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Journal Abstract Search

454 related items for PubMed ID: 15630703

  • 1. Double-stranded RNA-activated protein kinase is required for the LPS-induced activation of STAT1 inflammatory signaling in rat brain glial cells.
    Lee JH, Park EJ, Kim OS, Kim HY, Joe EH, Jou I.
    Glia; 2005 Apr 01; 50(1):66-79. PubMed ID: 15630703
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  • 2. Physical association between STAT1 and the interferon-inducible protein kinase PKR and implications for interferon and double-stranded RNA signaling pathways.
    Wong AH, Tam NW, Yang YL, Cuddihy AR, Li S, Kirchhoff S, Hauser H, Decker T, Koromilas AE.
    EMBO J; 1997 Mar 17; 16(6):1291-304. PubMed ID: 9135145
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  • 3. Astrocyte TLR4 activation induces a proinflammatory environment through the interplay between MyD88-dependent NFκB signaling, MAPK, and Jak1/Stat1 pathways.
    Gorina R, Font-Nieves M, Márquez-Kisinousky L, Santalucia T, Planas AM.
    Glia; 2011 Feb 17; 59(2):242-55. PubMed ID: 21125645
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  • 5. NF-kappaB independent signaling pathway is responsible for LPS-induced GDNF gene expression in primary rat glial cultures.
    Tanaka T, Oh-Hashi K, Shitara H, Hirata Y, Kiuchi K.
    Neurosci Lett; 2008 Feb 06; 431(3):262-7. PubMed ID: 18164817
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  • 6. Differential effects of ethanol on glial signal transduction initiated by lipopolysaccharide and interferon-gamma.
    Choi DK, Lee H, Jeong J, Lim B, Suk K.
    J Neurosci Res; 2005 Oct 15; 82(2):225-31. PubMed ID: 16175582
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  • 9. Potential requirement of a functional double-stranded RNA-dependent protein kinase (PKR) for the tumoricidal activation of macrophages by lipopolysaccharide or IFN-alpha beta, but not IFN-gamma.
    Gusella GL, Musso T, Rottschafer SE, Pulkki K, Varesio L.
    J Immunol; 1995 Jan 01; 154(1):345-54. PubMed ID: 7995954
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  • 10. IFN-gamma-induced upregulation of Fcgamma-receptor-I during activation of monocytic cells requires the PKR and NFkappaB pathways.
    Kårehed K, Dimberg A, Dahl S, Nilsson K, Oberg F.
    Mol Immunol; 2007 Jan 01; 44(4):615-24. PubMed ID: 16516295
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  • 11. Genetic deletion of PKR abrogates TNF-induced activation of IkappaBalpha kinase, JNK, Akt and cell proliferation but potentiates p44/p42 MAPK and p38 MAPK activation.
    Takada Y, Ichikawa H, Pataer A, Swisher S, Aggarwal BB.
    Oncogene; 2007 Feb 22; 26(8):1201-12. PubMed ID: 16924232
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  • 14. NF-kappaB signaling pathway, not IFN-beta/STAT1, is responsible for the selenium suppression of LPS-induced nitric oxide production.
    Yun CH, Yang JS, Kang SS, Yang Y, Cho JH, Son CG, Han SH.
    Int Immunopharmacol; 2007 Sep 22; 7(9):1192-8. PubMed ID: 17630198
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  • 18. The role of STAT1/IRF-1 on synergistic ROS production and loss of mitochondrial transmembrane potential during hepatic cell death induced by LPS/d-GalN.
    Lee HJ, Oh YK, Rhee M, Lim JY, Hwang JY, Park YS, Kwon Y, Choi KH, Jo I, Park SI, Gao B, Kim WH.
    J Mol Biol; 2007 Jun 15; 369(4):967-84. PubMed ID: 17475277
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  • 19. Distinct signaling pathways for induction of type II NOS by IFNgamma and LPS in BV-2 microglial cells.
    Shen S, Yu S, Binek J, Chalimoniuk M, Zhang X, Lo SC, Hannink M, Wu J, Fritsche K, Donato R, Sun GY.
    Neurochem Int; 2005 Sep 15; 47(4):298-307. PubMed ID: 15955597
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  • 20. Inhibition of inducible nitric-oxide synthase expression by (5R)-5-hydroxytriptolide in interferon-gamma- and bacterial lipopolysaccharide-stimulated macrophages.
    Zhou R, Zheng SX, Tang W, He PL, Li XY, Yang YF, Li YC, Geng JG, Zuo JP.
    J Pharmacol Exp Ther; 2006 Jan 15; 316(1):121-8. PubMed ID: 16166270
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