These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
5. Abnormal synaptic plasticity in the Ts1Cje segmental trisomy 16 mouse model of Down syndrome. Siarey RJ, Villar AJ, Epstein CJ, Galdzicki Z. Neuropharmacology; 2005 Jul; 49(1):122-8. PubMed ID: 15992587 [Abstract] [Full Text] [Related]
11. Towards the understanding of Down syndrome using mouse models. Yamakawa K. Congenit Anom (Kyoto); 2012 Jun; 52(2):67-71. PubMed ID: 22639990 [Abstract] [Full Text] [Related]
17. Duplication of the entire 22.9 Mb human chromosome 21 syntenic region on mouse chromosome 16 causes cardiovascular and gastrointestinal abnormalities. Li Z, Yu T, Morishima M, Pao A, LaDuca J, Conroy J, Nowak N, Matsui S, Shiraishi I, Yu YE. Hum Mol Genet; 2007 Jun 01; 16(11):1359-66. PubMed ID: 17412756 [Abstract] [Full Text] [Related]
18. Gene expression profiling in the adult Down syndrome brain. Lockstone HE, Harris LW, Swatton JE, Wayland MT, Holland AJ, Bahn S. Genomics; 2007 Dec 01; 90(6):647-60. PubMed ID: 17950572 [Abstract] [Full Text] [Related]
20. A chromosome 21 critical region does not cause specific Down syndrome phenotypes. Olson LE, Richtsmeier JT, Leszl J, Reeves RH. Science; 2004 Oct 22; 306(5696):687-90. PubMed ID: 15499018 [Abstract] [Full Text] [Related] Page: [Next] [New Search]