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604 related items for PubMed ID: 15573402

  • 1. Mitochondrial permeability transition in CNS trauma: cause or effect of neuronal cell death?
    Sullivan PG, Rabchevsky AG, Waldmeier PC, Springer JE.
    J Neurosci Res; ; 79(1-2):231-9. PubMed ID: 15573402
    [Abstract] [Full Text] [Related]

  • 2. Cyclosporin A attenuates acute mitochondrial dysfunction following traumatic brain injury.
    Sullivan PG, Thompson MB, Scheff SW.
    Exp Neurol; 1999 Nov; 160(1):226-34. PubMed ID: 10630207
    [Abstract] [Full Text] [Related]

  • 3. The optimal dosage and window of opportunity to maintain mitochondrial homeostasis following traumatic brain injury using the uncoupler FCCP.
    Pandya JD, Pauly JR, Sullivan PG.
    Exp Neurol; 2009 Aug; 218(2):381-9. PubMed ID: 19477175
    [Abstract] [Full Text] [Related]

  • 4. High fluence low-power laser irradiation induces mitochondrial permeability transition mediated by reactive oxygen species.
    Wu S, Xing D, Gao X, Chen WR.
    J Cell Physiol; 2009 Mar; 218(3):603-11. PubMed ID: 19006121
    [Abstract] [Full Text] [Related]

  • 5. [The roles of mitochondrial permeability transition in brain ischemia].
    Kobayashi T.
    Hokkaido Igaku Zasshi; 2000 Jul; 75(4):243-52. PubMed ID: 10976404
    [Abstract] [Full Text] [Related]

  • 6. Continuous infusion of cyclosporin A postinjury significantly ameliorates cortical damage following traumatic brain injury.
    Sullivan PG, Thompson M, Scheff SW.
    Exp Neurol; 2000 Feb; 161(2):631-7. PubMed ID: 10686082
    [Abstract] [Full Text] [Related]

  • 7. The role of mitochondrial transition pore, and its modulation, in traumatic brain injury and delayed neurodegeneration after TBI.
    Mazzeo AT, Beat A, Singh A, Bullock MR.
    Exp Neurol; 2009 Aug; 218(2):363-70. PubMed ID: 19481077
    [Abstract] [Full Text] [Related]

  • 8. The powerhouse takes control of the cell: is the mitochondrial permeability transition a viable therapeutic target against neuronal dysfunction and death?
    Stavrovskaya IG, Kristal BS.
    Free Radic Biol Med; 2005 Mar 15; 38(6):687-97. PubMed ID: 15721979
    [Abstract] [Full Text] [Related]

  • 9. An intrathecal bolus of cyclosporin A before injury preserves mitochondrial integrity and attenuates axonal disruption in traumatic brain injury.
    Okonkwo DO, Povlishock JT.
    J Cereb Blood Flow Metab; 1999 Apr 15; 19(4):443-51. PubMed ID: 10197514
    [Abstract] [Full Text] [Related]

  • 10. Mitochondrial uncoupling as a potential therapeutic target in acute central nervous system injury.
    Maragos WF, Korde AS.
    J Neurochem; 2004 Oct 15; 91(2):257-62. PubMed ID: 15447659
    [Abstract] [Full Text] [Related]

  • 11. Clinical treatments for mitochondrial dysfunctions after brain injury.
    Merenda A, Bullock R.
    Curr Opin Crit Care; 2006 Apr 15; 12(2):90-6. PubMed ID: 16543782
    [Abstract] [Full Text] [Related]

  • 12. Role of the mitochondrial membrane permeability transition in cell death.
    Tsujimoto Y, Shimizu S.
    Apoptosis; 2007 May 15; 12(5):835-40. PubMed ID: 17136322
    [Abstract] [Full Text] [Related]

  • 13. Brain-derived respiring mitochondria exhibit homogeneous, complete and cyclosporin-sensitive permeability transition.
    Hansson MJ, Månsson R, Mattiasson G, Ohlsson J, Karlsson J, Keep MF, Elmér E.
    J Neurochem; 2004 May 15; 89(3):715-29. PubMed ID: 15086528
    [Abstract] [Full Text] [Related]

  • 14. Mitochondrial permeability transition pore opening as a promising therapeutic target in cardiac diseases.
    Javadov S, Karmazyn M, Escobales N.
    J Pharmacol Exp Ther; 2009 Sep 15; 330(3):670-8. PubMed ID: 19509316
    [Abstract] [Full Text] [Related]

  • 15. Caspase-dependent alteration of the ADP/ATP translocator triggers the mitochondrial permeability transition which is not required for the low-potassium-dependent apoptosis of cerebellar granule cells.
    Atlante A, Bobba A, de Bari L, Fontana F, Calissano P, Marra E, Passarella S.
    J Neurochem; 2006 May 15; 97(4):1166-81. PubMed ID: 16606362
    [Abstract] [Full Text] [Related]

  • 16. Opening of mitochondrial permeability transition pore induces hypercontracture in Ca2+ overloaded cardiac myocytes.
    Ruiz-Meana M, Abellán A, Miró-Casas E, Garcia-Dorado D.
    Basic Res Cardiol; 2007 Nov 15; 102(6):542-52. PubMed ID: 17891523
    [Abstract] [Full Text] [Related]

  • 17. Direct inhibition of the mitochondrial permeability transition pore: a possible mechanism for better neuroprotective effects of allopregnanolone over progesterone.
    Sayeed I, Parvez S, Wali B, Siemen D, Stein DG.
    Brain Res; 2009 Mar 31; 1263():165-73. PubMed ID: 19368823
    [Abstract] [Full Text] [Related]

  • 18. Mitochondrial defects and dysfunction in calcium regulation in glaucomatous trabecular meshwork cells.
    He Y, Ge J, Tombran-Tink J.
    Invest Ophthalmol Vis Sci; 2008 Nov 31; 49(11):4912-22. PubMed ID: 18614807
    [Abstract] [Full Text] [Related]

  • 19. Novel neuroprotective compound SCH-20148 rescues thymocytes and SH-SY5Y cells from thapsigargin-induced mitochondrial membrane potential reduction and cell death.
    Muramatsu Y, Maemoto T, Iwashita A, Matsuoka N.
    Eur J Pharmacol; 2007 Jun 01; 563(1-3):40-8. PubMed ID: 17343843
    [Abstract] [Full Text] [Related]

  • 20. Inhibition of mitochondrial membrane bound-glutathione transferase by mitochondrial permeability transition inhibitors including cyclosporin A.
    Ulziikhishig E, Lee KK, Hossain QS, Higa Y, Imaizumi N, Aniya Y.
    Life Sci; 2010 May 08; 86(19-20):726-32. PubMed ID: 20226794
    [Abstract] [Full Text] [Related]

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