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722 related items for PubMed ID: 15617736

  • 1. Phasic cardiovascular responses to mevinphos are mediated through differential activation of cGMP/PKG cascade and peroxynitrite via nitric oxide generated in the rat rostral ventrolateral medulla by NOS I and II isoforms.
    Chan JY, Chan SH, Li FC, Tsai CY, Cheng HL, Chang AY.
    Neuropharmacology; 2005 Jan; 48(1):161-72. PubMed ID: 15617736
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  • 2. Differential contributions of NOS isoforms in the rostral ventrolateral medulla to cardiovascular responses associated with mevinphos intoxication in the rat.
    Chan JY, Chan SH, Chang AY.
    Neuropharmacology; 2004 Jun; 46(8):1184-94. PubMed ID: 15111025
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  • 3. Muscarinic receptor-independent activation of cyclic adenosine monophosphate-dependent protein kinase in rostral ventrolateral medulla underlies the sympathoexcitatory phase of cardiovascular responses during mevinphos intoxication in the rat.
    Tsai CY, Wu CH, Chan SH, Chang AY.
    Shock; 2007 May; 27(5):559-64. PubMed ID: 17438462
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  • 4. In the rostral ventrolateral medulla, the 70-kDa heat shock protein (HSP70), but not HSP90, confers neuroprotection against fatal endotoxemia via augmentation of nitric-oxide synthase I (NOS I)/protein kinase G signaling pathway and inhibition of NOS II/peroxynitrite cascade.
    Li FC, Chan JY, Chan SH, Chang AY.
    Mol Pharmacol; 2005 Jul; 68(1):179-92. PubMed ID: 15827295
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  • 5. Activation of PI3K/Akt signaling in rostral ventrolateral medulla impairs brain stem cardiovascular regulation that underpins circulatory depression during mevinphos intoxication.
    Tsai CY, Chang AY, Chan JY, Chan SH.
    Biochem Pharmacol; 2014 Mar 01; 88(1):75-85. PubMed ID: 24462917
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  • 6. Contribution of peroxynitrite to fatal cardiovascular depression induced by overproduction of nitric oxide in rostral ventrolateral medulla of the rat.
    Chan SH, Wang LL, Ou CC, Chan JY.
    Neuropharmacology; 2002 Oct 01; 43(5):889-98. PubMed ID: 12384174
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  • 8. Differential engagements of glutamate and GABA receptors in cardiovascular actions of endogenous nNOS or iNOS at rostral ventrolateral medulla of rats.
    Chan SH, Wang LL, Chan JY.
    Br J Pharmacol; 2003 Feb 01; 138(4):584-93. PubMed ID: 12598412
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  • 12. Contribution of nitric oxide, superoxide anion, and peroxynitrite to activation of mitochondrial apoptotic signaling in hippocampal CA3 subfield following experimental temporal lobe status epilepticus.
    Chuang YC, Chen SD, Liou CW, Lin TK, Chang WN, Chan SH, Chang AY.
    Epilepsia; 2009 Apr 01; 50(4):731-46. PubMed ID: 19178557
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  • 14. Overexpression of inducible nitric oxide synthase in rostral ventrolateral medulla causes hypertension and sympathoexcitation via an increase in oxidative stress.
    Kimura Y, Hirooka Y, Sagara Y, Ito K, Kishi T, Shimokawa H, Takeshita A, Sunagawa K.
    Circ Res; 2005 Feb 04; 96(2):252-60. PubMed ID: 15591232
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  • 15. Neurochemistry within ventrolateral medulla and cardiovascular effects during static exercise following eNOS antagonism.
    Ishide T, Preuss CV, Maher TJ, Ally A.
    Neurosci Res; 2005 May 04; 52(1):21-30. PubMed ID: 15811549
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  • 19. Heme oxygenase-1 plays a pro-life role in experimental brain stem death via nitric oxide synthase I/protein kinase G signaling at rostral ventrolateral medulla.
    Dai KY, Chan SH, Chang AY.
    J Biomed Sci; 2010 Sep 07; 17(1):72. PubMed ID: 20819234
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