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127 related items for PubMed ID: 15546955

  • 1. Donor T-cell production of RANTES significantly contributes to the development of idiopathic pneumonia syndrome after allogeneic stem cell transplantation.
    Hildebrandt GC, Olkiewicz KM, Choi S, Corrion LA, Clouthier SG, Liu C, Serody JS, Cooke KR.
    Blood; 2005 Mar 15; 105(6):2249-57. PubMed ID: 15546955
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  • 2. Blockade of CXCR3 receptor:ligand interactions reduces leukocyte recruitment to the lung and the severity of experimental idiopathic pneumonia syndrome.
    Hildebrandt GC, Corrion LA, Olkiewicz KM, Lu B, Lowler K, Duffner UA, Moore BB, Kuziel WA, Liu C, Cooke KR.
    J Immunol; 2004 Aug 01; 173(3):2050-9. PubMed ID: 15265940
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  • 4. Donor-derived TNF-alpha regulates pulmonary chemokine expression and the development of idiopathic pneumonia syndrome after allogeneic bone marrow transplantation.
    Hildebrandt GC, Olkiewicz KM, Corrion LA, Chang Y, Clouthier SG, Liu C, Cooke KR.
    Blood; 2004 Jul 15; 104(2):586-93. PubMed ID: 15069018
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  • 5. CCR1/CCL5 (RANTES) receptor-ligand interactions modulate allogeneic T-cell responses and graft-versus-host disease following stem-cell transplantation.
    Choi SW, Hildebrandt GC, Olkiewicz KM, Hanauer DA, Chaudhary MN, Silva IA, Rogers CE, Deurloo DT, Fisher JM, Liu C, Adams D, Chensue SW, Cooke KR.
    Blood; 2007 Nov 01; 110(9):3447-55. PubMed ID: 17641205
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  • 7. Acceleration of idiopathic pneumonia syndrome (IPS) in the absence of donor MIP-1 alpha (CCL3) after allogeneic BMT in mice.
    Panoskaltsis-Mortari A, Hermanson JR, Taras E, Wangensteen OD, Serody JS, Blazar BR.
    Blood; 2003 May 01; 101(9):3714-21. PubMed ID: 12511416
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  • 11. The critical early proinflammatory events associated with idiopathic pneumonia syndrome in irradiated murine allogeneic recipients are due to donor T cell infusion and potentiated by cyclophosphamide.
    Panoskaltsis-Mortari A, Taylor PA, Yaeger TM, Wangensteen OD, Bitterman PB, Ingbar DH, Vallera DA, Blazar BR.
    J Clin Invest; 1997 Sep 01; 100(5):1015-27. PubMed ID: 9276718
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  • 12. Intercellular adhesion molecule-I (ICAM-I, CD54) deficiency segregates the unique pathophysiological requirements for generating idiopathic pneumonia syndrome (IPS) versus graft-versus-host disease following allogeneic murine bone marrow transplantation.
    Panoskaltsis-Mortari A, Hermanson JR, Haddad IY, Wangensteen OD, Blazar BR.
    Biol Blood Marrow Transplant; 2001 Sep 01; 7(7):368-77. PubMed ID: 11529486
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  • 13. Prevention of non-infectious pulmonary complications after intra-bone marrow stem cell transplantation in mice.
    Yamasuji-Maeda Y, Nishimori H, Seike K, Yamamoto A, Fujiwara H, Kuroi T, Saeki K, Fujinaga H, Okamoto S, Matsuoka KI, Fujii N, Tanaka T, Fujii M, Mominoki K, Kanekura T, Maeda Y.
    PLoS One; 2022 Sep 01; 17(9):e0273749. PubMed ID: 36084023
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  • 14. Hyporesponsiveness of donor cells to lipopolysaccharide stimulation reduces the severity of experimental idiopathic pneumonia syndrome: potential role for a gut-lung axis of inflammation.
    Cooke KR, Hill GR, Gerbitz A, Kobzik L, Martin TR, Crawford JM, Brewer JP, Ferrara JL.
    J Immunol; 2000 Dec 01; 165(11):6612-9. PubMed ID: 11086106
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  • 17. Differential roles for CCR5 expression on donor T cells during graft-versus-host disease based on pretransplant conditioning.
    Wysocki CA, Burkett SB, Panoskaltsis-Mortari A, Kirby SL, Luster AD, McKinnon K, Blazar BR, Serody JS.
    J Immunol; 2004 Jul 15; 173(2):845-54. PubMed ID: 15240671
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  • 18. The role of the CC chemokine, RANTES, in acute lung allograft rejection.
    Belperio JA, Burdick MD, Keane MP, Xue YY, Lynch JP, Daugherty BL, Kunkel SL, Strieter RM.
    J Immunol; 2000 Jul 01; 165(1):461-72. PubMed ID: 10861085
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  • 20. Inhibition of airway inflammation by amino-terminally modified RANTES/CC chemokine ligand 5 analogues is not mediated through CCR3.
    Chvatchko Y, Proudfoot AE, Buser R, Juillard P, Alouani S, Kosco-Vilbois M, Coyle AJ, Nibbs RJ, Graham G, Offord RE, Wells TN.
    J Immunol; 2003 Nov 15; 171(10):5498-506. PubMed ID: 14607956
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