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Journal Abstract Search

201 related items for PubMed ID: 15836623

  • 1. Decreased expression of hypothalamic neuropeptides in Huntington disease transgenic mice with expanded polyglutamine-EGFP fluorescent aggregates.
    Kotliarova S, Jana NR, Sakamoto N, Kurosawa M, Miyazaki H, Nekooki M, Doi H, Machida Y, Wong HK, Suzuki T, Uchikawa C, Kotliarov Y, Uchida K, Nagao Y, Nagaoka U, Tamaoka A, Oyanagi K, Oyama F, Nukina N.
    J Neurochem; 2005 May; 93(3):641-53. PubMed ID: 15836623
    [Abstract] [Full Text] [Related]

  • 2. RNA-binding protein TLS is a major nuclear aggregate-interacting protein in huntingtin exon 1 with expanded polyglutamine-expressing cells.
    Doi H, Okamura K, Bauer PO, Furukawa Y, Shimizu H, Kurosawa M, Machida Y, Miyazaki H, Mitsui K, Kuroiwa Y, Nukina N.
    J Biol Chem; 2008 Mar 07; 283(10):6489-500. PubMed ID: 18167354
    [Abstract] [Full Text] [Related]

  • 3. Proteomics of polyglutamine aggregates.
    Mitsui K, Doi H, Nukina N.
    Methods Enzymol; 2006 Mar 07; 412():63-76. PubMed ID: 17046652
    [Abstract] [Full Text] [Related]

  • 4. 14-3-3zeta is indispensable for aggregate formation of polyglutamine-expanded huntingtin protein.
    Omi K, Hachiya NS, Tanaka M, Tokunaga K, Kaneko K.
    Neurosci Lett; 2008 Jan 24; 431(1):45-50. PubMed ID: 18078716
    [Abstract] [Full Text] [Related]

  • 5. Transgenic mice expressing mutated full-length HD cDNA: a paradigm for locomotor changes and selective neuronal loss in Huntington's disease.
    Reddy PH, Charles V, Williams M, Miller G, Whetsell WO, Tagle DA.
    Philos Trans R Soc Lond B Biol Sci; 1999 Jun 29; 354(1386):1035-45. PubMed ID: 10434303
    [Abstract] [Full Text] [Related]

  • 6. A structure-based analysis of huntingtin mutant polyglutamine aggregation and toxicity: evidence for a compact beta-sheet structure.
    Poirier MA, Jiang H, Ross CA.
    Hum Mol Genet; 2005 Mar 15; 14(6):765-74. PubMed ID: 15689354
    [Abstract] [Full Text] [Related]

  • 7. Modulating huntingtin half-life alters polyglutamine-dependent aggregate formation and cell toxicity.
    Kaytor MD, Wilkinson KD, Warren ST.
    J Neurochem; 2004 May 15; 89(4):962-73. PubMed ID: 15140195
    [Abstract] [Full Text] [Related]

  • 8. Disease progression despite early loss of polyglutamine protein expression in SCA7 mouse model.
    Helmlinger D, Abou-Sleymane G, Yvert G, Rousseau S, Weber C, Trottier Y, Mandel JL, Devys D.
    J Neurosci; 2004 Feb 25; 24(8):1881-7. PubMed ID: 14985428
    [Abstract] [Full Text] [Related]

  • 9. Human cytomegalovirus UL97 kinase prevents the deposition of mutant protein aggregates in cellular models of Huntington's disease and ataxia.
    Tower C, Fu L, Gill R, Prichard M, Lesort M, Sztul E.
    Neurobiol Dis; 2011 Jan 25; 41(1):11-22. PubMed ID: 20732421
    [Abstract] [Full Text] [Related]

  • 10. Significantly differential diffusion of neuropathological aggregates in the brain of transgenic mice carrying N-terminal mutant huntingtin fused with green fluorescent protein.
    Cheng PH, Li CL, Her LS, Chang YF, Chan AW, Chen CM, Yang SH.
    Brain Struct Funct; 2013 Jan 25; 218(1):283-94. PubMed ID: 22422149
    [Abstract] [Full Text] [Related]

  • 11. Dopamine enhances motor and neuropathological consequences of polyglutamine expanded huntingtin.
    Cyr M, Sotnikova TD, Gainetdinov RR, Caron MG.
    FASEB J; 2006 Dec 25; 20(14):2541-3. PubMed ID: 17065224
    [Abstract] [Full Text] [Related]

  • 12. Altered proteasomal function due to the expression of polyglutamine-expanded truncated N-terminal huntingtin induces apoptosis by caspase activation through mitochondrial cytochrome c release.
    Jana NR, Zemskov EA, Wang Gh, Nukina N.
    Hum Mol Genet; 2001 May 01; 10(10):1049-59. PubMed ID: 11331615
    [Abstract] [Full Text] [Related]

  • 13. Amyloid formation by mutant huntingtin: threshold, progressivity and recruitment of normal polyglutamine proteins.
    Huang CC, Faber PW, Persichetti F, Mittal V, Vonsattel JP, MacDonald ME, Gusella JF.
    Somat Cell Mol Genet; 1998 Jul 01; 24(4):217-33. PubMed ID: 10410676
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  • 17. Polyglutamine length-dependent interaction of Hsp40 and Hsp70 family chaperones with truncated N-terminal huntingtin: their role in suppression of aggregation and cellular toxicity.
    Jana NR, Tanaka M, Wang Gh, Nukina N.
    Hum Mol Genet; 2000 Aug 12; 9(13):2009-18. PubMed ID: 10942430
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  • 18. Inactivation of Drosophila Apaf-1 related killer suppresses formation of polyglutamine aggregates and blocks polyglutamine pathogenesis.
    Sang TK, Li C, Liu W, Rodriguez A, Abrams JM, Zipursky SL, Jackson GR.
    Hum Mol Genet; 2005 Feb 01; 14(3):357-72. PubMed ID: 15590702
    [Abstract] [Full Text] [Related]

  • 19. Huntingtin affinity for partners is not changed by polyglutamine length: aggregation itself triggers aberrant interactions.
    Davranche A, Aviolat H, Zeder-Lutz G, Busso D, Altschuh D, Trottier Y, Klein FA.
    Hum Mol Genet; 2011 Jul 15; 20(14):2795-806. PubMed ID: 21518730
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