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307 related items for PubMed ID: 15661755

  • 1. Neuronal dysfunction in a polyglutamine disease model occurs in the absence of ubiquitin-proteasome system impairment and inversely correlates with the degree of nuclear inclusion formation.
    Bowman AB, Yoo SY, Dantuma NP, Zoghbi HY.
    Hum Mol Genet; 2005 Mar 01; 14(5):679-91. PubMed ID: 15661755
    [Abstract] [Full Text] [Related]

  • 2. Polyglutamine and polyalanine expansions in ataxin7 result in different types of aggregation and levels of toxicity.
    Latouche M, Fragner P, Martin E, El Hachimi KH, Zander C, Sittler A, Ruberg M, Brice A, Stevanin G.
    Mol Cell Neurosci; 2006 Mar 01; 31(3):438-45. PubMed ID: 16325416
    [Abstract] [Full Text] [Related]

  • 3. Proteasome impairment does not contribute to pathogenesis in R6/2 Huntington's disease mice: exclusion of proteasome activator REGgamma as a therapeutic target.
    Bett JS, Goellner GM, Woodman B, Pratt G, Rechsteiner M, Bates GP.
    Hum Mol Genet; 2006 Jan 01; 15(1):33-44. PubMed ID: 16311253
    [Abstract] [Full Text] [Related]

  • 4. Global changes to the ubiquitin system in Huntington's disease.
    Bennett EJ, Shaler TA, Woodman B, Ryu KY, Zaitseva TS, Becker CH, Bates GP, Schulman H, Kopito RR.
    Nature; 2007 Aug 09; 448(7154):704-8. PubMed ID: 17687326
    [Abstract] [Full Text] [Related]

  • 5. Polyglutamine-expanded ataxin-7 decreases nuclear translocation of NF-kappaB p65 and impairs NF-kappaB activity by inhibiting proteasome activity of cerebellar neurons.
    Wang HL, He CY, Chou AH, Yeh TH, Chen YL, Li AH.
    Cell Signal; 2007 Mar 09; 19(3):573-81. PubMed ID: 17005371
    [Abstract] [Full Text] [Related]

  • 6. Polyglutamine-expanded ataxin-7 upregulates Bax expression by activating p53 in cerebellar and inferior olivary neurons.
    Wang HL, Chou AH, Lin AC, Chen SY, Weng YH, Yeh TH.
    Exp Neurol; 2010 Aug 09; 224(2):486-94. PubMed ID: 20546728
    [Abstract] [Full Text] [Related]

  • 7. The DNA repair-ubiquitin-associated HR23 proteins are constituents of neuronal inclusions in specific neurodegenerative disorders without hampering DNA repair.
    Bergink S, Severijnen LA, Wijgers N, Sugasawa K, Yousaf H, Kros JM, van Swieten J, Oostra BA, Hoeijmakers JH, Vermeulen W, Willemsen R.
    Neurobiol Dis; 2006 Sep 09; 23(3):708-16. PubMed ID: 16860562
    [Abstract] [Full Text] [Related]

  • 8. SUMOylation attenuates the aggregation propensity and cellular toxicity of the polyglutamine expanded ataxin-7.
    Janer A, Werner A, Takahashi-Fujigasaki J, Daret A, Fujigasaki H, Takada K, Duyckaerts C, Brice A, Dejean A, Sittler A.
    Hum Mol Genet; 2010 Jan 01; 19(1):181-95. PubMed ID: 19843541
    [Abstract] [Full Text] [Related]

  • 9. Transcriptional alterations and chromatin remodeling in polyglutamine diseases.
    Helmlinger D, Tora L, Devys D.
    Trends Genet; 2006 Oct 01; 22(10):562-70. PubMed ID: 16911843
    [Abstract] [Full Text] [Related]

  • 10. The neuronal ubiquitin-proteasome system: murine models and their neurological phenotype.
    van Tijn P, Hol EM, van Leeuwen FW, Fischer DF.
    Prog Neurobiol; 2008 Jun 01; 85(2):176-93. PubMed ID: 18448229
    [Abstract] [Full Text] [Related]

