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Journal Abstract Search
659 related items for PubMed ID: 15276623
1. Partitioning of 5alpha-dihydrotestosterone and 5alpha-androstane-3alpha, 17beta-diol activated pathways for stimulating human prostate cancer LNCaP cell proliferation. Nunlist EH, Dozmorov I, Tang Y, Cowan R, Centola M, Lin HK. J Steroid Biochem Mol Biol; 2004 Jul; 91(3):157-70. PubMed ID: 15276623 [Abstract] [Full Text] [Related]
2. 5alpha-androstane-3alpha,17beta-diol supports human prostate cancer cell survival and proliferation through androgen receptor-independent signaling pathways: implication of androgen-independent prostate cancer progression. Yang Q, Titus MA, Fung KM, Lin HK. J Cell Biochem; 2008 Aug 01; 104(5):1612-24. PubMed ID: 18320593 [Abstract] [Full Text] [Related]
3. 5alpha-Androstane-3alpha,17beta-diol activates pathway that resembles the epidermal growth factor responsive pathways in stimulating human prostate cancer LNCaP cell proliferation. Zimmerman RA, Dozmorov I, Nunlist EH, Tang Y, Li X, Cowan R, Centola M, Frank MB, Culkin DJ, Lin HK. Prostate Cancer Prostatic Dis; 2004 Aug 01; 7(4):364-74. PubMed ID: 15452555 [Abstract] [Full Text] [Related]
4. Androgen inactivation and steroid-converting enzyme expression in abdominal adipose tissue in men. Blouin K, Richard C, Brochu G, Hould FS, Lebel S, Marceau S, Biron S, Luu-The V, Tchernof A. J Endocrinol; 2006 Dec 01; 191(3):637-49. PubMed ID: 17170221 [Abstract] [Full Text] [Related]
5. A neuroactive steroid 5alpha-androstane-3alpha,17beta-diol regulates androgen receptor level in astrocytes. Agapova OA, Malone PE, Hernandez MR. J Neurochem; 2006 Jul 01; 98(2):355-63. PubMed ID: 16638015 [Abstract] [Full Text] [Related]
6. Changes in androgen receptor nongenotropic signaling correlate with transition of LNCaP cells to androgen independence. Unni E, Sun S, Nan B, McPhaul MJ, Cheskis B, Mancini MA, Marcelli M. Cancer Res; 2004 Oct 01; 64(19):7156-68. PubMed ID: 15466214 [Abstract] [Full Text] [Related]
7. Increased expression of type 2 3alpha-hydroxysteroid dehydrogenase/type 5 17beta-hydroxysteroid dehydrogenase (AKR1C3) and its relationship with androgen receptor in prostate carcinoma. Fung KM, Samara EN, Wong C, Metwalli A, Krlin R, Bane B, Liu CZ, Yang JT, Pitha JV, Culkin DJ, Kropp BP, Penning TM, Lin HK. Endocr Relat Cancer; 2006 Mar 01; 13(1):169-80. PubMed ID: 16601286 [Abstract] [Full Text] [Related]
8. Enhanced formation of non-phenolic androgen metabolites with intrinsic oestrogen-like gene transactivation potency in human breast cancer cells: a distinctive metabolic pattern. Pérez-Palacios G, Santillán R, García-Becerra R, Borja-Cacho E, Larrea F, Damián-Matsumura P, González L, Lemus AE. J Endocrinol; 2006 Sep 01; 190(3):805-18. PubMed ID: 17003281 [Abstract] [Full Text] [Related]
9. 5alpha-Androstane-3beta,17beta-diol (3beta-diol), an estrogenic metabolite of 5alpha-dihydrotestosterone, is a potent modulator of estrogen receptor ERbeta expression in the ventral prostrate of adult rats. Oliveira AG, Coelho PH, Guedes FD, Mahecha GA, Hess RA, Oliveira CA. Steroids; 2007 Dec 01; 72(14):914-22. PubMed ID: 17854852 [Abstract] [Full Text] [Related]
10. Identification of the major oxidative 3alpha-hydroxysteroid dehydrogenase in human prostate that converts 5alpha-androstane-3alpha,17beta-diol to 5alpha-dihydrotestosterone: a potential therapeutic target for androgen-dependent disease. Bauman DR, Steckelbroeck S, Williams MV, Peehl DM, Penning TM. Mol Endocrinol; 2006 Feb 01; 20(2):444-58. PubMed ID: 16179381 [Abstract] [Full Text] [Related]
11. Formation of 5α-dihydrotestosterone from 5α-androstane-3α,17β-diol in prostate cancer LAPC-4 cells - Identifying inhibitors of non-classical pathways producing the most potent androgen. Boutin S, Roy J, Maltais R, Poirier D. Bioorg Med Chem Lett; 2020 Jan 15; 30(2):126783. PubMed ID: 31753699 [Abstract] [Full Text] [Related]
14. Prolactin influences upon androgen action in male accessory sex organs. Thomas JA, Keenan EJ. Adv Sex Horm Res; 1976 Jan 15; 2():425-70. PubMed ID: 189591 [Abstract] [Full Text] [Related]
18. 5alpha-androstane-3alpha,17beta-diol selectively activates the canonical PI3K/AKT pathway: a bioinformatics-based evidence for androgen-activated cytoplasmic signaling. Dozmorov MG, Yang Q, Matwalli A, Hurst RE, Culkin DJ, Kropp BP, Lin HK. Genomic Med; 2007 Jan 15; 1(3-4):139-46. PubMed ID: 18923939 [Abstract] [Full Text] [Related]