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688 related items for PubMed ID: 15799971

  • 1. Identification of WNK1 as a substrate of Akt/protein kinase B and a negative regulator of insulin-stimulated mitogenesis in 3T3-L1 cells.
    Jiang ZY, Zhou QL, Holik J, Patel S, Leszyk J, Coleman K, Chouinard M, Czech MP.
    J Biol Chem; 2005 Jun 03; 280(22):21622-8. PubMed ID: 15799971
    [Abstract] [Full Text] [Related]

  • 2. Activation of the mammalian target of rapamycin pathway acutely inhibits insulin signaling to Akt and glucose transport in 3T3-L1 and human adipocytes.
    Tremblay F, Gagnon A, Veilleux A, Sorisky A, Marette A.
    Endocrinology; 2005 Mar 03; 146(3):1328-37. PubMed ID: 15576463
    [Abstract] [Full Text] [Related]

  • 3. Protein phosphatase 2A negatively regulates insulin's metabolic signaling pathway by inhibiting Akt (protein kinase B) activity in 3T3-L1 adipocytes.
    Ugi S, Imamura T, Maegawa H, Egawa K, Yoshizaki T, Shi K, Obata T, Ebina Y, Kashiwagi A, Olefsky JM.
    Mol Cell Biol; 2004 Oct 03; 24(19):8778-89. PubMed ID: 15367694
    [Abstract] [Full Text] [Related]

  • 4. Regulation of the phosphatidylinositol 3-kinase, Akt/protein kinase B, FRAP/mammalian target of rapamycin, and ribosomal S6 kinase 1 signaling pathways by thyroid-stimulating hormone (TSH) and stimulating type TSH receptor antibodies in the thyroid gland.
    Suh JM, Song JH, Kim DW, Kim H, Chung HK, Hwang JH, Kim JM, Hwang ES, Chung J, Han JH, Cho BY, Ro HK, Shong M.
    J Biol Chem; 2003 Jun 13; 278(24):21960-71. PubMed ID: 12668683
    [Abstract] [Full Text] [Related]

  • 5. WNK1, the kinase mutated in an inherited high-blood-pressure syndrome, is a novel PKB (protein kinase B)/Akt substrate.
    Vitari AC, Deak M, Collins BJ, Morrice N, Prescott AR, Phelan A, Humphreys S, Alessi DR.
    Biochem J; 2004 Feb 15; 378(Pt 1):257-68. PubMed ID: 14611643
    [Abstract] [Full Text] [Related]

  • 6. Protein kinase B/AKT 1 plays a pivotal role in insulin-like growth factor-1 receptor signaling induced 3T3-L1 adipocyte differentiation.
    Xu J, Liao K.
    J Biol Chem; 2004 Aug 20; 279(34):35914-22. PubMed ID: 15192111
    [Abstract] [Full Text] [Related]

  • 7. Akt substrate TBC1D1 regulates GLUT1 expression through the mTOR pathway in 3T3-L1 adipocytes.
    Zhou QL, Jiang ZY, Holik J, Chawla A, Hagan GN, Leszyk J, Czech MP.
    Biochem J; 2008 May 01; 411(3):647-55. PubMed ID: 18215134
    [Abstract] [Full Text] [Related]

  • 8. Identification of p122RhoGAP (deleted in liver cancer-1) Serine 322 as a substrate for protein kinase B and ribosomal S6 kinase in insulin-stimulated cells.
    Hers I, Wherlock M, Homma Y, Yagisawa H, Tavaré JM.
    J Biol Chem; 2006 Feb 24; 281(8):4762-70. PubMed ID: 16338927
    [Abstract] [Full Text] [Related]

  • 9. PTEN, but not SHIP2, suppresses insulin signaling through the phosphatidylinositol 3-kinase/Akt pathway in 3T3-L1 adipocytes.
    Tang X, Powelka AM, Soriano NA, Czech MP, Guilherme A.
    J Biol Chem; 2005 Jun 10; 280(23):22523-9. PubMed ID: 15824124
    [Abstract] [Full Text] [Related]

  • 10. Inhibition of insulin signaling and adipogenesis by rapamycin: effect on phosphorylation of p70 S6 kinase vs eIF4E-BP1.
    El-Chaâr D, Gagnon A, Sorisky A.
    Int J Obes Relat Metab Disord; 2004 Feb 10; 28(2):191-8. PubMed ID: 14970836
    [Abstract] [Full Text] [Related]

