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Journal Abstract Search


58 related items for PubMed ID: 15204019

  • 1. Long term proteasome inhibition does not preferentially afflict motor neurons in organotypical spinal cord cultures.
    Vlug AS, Jaarsma D.
    Amyotroph Lateral Scler Other Motor Neuron Disord; 2004 Mar; 5(1):16-21. PubMed ID: 15204019
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  • 2. Proteasome inhibition induces selective motor neuron death in organotypic slice cultures.
    Tsuji S, Kikuchi S, Shinpo K, Tashiro J, Kishimoto R, Yabe I, Yamagishi S, Takeuchi M, Sasaki H.
    J Neurosci Res; 2005 Nov 15; 82(4):443-51. PubMed ID: 16235246
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  • 3. Sertoli cells improve survival of motor neurons in SOD1 transgenic mice, a model of amyotrophic lateral sclerosis.
    Hemendinger R, Wang J, Malik S, Persinski R, Copeland J, Emerich D, Gores P, Halberstadt C, Rosenfeld J.
    Exp Neurol; 2005 Dec 15; 196(2):235-43. PubMed ID: 16242126
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  • 4. Decrease of Hsp25 protein expression precedes degeneration of motoneurons in ALS-SOD1 mice.
    Maatkamp A, Vlug A, Haasdijk E, Troost D, French PJ, Jaarsma D.
    Eur J Neurosci; 2004 Jul 15; 20(1):14-28. PubMed ID: 15245475
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  • 5. Inducible superoxide dismutase 1 aggregation in transgenic amyotrophic lateral sclerosis mouse fibroblasts.
    Turner BJ, Lopes EC, Cheema SS.
    J Cell Biochem; 2004 Apr 01; 91(5):1074-84. PubMed ID: 15034941
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  • 6. Wild-type nonneuronal cells extend survival of SOD1 mutant motor neurons in ALS mice.
    Clement AM, Nguyen MD, Roberts EA, Garcia ML, Boillée S, Rule M, McMahon AP, Doucette W, Siwek D, Ferrante RJ, Brown RH, Julien JP, Goldstein LS, Cleveland DW.
    Science; 2003 Oct 03; 302(5642):113-7. PubMed ID: 14526083
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  • 7. Histological evidence of protein aggregation in mutant SOD1 transgenic mice and in amyotrophic lateral sclerosis neural tissues.
    Watanabe M, Dykes-Hoberg M, Culotta VC, Price DL, Wong PC, Rothstein JD.
    Neurobiol Dis; 2001 Dec 03; 8(6):933-41. PubMed ID: 11741389
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  • 10. Glutamate AMPA receptors change in motor neurons of SOD1G93A transgenic mice and their inhibition by a noncompetitive antagonist ameliorates the progression of amytrophic lateral sclerosis-like disease.
    Tortarolo M, Grignaschi G, Calvaresi N, Zennaro E, Spaltro G, Colovic M, Fracasso C, Guiso G, Elger B, Schneider H, Seilheimer B, Caccia S, Bendotti C.
    J Neurosci Res; 2006 Jan 03; 83(1):134-46. PubMed ID: 16323214
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  • 13. Time course of neuropathology in the spinal cord of G86R superoxide dismutase transgenic mice.
    Morrison BM, Janssen WG, Gordon JW, Morrison JH.
    J Comp Neurol; 1998 Feb 02; 391(1):64-77. PubMed ID: 9527542
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  • 16. Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1.
    Jaarsma D, Haasdijk ED, Grashorn JA, Hawkins R, van Duijn W, Verspaget HW, London J, Holstege JC.
    Neurobiol Dis; 2000 Dec 02; 7(6 Pt B):623-43. PubMed ID: 11114261
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  • 20. Progressive loss of a glial potassium channel (KCNJ10) in the spinal cord of the SOD1 (G93A) transgenic mouse model of amyotrophic lateral sclerosis.
    Kaiser M, Maletzki I, Hülsmann S, Holtmann B, Schulz-Schaeffer W, Kirchhoff F, Bähr M, Neusch C.
    J Neurochem; 2006 Nov 02; 99(3):900-12. PubMed ID: 16925593
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