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PUBMED FOR HANDHELDS

Journal Abstract Search


370 related items for PubMed ID: 15665300

  • 1. Glioma formation in neurofibromatosis 1 reflects preferential activation of K-RAS in astrocytes.
    Dasgupta B, Li W, Perry A, Gutmann DH.
    Cancer Res; 2005 Jan 01; 65(1):236-45. PubMed ID: 15665300
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  • 4. Nucleophosmin mediates mammalian target of rapamycin-dependent actin cytoskeleton dynamics and proliferation in neurofibromin-deficient astrocytes.
    Sandsmark DK, Zhang H, Hegedus B, Pelletier CL, Weber JD, Gutmann DH.
    Cancer Res; 2007 May 15; 67(10):4790-9. PubMed ID: 17510408
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  • 5. Neurofibromatosis-1 (Nf1) heterozygous brain microglia elaborate paracrine factors that promote Nf1-deficient astrocyte and glioma growth.
    Daginakatte GC, Gutmann DH.
    Hum Mol Genet; 2007 May 01; 16(9):1098-112. PubMed ID: 17400655
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  • 6. Optic nerve glioma in mice requires astrocyte Nf1 gene inactivation and Nf1 brain heterozygosity.
    Bajenaru ML, Hernandez MR, Perry A, Zhu Y, Parada LF, Garbow JR, Gutmann DH.
    Cancer Res; 2003 Dec 15; 63(24):8573-7. PubMed ID: 14695164
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  • 7. NF1 regulates a Ras-dependent vascular smooth muscle proliferative injury response.
    Xu J, Ismat FA, Wang T, Yang J, Epstein JA.
    Circulation; 2007 Nov 06; 116(19):2148-56. PubMed ID: 17967772
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  • 9. Increased neurofibromatosis 1 gene expression in astrocytic tumors: positive regulation by p21-ras.
    Gutmann DH, Giordano MJ, Mahadeo DK, Lau N, Silbergeld D, Guha A.
    Oncogene; 1996 May 16; 12(10):2121-7. PubMed ID: 8668337
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  • 10. The neurofibromatosis 1 gene product neurofibromin regulates pituitary adenylate cyclase-activating polypeptide-mediated signaling in astrocytes.
    Dasgupta B, Dugan LL, Gutmann DH.
    J Neurosci; 2003 Oct 01; 23(26):8949-54. PubMed ID: 14523097
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  • 11. Mechanism for the learning deficits in a mouse model of neurofibromatosis type 1.
    Costa RM, Federov NB, Kogan JH, Murphy GG, Stern J, Ohno M, Kucherlapati R, Jacks T, Silva AJ.
    Nature; 2002 Jan 31; 415(6871):526-30. PubMed ID: 11793011
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  • 12. Proteomic analysis reveals hyperactivation of the mammalian target of rapamycin pathway in neurofibromatosis 1-associated human and mouse brain tumors.
    Dasgupta B, Yi Y, Chen DY, Weber JD, Gutmann DH.
    Cancer Res; 2005 Apr 01; 65(7):2755-60. PubMed ID: 15805275
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  • 13. Neurofibromin and its inactivation of Ras are prerequisites for osteoblast functioning.
    Yu X, Chen S, Potter OL, Murthy SM, Li J, Pulcini JM, Ohashi N, Winata T, Everett ET, Ingram D, Clapp WD, Hock JM.
    Bone; 2005 May 01; 36(5):793-802. PubMed ID: 15804420
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  • 15. The tumor suppressor neurofibromin confers sensitivity to apoptosis by Ras-dependent and Ras-independent pathways.
    Shapira S, Barkan B, Friedman E, Kloog Y, Stein R.
    Cell Death Differ; 2007 May 01; 14(5):895-906. PubMed ID: 17096025
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  • 16. Akt- or MEK-mediated mTOR inhibition suppresses Nf1 optic glioma growth.
    Kaul A, Toonen JA, Cimino PJ, Gianino SM, Gutmann DH.
    Neuro Oncol; 2015 Jun 01; 17(6):843-53. PubMed ID: 25534823
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  • 17. Hyperactive Ras as a therapeutic target in neurofibromatosis type 1.
    Weiss B, Bollag G, Shannon K.
    Am J Med Genet; 1999 Mar 26; 89(1):14-22. PubMed ID: 10469432
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  • 18. Loss of NF1 results in activation of the Ras signaling pathway and leads to aberrant growth in haematopoietic cells.
    Bollag G, Clapp DW, Shih S, Adler F, Zhang YY, Thompson P, Lange BJ, Freedman MH, McCormick F, Jacks T, Shannon K.
    Nat Genet; 1996 Feb 26; 12(2):144-8. PubMed ID: 8563751
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  • 19. NF1 inactivation cooperates with N-ras in in vivo lymphogenesis activating Erk by a mechanism independent of its Ras-GTPase accelerating activity.
    Mangues R, Corral T, Lu S, Symmans WF, Liu L, Pellicer A.
    Oncogene; 1998 Oct 01; 17(13):1705-16. PubMed ID: 9796699
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  • 20. Modulation of neurofibromatosis type 1 gene expression during in vitro myoblast differentiation.
    Gutmann DH, Cole JL, Collins FS.
    J Neurosci Res; 1994 Feb 15; 37(3):398-405. PubMed ID: 8176761
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