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252 related items for PubMed ID: 15192111

  • 1. Protein kinase B/AKT 1 plays a pivotal role in insulin-like growth factor-1 receptor signaling induced 3T3-L1 adipocyte differentiation.
    Xu J, Liao K.
    J Biol Chem; 2004 Aug 20; 279(34):35914-22. PubMed ID: 15192111
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  • 4. Wnt signaling protects 3T3-L1 preadipocytes from apoptosis through induction of insulin-like growth factors.
    Longo KA, Kennell JA, Ochocinska MJ, Ross SE, Wright WS, MacDougald OA.
    J Biol Chem; 2002 Oct 11; 277(41):38239-44. PubMed ID: 12154096
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  • 5. Phosphatidylinositol-3,4,5-trisphosphate is required for insulin-like growth factor 1-mediated survival of 3T3-L1 preadipocytes.
    Gagnon A, Dods P, Roustan-Delatour N, Chen CS, Sorisky A.
    Endocrinology; 2001 Jan 11; 142(1):205-12. PubMed ID: 11145583
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  • 6. Differential activation of mitogen-activated protein kinase by insulin and epidermal growth factor in 3T3-L1 adipocytes: a possible involvement of PI3-kinase in the activation of the MAP kinase by insulin.
    Suga J, Yoshimasa Y, Yamada K, Yamamoto Y, Inoue G, Okamoto M, Hayashi T, Shigemoto M, Kosaki A, Kuzuya H, Nakao K.
    Diabetes; 1997 May 11; 46(5):735-41. PubMed ID: 9133538
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  • 8. Growth arrest induces primary-cilium formation and sensitizes IGF-1-receptor signaling during differentiation induction of 3T3-L1 preadipocytes.
    Zhu D, Shi S, Wang H, Liao K.
    J Cell Sci; 2009 Aug 01; 122(Pt 15):2760-8. PubMed ID: 19596798
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  • 10. Insulin stimulation of the fatty acid synthase promoter is mediated by the phosphatidylinositol 3-kinase pathway. Involvement of protein kinase B/Akt.
    Wang D, Sul HS.
    J Biol Chem; 1998 Sep 25; 273(39):25420-6. PubMed ID: 9738010
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  • 11. Essential role of insulin-like growth factor I receptor in insulin-induced fetal brown adipocyte differentiation.
    Mur C, Arribas M, Benito M, Valverde AM.
    Endocrinology; 2003 Feb 25; 144(2):581-93. PubMed ID: 12538620
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  • 12. Isoliquiritigenin impairs insulin signaling and adipocyte differentiation through the inhibition of protein-tyrosine phosphatase 1B oxidation in 3T3-L1 preadipocytes.
    Park SJ, Choe YG, Kim JH, Chang KT, Lee HS, Lee DS.
    Food Chem Toxicol; 2016 Jul 25; 93():5-12. PubMed ID: 27117918
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  • 13. On the mechanism for neomycin reversal of wortmannin inhibition of insulin stimulation of glucose uptake.
    Shimaya A, Kovacina KS, Roth RA.
    J Biol Chem; 2004 Dec 31; 279(53):55277-82. PubMed ID: 15504741
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  • 15. Roles of phosphatidylinositol 3-kinase and mitogen-activated protein kinase pathways in stimulation of vascular smooth muscle cell migration and deoxyriboncleic acid synthesis by insulin-like growth factor-I.
    Imai Y, Clemmons DR.
    Endocrinology; 1999 Sep 31; 140(9):4228-35. PubMed ID: 10465296
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  • 16. Insulin-like growth factor (IGF) binding protein-3 potentiation of IGF action is mediated through the phosphatidylinositol-3-kinase pathway and is associated with alteration in protein kinase B/AKT sensitivity.
    Conover CA, Bale LK, Durham SK, Powell DR.
    Endocrinology; 2000 Sep 31; 141(9):3098-103. PubMed ID: 10965879
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  • 17. Activation of the mammalian target of rapamycin pathway acutely inhibits insulin signaling to Akt and glucose transport in 3T3-L1 and human adipocytes.
    Tremblay F, Gagnon A, Veilleux A, Sorisky A, Marette A.
    Endocrinology; 2005 Mar 31; 146(3):1328-37. PubMed ID: 15576463
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  • 19. Expression of a constitutively activated form of protein kinase B (c-Akt) in 3T3-L1 preadipose cells causes spontaneous differentiation.
    Magun R, Burgering BM, Coffer PJ, Pardasani D, Lin Y, Chabot J, Sorisky A.
    Endocrinology; 1996 Aug 31; 137(8):3590-3. PubMed ID: 8754791
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  • 20. Antiapoptotic signalling by the insulin-like growth factor I receptor, phosphatidylinositol 3-kinase, and Akt.
    Kulik G, Klippel A, Weber MJ.
    Mol Cell Biol; 1997 Mar 31; 17(3):1595-606. PubMed ID: 9032287
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