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Pubmed for Handhelds

PUBMED FOR HANDHELDS

Journal Abstract Search

224 related items for PubMed ID: 15333588

  • 61.
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  • 62. A G220V substitution within the N-terminal transcription regulating domain of HOXD13 causes a variant synpolydactyly phenotype.
    Fantini S, Vaccari G, Brison N, Debeer P, Tylzanowski P, Zappavigna V.
    Hum Mol Genet; 2009 Mar 01; 18(5):847-60. PubMed ID: 19060004
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  • 63.
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  • 64. Synpolydactyly phenotypes correlate with size of expansions in HOXD13 polyalanine tract.
    Goodman FR, Mundlos S, Muragaki Y, Donnai D, Giovannucci-Uzielli ML, Lapi E, Majewski F, McGaughran J, McKeown C, Reardon W, Upton J, Winter RM, Olsen BR, Scambler PJ.
    Proc Natl Acad Sci U S A; 1997 Jul 08; 94(14):7458-63. PubMed ID: 9207113
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  • 72. Polyglutamine-expanded androgen receptors form aggregates that sequester heat shock proteins, proteasome components and SRC-1, and are suppressed by the HDJ-2 chaperone.
    Stenoien DL, Cummings CJ, Adams HP, Mancini MG, Patel K, DeMartino GN, Marcelli M, Weigel NL, Mancini MA.
    Hum Mol Genet; 1999 May 08; 8(5):731-41. PubMed ID: 10196362
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  • 77. Genomic structure of HOXD13 gene: a nine polyalanine duplication causes synpolydactyly in two unrelated families.
    Akarsu AN, Stoilov I, Yilmaz E, Sayli BS, Sarfarazi M.
    Hum Mol Genet; 1996 Jul 08; 5(7):945-52. PubMed ID: 8817328
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  • 78. PolyQ proteins interfere with nuclear degradation of cytosolic proteins by sequestering the Sis1p chaperone.
    Park SH, Kukushkin Y, Gupta R, Chen T, Konagai A, Hipp MS, Hayer-Hartl M, Hartl FU.
    Cell; 2013 Jul 03; 154(1):134-45. PubMed ID: 23791384
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