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223 related items for PubMed ID: 15485806
1. Recruitment of thyroid hormone receptor/retinoblastoma-interacting protein 230 by the aryl hydrocarbon receptor nuclear translocator is required for the transcriptional response to both dioxin and hypoxia. Beischlag TV, Taylor RT, Rose DW, Yoon D, Chen Y, Lee WH, Rosenfeld MG, Hankinson O. J Biol Chem; 2004 Dec 24; 279(52):54620-8. PubMed ID: 15485806 [Abstract] [Full Text] [Related]
2. Functional interference between hypoxia and dioxin signal transduction pathways: competition for recruitment of the Arnt transcription factor. Gradin K, McGuire J, Wenger RH, Kvietikova I, fhitelaw ML, Toftgård R, Tora L, Gassmann M, Poellinger L. Mol Cell Biol; 1996 Oct 24; 16(10):5221-31. PubMed ID: 8816435 [Abstract] [Full Text] [Related]
3. Recruitment of the NCoA/SRC-1/p160 family of transcriptional coactivators by the aryl hydrocarbon receptor/aryl hydrocarbon receptor nuclear translocator complex. Beischlag TV, Wang S, Rose DW, Torchia J, Reisz-Porszasz S, Muhammad K, Nelson WE, Probst MR, Rosenfeld MG, Hankinson O. Mol Cell Biol; 2002 Jun 24; 22(12):4319-33. PubMed ID: 12024042 [Abstract] [Full Text] [Related]
4. The transcription factor aryl hydrocarbon receptor nuclear translocator functions as an estrogen receptor beta-selective coactivator, and its recruitment to alternative pathways mediates antiestrogenic effects of dioxin. Rüegg J, Swedenborg E, Wahlström D, Escande A, Balaguer P, Pettersson K, Pongratz I. Mol Endocrinol; 2008 Feb 24; 22(2):304-16. PubMed ID: 17991765 [Abstract] [Full Text] [Related]
5. Aromatic hydrocarbon nuclear translocator as a common component for the hypoxia- and dioxin-induced gene expression. Park H. Mol Cells; 1999 Apr 30; 9(2):172-8. PubMed ID: 10340472 [Abstract] [Full Text] [Related]
6. Conditional disruption of the aryl hydrocarbon receptor nuclear translocator (Arnt) gene leads to loss of target gene induction by the aryl hydrocarbon receptor and hypoxia-inducible factor 1alpha. Tomita S, Sinal CJ, Yim SH, Gonzalez FJ. Mol Endocrinol; 2000 Oct 30; 14(10):1674-81. PubMed ID: 11043581 [Abstract] [Full Text] [Related]
7. ER alpha-AHR-ARNT protein-protein interactions mediate estradiol-dependent transrepression of dioxin-inducible gene transcription. Beischlag TV, Perdew GH. J Biol Chem; 2005 Jun 03; 280(22):21607-11. PubMed ID: 15837795 [Abstract] [Full Text] [Related]
8. Induction and superinduction of 2,3,7,8-tetrachlorodibenzo-rho-dioxin-inducible poly(ADP-ribose) polymerase: role of the aryl hydrocarbon receptor/aryl hydrocarbon receptor nuclear translocator transcription activation domains and a labile transcription repressor. Ma Q. Arch Biochem Biophys; 2002 Aug 15; 404(2):309-16. PubMed ID: 12147270 [Abstract] [Full Text] [Related]
9. Functional analysis of aryl hydrocarbon receptor nuclear translocator interactions with aryl hydrocarbon receptor in the yeast two-hybrid system. Yamaguchi Y, Kuo MT. Biochem Pharmacol; 1995 Oct 12; 50(8):1295-302. PubMed ID: 7488247 [Abstract] [Full Text] [Related]
10. Expression of aryl hydrocarbon receptor and aryl hydrocarbon receptor nuclear translocator messenger ribonucleic acids and proteins in rat and human testis. Schultz R, Suominen J, Värre T, Hakovirta H, Parvinen M, Toppari J, Pelto-Huikko M. Endocrinology; 2003 Mar 12; 144(3):767-76. PubMed ID: 12586752 [Abstract] [Full Text] [Related]
12. Significance of prolyl hydroxylase 2 in the interference of aryl hydrocarbon receptor and hypoxia-inducible factor-1 alpha signaling. Seifert A, Katschinski DM, Tonack S, Fischer B, Navarrete Santos A. Chem Res Toxicol; 2008 Feb 25; 21(2):341-8. PubMed ID: 18072750 [Abstract] [Full Text] [Related]
13. Analysis of aryl hydrocarbon receptor-mediated signaling during physiological hypoxia reveals lack of competition for the aryl hydrocarbon nuclear translocator transcription factor. Pollenz RS, Davarinos NA, Shearer TP. Mol Pharmacol; 1999 Dec 25; 56(6):1127-37. PubMed ID: 10570039 [Abstract] [Full Text] [Related]
14. 2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) induces plasminogen activator inhibitor-1 through an aryl hydrocarbon receptor-mediated pathway in mouse hepatoma cell lines. Son DS, Rozman KK. Arch Toxicol; 2002 Jul 25; 76(7):404-13. PubMed ID: 12111005 [Abstract] [Full Text] [Related]
15. Aryl hydrocarbon receptor expression and activity in cerebellar granule neuroblasts: implications for development and dioxin neurotoxicity. Williamson MA, Gasiewicz TA, Opanashuk LA. Toxicol Sci; 2005 Feb 25; 83(2):340-8. PubMed ID: 15537747 [Abstract] [Full Text] [Related]
16. An aryl hydrocarbon receptor conformation acts as the functional core of nuclear dioxin signaling. Kronenberg S, Esser C, Carlberg C. Nucleic Acids Res; 2000 Jun 15; 28(12):2286-91. PubMed ID: 10871357 [Abstract] [Full Text] [Related]
17. T cell-specific disruption of arylhydrocarbon receptor nuclear translocator (Arnt) gene causes resistance to 2,3,7,8-tetrachlorodibenzo-p-dioxin-induced thymic involution. Tomita S, Jiang HB, Ueno T, Takagi S, Tohi K, Maekawa S, Miyatake A, Furukawa A, Gonzalez FJ, Takeda J, Ichikawa Y, Takahama Y. J Immunol; 2003 Oct 15; 171(8):4113-20. PubMed ID: 14530333 [Abstract] [Full Text] [Related]
18. The aryl hydrocarbon receptor interacts with estrogen receptor alpha and orphan receptors COUP-TFI and ERRalpha1. Klinge CM, Kaur K, Swanson HI. Arch Biochem Biophys; 2000 Jan 01; 373(1):163-74. PubMed ID: 10620335 [Abstract] [Full Text] [Related]
19. Aryl hydrocarbon receptor (AHR) and AHR nuclear translocator (ARNT) expression in Baikal seal (Pusa sibirica) and association with 2,3,7,8-TCDD toxic equivalents and CYP1 expression levels. Kim EY, Iwata H, Suda T, Tanabe S, Amano M, Miyazaki N, Petrov EA. Comp Biochem Physiol C Toxicol Pharmacol; 2005 Jul 01; 141(3):281-91. PubMed ID: 16111922 [Abstract] [Full Text] [Related]
20. The silencing mediator of retinoic acid and thyroid hormone receptors can interact with the aryl hydrocarbon (Ah) receptor but fails to repress Ah receptor-dependent gene expression. Rushing SR, Denison MS. Arch Biochem Biophys; 2002 Jul 15; 403(2):189-201. PubMed ID: 12139968 [Abstract] [Full Text] [Related] Page: [Next] [New Search]