  • 11. Hydrogen peroxide generated during cellular insulin stimulation is integral to activation of the distal insulin signaling cascade in 3T3-L1 adipocytes.
    Mahadev K, Wu X, Zilbering A, Zhu L, Lawrence JT, Goldstein BJ.
    J Biol Chem; 2001 Dec 28; 276(52):48662-9. PubMed ID: 11598110
    [Abstract] [Full Text] [Related]

  • 12. Protein kinase B phosphorylation of PIKfyve regulates the trafficking of GLUT4 vesicles.
    Berwick DC, Dell GC, Welsh GI, Heesom KJ, Hers I, Fletcher LM, Cooke FT, Tavaré JM.
    J Cell Sci; 2004 Dec 01; 117(Pt 25):5985-93. PubMed ID: 15546921
    [Abstract] [Full Text] [Related]

  • 13. Engagement of the insulin-sensitive pathway in the stimulation of glucose transport by alpha-lipoic acid in 3T3-L1 adipocytes.
    Yaworsky K, Somwar R, Ramlal T, Tritschler HJ, Klip A.
    Diabetologia; 2000 Mar 01; 43(3):294-303. PubMed ID: 10768090
    [Abstract] [Full Text] [Related]

  • 14. Shikonin stimulates glucose uptake in 3T3-L1 adipocytes via an insulin-independent tyrosine kinase pathway.
    Kamei R, Kitagawa Y, Kadokura M, Hattori F, Hazeki O, Ebina Y, Nishihara T, Oikawa S.
    Biochem Biophys Res Commun; 2002 Apr 05; 292(3):642-51. PubMed ID: 11922615
    [Abstract] [Full Text] [Related]

  • 15. Nelfinavir-induced insulin resistance is associated with impaired plasma membrane recruitment of the PI 3-kinase effectors Akt/PKB and PKC-zeta.
    Ben-Romano R, Rudich A, Tirosh A, Potashnik R, Sasaoka T, Riesenberg K, Schlaeffer F, Bashan N.
    Diabetologia; 2004 Jun 05; 47(6):1107-17. PubMed ID: 15168016
    [Abstract] [Full Text] [Related]

  • 16. On the mechanism for neomycin reversal of wortmannin inhibition of insulin stimulation of glucose uptake.
    Shimaya A, Kovacina KS, Roth RA.
    J Biol Chem; 2004 Dec 31; 279(53):55277-82. PubMed ID: 15504741
    [Abstract] [Full Text] [Related]

  • 17. Characterization of the mitogen-activated protein kinase/90-kilodalton ribosomal protein S6 kinase signaling pathway in 3T3-L1 adipocytes and its role in insulin-stimulated glucose transport.
    Fingar DC, Birnbaum MJ.
    Endocrinology; 1994 Feb 31; 134(2):728-35. PubMed ID: 8299568
    [Abstract] [Full Text] [Related]

  • 18. Cholesterol depletion disrupts caveolae and insulin receptor signaling for metabolic control via insulin receptor substrate-1, but not for mitogen-activated protein kinase control.
    Parpal S, Karlsson M, Thorn H, Strålfors P.
    J Biol Chem; 2001 Mar 30; 276(13):9670-8. PubMed ID: 11121405
    [Abstract] [Full Text] [Related]

  • 19. PI3 kinase directly phosphorylates Akt1/2 at Ser473/474 in the insulin signal transduction pathway.
    Tsuchiya A, Kanno T, Nishizaki T.
    J Endocrinol; 2014 Jan 30; 220(1):49-59. PubMed ID: 24169049
    [Abstract] [Full Text] [Related]

  • 20. Membrane localization of 3-phosphoinositide-dependent protein kinase-1 stimulates activities of Akt and atypical protein kinase C but does not stimulate glucose transport and glycogen synthesis in 3T3-L1 adipocytes.
    Egawa K, Maegawa H, Shi K, Nakamura T, Obata T, Yoshizaki T, Morino K, Shimizu S, Nishio Y, Suzuki E, Kashiwagi A.
    J Biol Chem; 2002 Oct 11; 277(41):38863-9. PubMed ID: 12147684
    [Abstract] [Full Text] [Related]

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