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204 related items for PubMed ID: 15642137

  • 1. JNK1 is not essential for TNF-mediated joint disease.
    Köller M, Hayer S, Redlich K, Ricci R, David JP, Steiner G, Smolen JS, Wagner EF, Schett G.
    Arthritis Res Ther; 2005; 7(1):R166-73. PubMed ID: 15642137
    [Abstract] [Full Text] [Related]

  • 2. Osteoclasts are essential for TNF-alpha-mediated joint destruction.
    Redlich K, Hayer S, Ricci R, David JP, Tohidast-Akrad M, Kollias G, Steiner G, Smolen JS, Wagner EF, Schett G.
    J Clin Invest; 2002 Nov; 110(10):1419-27. PubMed ID: 12438440
    [Abstract] [Full Text] [Related]

  • 3. Activation of p38 MAPK is a key step in tumor necrosis factor-mediated inflammatory bone destruction.
    Zwerina J, Hayer S, Redlich K, Bobacz K, Kollias G, Smolen JS, Schett G.
    Arthritis Rheum; 2006 Feb; 54(2):463-72. PubMed ID: 16447221
    [Abstract] [Full Text] [Related]

  • 4. Antiapoptotic effect of c-Jun N-terminal Kinase-1 through Mcl-1 stabilization in TNF-induced hepatocyte apoptosis.
    Kodama Y, Taura K, Miura K, Schnabl B, Osawa Y, Brenner DA.
    Gastroenterology; 2009 Apr; 136(4):1423-34. PubMed ID: 19249395
    [Abstract] [Full Text] [Related]

  • 5. Differentially regulated expression of growth differentiation factor 5 and bone morphogenetic protein 7 in articular cartilage and synovium in murine chronic arthritis: potential importance for cartilage breakdown and synovial hypertrophy.
    Bobacz K, Sunk IG, Hayer S, Amoyo L, Tohidast-Akrad M, Kollias G, Smolen JS, Schett G.
    Arthritis Rheum; 2008 Jan; 58(1):109-18. PubMed ID: 18163510
    [Abstract] [Full Text] [Related]

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  • 7. Joint damage and inflammation in c-Jun N-terminal kinase 2 knockout mice with passive murine collagen-induced arthritis.
    Han Z, Chang L, Yamanishi Y, Karin M, Firestein GS.
    Arthritis Rheum; 2002 Mar; 46(3):818-23. PubMed ID: 11920420
    [Abstract] [Full Text] [Related]

  • 8. A model of inflammatory arthritis highlights a role for oncostatin M in pro-inflammatory cytokine-induced bone destruction via RANK/RANKL.
    Hui W, Cawston TE, Richards CD, Rowan AD.
    Arthritis Res Ther; 2005 Mar; 7(1):R57-64. PubMed ID: 15642143
    [Abstract] [Full Text] [Related]

  • 9. Osteoprotegerin protects against generalized bone loss in tumor necrosis factor-transgenic mice.
    Schett G, Redlich K, Hayer S, Zwerina J, Bolon B, Dunstan C, Görtz B, Schulz A, Bergmeister H, Kollias G, Steiner G, Smolen JS.
    Arthritis Rheum; 2003 Jul; 48(7):2042-51. PubMed ID: 12847699
    [Abstract] [Full Text] [Related]

  • 10. Cartilage damage and bone erosion are more prominent determinants of functional impairment in longstanding experimental arthritis than synovial inflammation.
    Hayer S, Bauer G, Willburger M, Sinn K, Alasti F, Plasenzotti R, Shvets T, Niederreiter B, Aschauer C, Steiner G, Podesser BK, Smolen JS, Redlich K.
    Dis Model Mech; 2016 Nov 01; 9(11):1329-1338. PubMed ID: 27638666
    [Abstract] [Full Text] [Related]

  • 11. Tumour necrosis factor activates the mitogen-activated protein kinases p38alpha and ERK in the synovial membrane in vivo.
    Görtz B, Hayer S, Tuerck B, Zwerina J, Smolen JS, Schett G.
    Arthritis Res Ther; 2005 Nov 01; 7(5):R1140-7. PubMed ID: 16207331
    [Abstract] [Full Text] [Related]

  • 12. The lonely death: chondrocyte apoptosis in TNF-induced arthritis.
    Polzer K, Schett G, Zwerina J.
    Autoimmunity; 2007 Jun 01; 40(4):333-6. PubMed ID: 17516222
    [Abstract] [Full Text] [Related]

  • 13. Tenosynovitis and osteoclast formation as the initial preclinical changes in a murine model of inflammatory arthritis.
    Hayer S, Redlich K, Korb A, Hermann S, Smolen J, Schett G.
    Arthritis Rheum; 2007 Jan 01; 56(1):79-88. PubMed ID: 17195210
    [Abstract] [Full Text] [Related]

  • 14. Mammalian target of rapamycin signaling is crucial for joint destruction in experimental arthritis and is activated in osteoclasts from patients with rheumatoid arthritis.
    Cejka D, Hayer S, Niederreiter B, Sieghart W, Fuereder T, Zwerina J, Schett G.
    Arthritis Rheum; 2010 Aug 01; 62(8):2294-302. PubMed ID: 20506288
    [Abstract] [Full Text] [Related]

  • 15. c-Jun N-terminal kinases (JNKs) mediate pro-inflammatory actions of microglia.
    Waetzig V, Czeloth K, Hidding U, Mielke K, Kanzow M, Brecht S, Goetz M, Lucius R, Herdegen T, Hanisch UK.
    Glia; 2005 May 01; 50(3):235-46. PubMed ID: 15739188
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  • 17. c-Jun N-terminal protein kinase 1 (JNK1), but not JNK2, is essential for tumor necrosis factor alpha-induced c-Jun kinase activation and apoptosis.
    Liu J, Minemoto Y, Lin A.
    Mol Cell Biol; 2004 Dec 01; 24(24):10844-56. PubMed ID: 15572687
    [Abstract] [Full Text] [Related]

  • 18. Involvement of the c-jun N-terminal kinases JNK1 and JNK2 in complement-mediated cell death.
    Gancz D, Donin N, Fishelson Z.
    Mol Immunol; 2009 Dec 01; 47(2-3):310-7. PubMed ID: 19864026
    [Abstract] [Full Text] [Related]

  • 19. Deficiency of c-Jun-NH(2)-terminal kinase-1 in mice enhances skin tumor development by 12-O-tetradecanoylphorbol-13-acetate.
    She QB, Chen N, Bode AM, Flavell RA, Dong Z.
    Cancer Res; 2002 Mar 01; 62(5):1343-8. PubMed ID: 11888903
    [Abstract] [Full Text] [Related]

  • 20. JNK1, but not JNK2, is required for COX-2 induction by nickel compounds.
    Zhang D, Li J, Wu K, Ouyang W, Ding J, Liu ZG, Costa M, Huang C.
    Carcinogenesis; 2007 Apr 01; 28(4):883-91. PubMed ID: 17065197
    [Abstract] [Full Text] [Related]

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