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PUBMED FOR HANDHELDS

Journal Abstract Search


843 related items for PubMed ID: 15312868

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  • 43. Excessive sarcoplasmic/endoplasmic reticulum Ca2+-ATPase expression causes increased sarcoplasmic reticulum Ca2+ uptake but decreases myocyte shortening.
    Teucher N, Prestle J, Seidler T, Currie S, Elliott EB, Reynolds DF, Schott P, Wagner S, Kogler H, Inesi G, Bers DM, Hasenfuss G, Smith GL.
    Circulation; 2004 Dec 07; 110(23):3553-9. PubMed ID: 15505097
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  • 49. Reversal mechanisms of left ventricular remodeling: lessons from left ventricular assist device experiments.
    Margulies KB.
    J Card Fail; 2002 Dec 07; 8(6 Suppl):S500-5. PubMed ID: 12555165
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  • 52. Sorcin interacts with sarcoplasmic reticulum Ca(2+)-ATPase and modulates excitation-contraction coupling in the heart.
    Matsumoto T, Hisamatsu Y, Ohkusa T, Inoue N, Sato T, Suzuki S, Ikeda Y, Matsuzaki M.
    Basic Res Cardiol; 2005 May 07; 100(3):250-62. PubMed ID: 15754088
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  • 53. The Na+/Ca2+ exchanger/SR Ca2+ ATPase transport capacity regulates the contractility of normal and hypertrophied feline ventricular myocytes.
    Weisser-Thomas J, Kubo H, Hefner CA, Gaughan JP, McGowan BS, Ross R, Meyer M, Dillmann W, Houser SR.
    J Card Fail; 2005 Jun 07; 11(5):380-7. PubMed ID: 15948089
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  • 54. Irregular rhythm adversely influences calcium handling in ventricular myocardium: implications for the interaction between heart failure and atrial fibrillation.
    Ling LH, Khammy O, Byrne M, Amirahmadi F, Foster A, Li G, Zhang L, dos Remedios C, Chen C, Kaye DM.
    Circ Heart Fail; 2012 Nov 07; 5(6):786-93. PubMed ID: 23014130
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  • 57. Changes in sarcolemmal Ca entry and sarcoplasmic reticulum Ca content in ventricular myocytes from patients with end-stage heart failure following myocardial recovery after combined pharmacological and ventricular assist device therapy.
    Terracciano CM, Harding SE, Adamson D, Koban M, Tansley P, Birks EJ, Barton PJ, Yacoub MH.
    Eur Heart J; 2003 Jul 07; 24(14):1329-39. PubMed ID: 12871690
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  • 58. Left ventricular assist device support reverses altered cardiac expression and function of natriuretic peptides and receptors in end-stage heart failure.
    Kuhn M, Voss M, Mitko D, Stypmann J, Schmid C, Kawaguchi N, Grabellus F, Baba HA.
    Cardiovasc Res; 2004 Nov 01; 64(2):308-14. PubMed ID: 15485690
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  • 59. Myocyte nitric oxide synthase 2 contributes to blunted beta-adrenergic response in failing human hearts by decreasing Ca2+ transients.
    Ziolo MT, Maier LS, Piacentino V, Bossuyt J, Houser SR, Bers DM.
    Circulation; 2004 Apr 20; 109(15):1886-91. PubMed ID: 15037528
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