.
    Markey MP, Angus SP, Strobeck MW, Williams SL, Gunawardena RW, Aronow BJ, Knudsen ES.
    Cancer Res; 2002 Nov 15; 62(22):6587-97. PubMed ID: 12438254
    [Abstract] [Full Text] [Related]

  • 12. E2F sites in the Op18 promoter are required for high level of expression in the human prostate carcinoma cell line PC-3-M.
    Polzin RG, Benlhabib H, Trepel J, Herrera JE.
    Gene; 2004 Oct 27; 341():209-18. PubMed ID: 15474303
    [Abstract] [Full Text] [Related]

  • 13. BRD7 suppresses the growth of Nasopharyngeal Carcinoma cells (HNE1) through negatively regulating beta-catenin and ERK pathways.
    Peng C, Liu HY, Zhou M, Zhang LM, Li XL, Shen SR, Li GY.
    Mol Cell Biochem; 2007 Sep 27; 303(1-2):141-9. PubMed ID: 17458518
    [Abstract] [Full Text] [Related]

  • 14. Interaction and functional cooperation of the cancer-amplified transcriptional coactivator activating signal cointegrator-2 and E2F-1 in cell proliferation.
    Kong HJ, Yu HJ, Hong S, Park MJ, Choi YH, An WG, Lee JW, Cheong J.
    Mol Cancer Res; 2003 Nov 27; 1(13):948-58. PubMed ID: 14638867
    [Abstract] [Full Text] [Related]

  • 15. Ceap/BLOS2 interacts with BRD7 and selectively inhibits its transcription-suppressing effect on cellular proliferation-associated genes.
    Sun J, Nie J, Hao B, Li L, Xing G, Wang Z, Zhou Y, Sun Q, Li G, Zhang L, He F.
    Cell Signal; 2008 Jun 27; 20(6):1151-8. PubMed ID: 18329849
    [Abstract] [Full Text] [Related]

  • 16. Role of IFI 16, a member of the interferon-inducible p200-protein family, in prostate epithelial cellular senescence.
    Xin H, Curry J, Johnstone RW, Nickoloff BJ, Choubey D.
    Oncogene; 2003 Jul 31; 22(31):4831-40. PubMed ID: 12894224
    [Abstract] [Full Text] [Related]

  • 17. Bone morphogenetic protein-2 induces hypophosphorylation of Rb protein and repression of E2F in androgen-treated LNCaP human prostate cancer cells.
    Tomari K, Kumagai T, Shimizu T, Takeda K.
    Int J Mol Med; 2005 Feb 31; 15(2):253-8. PubMed ID: 15647840
    [Abstract] [Full Text] [Related]

  • 18. Expression analysis using DNA microarrays demonstrates that E2F-1 up-regulates expression of DNA replication genes including replication protein A2.
    Kalma Y, Marash L, Lamed Y, Ginsberg D.
    Oncogene; 2001 Mar 15; 20(11):1379-87. PubMed ID: 11313881
    [Abstract] [Full Text] [Related]

  • 19. The retinoblastoma protein is an essential mediator that links the interferon-inducible 204 gene to cell-cycle regulation.
    Hertel L, Rolle S, De Andrea M, Azzimonti B, Osello R, Gribaudo G, Gariglio M, Landolfo S.
    Oncogene; 2000 Jul 27; 19(32):3598-608. PubMed ID: 10951565
    [Abstract] [Full Text] [Related]

  • 20. The Rb/chromatin connection and epigenetic control: opinion.
    Ferreira R, Naguibneva I, Pritchard LL, Ait-Si-Ali S, Harel-Bellan A.
    Oncogene; 2001 May 28; 20(24):3128-33. PubMed ID: 11420729
    [Abstract] [Full Text] [Related]

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