  • 11. Limbic structures are prone to age-related impairments in proteasome activity and neuronal ubiquitinated inclusions in SAMP10 mouse: a model of cerebral degeneration.
    Shimada A, Keino H, Kawamura N, Chiba Y, Hosokawa M.
    Neuropathol Appl Neurobiol; 2008 Feb 01; 34(1):33-51. PubMed ID: 17973906
    [Abstract] [Full Text] [Related]

  • 12. The PML-nuclear inclusion of human supraoptic neurons: a new compartment with SUMO-1- and ubiquitin-proteasome-associated domains.
    Villagra NT, Navascues J, Casafont I, Val-Bernal JF, Lafarga M, Berciano MT.
    Neurobiol Dis; 2006 Jan 01; 21(1):181-93. PubMed ID: 16125395
    [Abstract] [Full Text] [Related]

  • 13. Role of proteolysis in polyglutamine disorders.
    Tarlac V, Storey E.
    J Neurosci Res; 2003 Nov 01; 74(3):406-16. PubMed ID: 14598317
    [Abstract] [Full Text] [Related]

  • 14. Low levels of mutant ubiquitin are degraded by the proteasome in vivo.
    van Tijn P, Verhage MC, Hobo B, van Leeuwen FW, Fischer DF.
    J Neurosci Res; 2010 Aug 15; 88(11):2325-37. PubMed ID: 20336771
    [Abstract] [Full Text] [Related]

  • 15. Functional alterations of the ubiquitin-proteasome system in motor neurons of a mouse model of familial amyotrophic lateral sclerosis.
    Cheroni C, Marino M, Tortarolo M, Veglianese P, De Biasi S, Fontana E, Zuccarello LV, Maynard CJ, Dantuma NP, Bendotti C.
    Hum Mol Genet; 2009 Jan 01; 18(1):82-96. PubMed ID: 18826962
    [Abstract] [Full Text] [Related]

  • 16. Amyloid precursor-like protein 2 cleavage contributes to neuronal intranuclear inclusions and cytotoxicity in spinocerebellar ataxia-7 (SCA7).
    Takahashi-Fujigasaki J, Breidert T, Fujigasaki H, Duyckaerts C, Camonis JH, Brice A, Lebre AS.
    Neurobiol Dis; 2011 Jan 01; 41(1):33-42. PubMed ID: 20732423
    [Abstract] [Full Text] [Related]

  • 17. Polyglutamine-expanded ataxin-7 causes cerebellar dysfunction by inducing transcriptional dysregulation.
    Chou AH, Chen CY, Chen SY, Chen WJ, Chen YL, Weng YS, Wang HL.
    Neurochem Int; 2010 Jan 01; 56(2):329-39. PubMed ID: 19909779
    [Abstract] [Full Text] [Related]

  • 18. A single amino acid substitution in a proteasome subunit triggers aggregation of ubiquitinated proteins in stressed neuronal cells.
    Li Z, Arnaud L, Rockwell P, Figueiredo-Pereira ME.
    J Neurochem; 2004 Jul 01; 90(1):19-28. PubMed ID: 15198663
    [Abstract] [Full Text] [Related]

  • 19. Accumulation of aberrant ubiquitin induces aggregate formation and cell death in polyglutamine diseases.
    de Pril R, Fischer DF, Maat-Schieman ML, Hobo B, de Vos RA, Brunt ER, Hol EM, Roos RA, van Leeuwen FW.
    Hum Mol Genet; 2004 Aug 15; 13(16):1803-13. PubMed ID: 15198995
    [Abstract] [Full Text] [Related]

  • 20. Ataxin-7 aggregation and ubiquitination in infantile SCA7 with 180 CAG repeats.
    Ansorge O, Giunti P, Michalik A, Van Broeckhoven C, Harding B, Wood N, Scaravilli F.
    Ann Neurol; 2004 Sep 15; 56(3):448-52. PubMed ID: 15349877
    [Abstract] [Full Text] [Related]

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