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Journal Abstract Search

733 related items for PubMed ID: 15579492

  • 21. Inhibition of FAAH confers increased stem cell migration via PPARα.
    Wollank Y, Ramer R, Ivanov I, Salamon A, Peters K, Hinz B.
    J Lipid Res; 2015 Oct; 56(10):1947-60. PubMed ID: 26263913
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  • 22. Anandamide administration alone and after inhibition of fatty acid amide hydrolase (FAAH) increases dopamine levels in the nucleus accumbens shell in rats.
    Solinas M, Justinova Z, Goldberg SR, Tanda G.
    J Neurochem; 2006 Jul; 98(2):408-19. PubMed ID: 16805835
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  • 23. Fatty acid amide hydrolase inhibitors confer anti-invasive and antimetastatic effects on lung cancer cells.
    Winkler K, Ramer R, Dithmer S, Ivanov I, Merkord J, Hinz B.
    Oncotarget; 2016 Mar 22; 7(12):15047-64. PubMed ID: 26930716
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  • 24. Endogenous fatty acid ethanolamides suppress nicotine-induced activation of mesolimbic dopamine neurons through nuclear receptors.
    Melis M, Pillolla G, Luchicchi A, Muntoni AL, Yasar S, Goldberg SR, Pistis M.
    J Neurosci; 2008 Dec 17; 28(51):13985-94. PubMed ID: 19091987
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  • 25. Fatty acid amide hydrolase (FAAH) inhibition enhances memory acquisition through activation of PPAR-alpha nuclear receptors.
    Mazzola C, Medalie J, Scherma M, Panlilio LV, Solinas M, Tanda G, Drago F, Cadet JL, Goldberg SR, Yasar S.
    Learn Mem; 2009 May 17; 16(5):332-7. PubMed ID: 19403796
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  • 26. Anandamide transport is independent of fatty-acid amide hydrolase activity and is blocked by the hydrolysis-resistant inhibitor AM1172.
    Fegley D, Kathuria S, Mercier R, Li C, Goutopoulos A, Makriyannis A, Piomelli D.
    Proc Natl Acad Sci U S A; 2004 Jun 08; 101(23):8756-61. PubMed ID: 15138300
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  • 27. The FAAH inhibitor URB597 efficiently reduces tyrosine hydroxylase expression through CB₁- and FAAH-independent mechanisms.
    Bosier B, Muccioli GG, Lambert DM.
    Br J Pharmacol; 2013 Jun 08; 169(4):794-807. PubMed ID: 22970888
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  • 28. Inhibition of fatty acid amide hydrolase and monoacylglycerol lipase by the anandamide uptake inhibitor VDM11: evidence that VDM11 acts as an FAAH substrate.
    Vandevoorde S, Fowler CJ.
    Br J Pharmacol; 2005 Aug 08; 145(7):885-93. PubMed ID: 15895107
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  • 30. Attenuation of cue-induced reinstatement of nicotine seeking by URB597 through cannabinoid CB1 receptor in rats.
    Forget B, Guranda M, Gamaleddin I, Goldberg SR, Le Foll B.
    Psychopharmacology (Berl); 2016 May 08; 233(10):1823-8. PubMed ID: 26864774
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  • 31. Arachidonoylserotonin and other novel inhibitors of fatty acid amide hydrolase.
    Bisogno T, Melck D, De Petrocellis L, Bobrov MYu, Gretskaya NM, Bezuglov VV, Sitachitta N, Gerwick WH, Di Marzo V.
    Biochem Biophys Res Commun; 1998 Jul 30; 248(3):515-22. PubMed ID: 9703957
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  • 37. FAAH deficiency promotes energy storage and enhances the motivation for food.
    Touriño C, Oveisi F, Lockney J, Piomelli D, Maldonado R.
    Int J Obes (Lond); 2010 Mar 30; 34(3):557-68. PubMed ID: 20029375
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  • 38. Pharmacological activity of fatty acid amides is regulated, but not mediated, by fatty acid amide hydrolase in vivo.
    Lichtman AH, Hawkins EG, Griffin G, Cravatt BF.
    J Pharmacol Exp Ther; 2002 Jul 30; 302(1):73-9. PubMed ID: 12065702
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  • 39. Biochemical and biological properties of 4-(3-phenyl-[1,2,4] thiadiazol-5-yl)-piperazine-1-carboxylic acid phenylamide, a mechanism-based inhibitor of fatty acid amide hydrolase.
    Karbarz MJ, Luo L, Chang L, Tham CS, Palmer JA, Wilson SJ, Wennerholm ML, Brown SM, Scott BP, Apodaca RL, Keith JM, Wu J, Breitenbucher JG, Chaplan SR, Webb M.
    Anesth Analg; 2009 Jan 30; 108(1):316-29. PubMed ID: 19095868
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  • 40. The novel reversible fatty acid amide hydrolase inhibitor ST4070 increases endocannabinoid brain levels and counteracts neuropathic pain in different animal models.
    Caprioli A, Coccurello R, Rapino C, Di Serio S, Di Tommaso M, Vertechy M, Vacca V, Battista N, Pavone F, Maccarrone M, Borsini F.
    J Pharmacol Exp Ther; 2012 Jul 30; 342(1):188-95. PubMed ID: 22514334
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