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Journal Abstract Search

239 related items for PubMed ID: 15890690

  • 1. c-Jun N-terminal kinase (JNK) is required for survival and proliferation of B-lymphoma cells.
    Gururajan M, Chui R, Karuppannan AK, Ke J, Jennings CD, Bondada S.
    Blood; 2005 Aug 15; 106(4):1382-91. PubMed ID: 15890690
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  • 2. The role of MAPKs in B cell receptor-induced down-regulation of Egr-1 in immature B lymphoma cells.
    Ke J, Gururajan M, Kumar A, Simmons A, Turcios L, Chelvarajan RL, Cohen DM, Wiest DL, Monroe JG, Bondada S.
    J Biol Chem; 2006 Dec 29; 281(52):39806-18. PubMed ID: 17065146
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  • 7. CpG oligodeoxynucleotides rescue BKS-2 immature B cell lymphoma from anti-IgM-mediated growth inhibition by up-regulation of egr-1.
    Han SS, Chung ST, Robertson DA, Chelvarajan RL, Bondada S.
    Int Immunol; 1999 Jun 29; 11(6):871-9. PubMed ID: 10360960
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  • 8. Extracellular signal-regulated kinase-2, but not c-Jun NH2-terminal kinase, activation correlates with surface IgM-mediated apoptosis in the WEHI 231 B cell line.
    Lee JR, Koretzky GA.
    J Immunol; 1998 Aug 15; 161(4):1637-44. PubMed ID: 9712025
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  • 9. [Hyperactivation of c-Jun NH(2)-terminal protein kinase contributes to the proliferation of B lymphoma cells].
    Guo YY, Cui J, Hou CM, Wang QY, Wang J, Ma YF, Zhang JY.
    Zhongguo Shi Yan Xue Ye Xue Za Zhi; 2011 Feb 15; 19(1):100-4. PubMed ID: 21362231
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  • 10. Basal c-Jun NH2-terminal protein kinase activity is essential for survival and proliferation of T-cell acute lymphoblastic leukemia cells.
    Cui J, Wang Q, Wang J, Lv M, Zhu N, Li Y, Feng J, Shen B, Zhang J.
    Mol Cancer Ther; 2009 Dec 15; 8(12):3214-22. PubMed ID: 19996270
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  • 11. A dominant negative inhibitor of the Egr family of transcription regulatory factors suppresses cerebellar granule cell apoptosis by blocking c-Jun activation.
    Levkovitz Y, Baraban JM.
    J Neurosci; 2001 Aug 15; 21(16):5893-901. PubMed ID: 11487612
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  • 12. JNK supports survival in melanoma cells by controlling cell cycle arrest and apoptosis.
    Alexaki VI, Javelaud D, Mauviel A.
    Pigment Cell Melanoma Res; 2008 Aug 15; 21(4):429-38. PubMed ID: 18541008
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  • 13. Jun N-terminal kinase is essential for CD40-mediated IgE class switching in B cells.
    Jabara HH, Geha RS.
    J Allergy Clin Immunol; 2005 Apr 15; 115(4):856-63. PubMed ID: 15806010
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  • 14. C-Jun N-terminal kinases/c-Jun and p38 pathways cooperate in ceramide-induced neuronal apoptosis.
    Willaime-Morawek S, Brami-Cherrier K, Mariani J, Caboche J, Brugg B.
    Neuroscience; 2003 Apr 15; 119(2):387-97. PubMed ID: 12770554
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  • 15. p53 and Egr-1 additively suppress transformed growth in HT1080 cells but Egr-1 counteracts p53-dependent apoptosis.
    de Belle I, Huang RP, Fan Y, Liu C, Mercola D, Adamson ED.
    Oncogene; 1999 Jun 17; 18(24):3633-42. PubMed ID: 10380885
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  • 16. c-JUN N-terminal kinase (JNK) is activated and contributes to tumor cell proliferation in classical Hodgkin lymphoma.
    Leventaki V, Drakos E, Karanikou M, Psatha K, Lin P, Schlette E, Eliopoulos A, Vassilakopoulos TP, Papadaki H, Patsouris E, Medeiros LJ, Rassidakis GZ.
    Hum Pathol; 2014 Mar 17; 45(3):565-72. PubMed ID: 24457077
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  • 19. Amyloid peptide-induced cytokine and chemokine expression in THP-1 monocytes is blocked by small inhibitory RNA duplexes for early growth response-1 messenger RNA.
    Giri RK, Selvaraj SK, Kalra VK.
    J Immunol; 2003 May 15; 170(10):5281-94. PubMed ID: 12734378
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  • 20. Identification of a membrane Ig-induced p38 mitogen-activated protein kinase module that regulates cAMP response element binding protein phosphorylation and transcriptional activation in CH31 B cell lymphomas.
    Swart JM, Bergeron DM, Chiles TC.
    J Immunol; 2000 Mar 01; 164(5):2311-9. PubMed ID: 10679065
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