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534 related items for PubMed ID: 15983830

  • 1. Redox system expression in the motor neurons in amyotrophic lateral sclerosis (ALS): immunohistochemical studies on sporadic ALS, superoxide dismutase 1 (SOD1)-mutated familial ALS, and SOD1-mutated ALS animal models.
    Kato S, Kato M, Abe Y, Matsumura T, Nishino T, Aoki M, Itoyama Y, Asayama K, Awaya A, Hirano A, Ohama E.
    Acta Neuropathol; 2005 Aug; 110(2):101-12. PubMed ID: 15983830
    [Abstract] [Full Text] [Related]

  • 2. Expression of hepatocyte growth factor and c-Met in the anterior horn cells of the spinal cord in the patients with amyotrophic lateral sclerosis (ALS): immunohistochemical studies on sporadic ALS and familial ALS with superoxide dismutase 1 gene mutation.
    Kato S, Funakoshi H, Nakamura T, Kato M, Nakano I, Hirano A, Ohama E.
    Acta Neuropathol; 2003 Aug; 106(2):112-20. PubMed ID: 12707786
    [Abstract] [Full Text] [Related]

  • 3. Histological evidence of redox system breakdown caused by superoxide dismutase 1 (SOD1) aggregation is common to SOD1-mutated motor neurons in humans and animal models.
    Kato S, Saeki Y, Aoki M, Nagai M, Ishigaki A, Itoyama Y, Kato M, Asayama K, Awaya A, Hirano A, Ohama E.
    Acta Neuropathol; 2004 Feb; 107(2):149-58. PubMed ID: 14648077
    [Abstract] [Full Text] [Related]

  • 4. Histological recovery of the hepatocytes is based on the redox system upregulation in the animal models of mutant superoxide dismutase (SOD)1-linked amyotrophic lateral sclerosis.
    Kato M, Kato S, Abe Y, Nishino T, Ohama E, Aoki M, Itoyama Y.
    Histol Histopathol; 2006 Jul; 21(7):729-42. PubMed ID: 16598672
    [Abstract] [Full Text] [Related]

  • 5. TDP-43 immunoreactivity in neuronal inclusions in familial amyotrophic lateral sclerosis with or without SOD1 gene mutation.
    Tan CF, Eguchi H, Tagawa A, Onodera O, Iwasaki T, Tsujino A, Nishizawa M, Kakita A, Takahashi H.
    Acta Neuropathol; 2007 May; 113(5):535-42. PubMed ID: 17333220
    [Abstract] [Full Text] [Related]

  • 6. Protein-bound crotonaldehyde accumulates in the spinal cord of superoxide dismutase-1 mutation-associated familial amyotrophic lateral sclerosis and its transgenic mouse model.
    Shibata N, Kawaguchi M, Uchida K, Kakita A, Takahashi H, Nakano R, Fujimura H, Sakoda S, Ihara Y, Nobukuni K, Takehisa Y, Kuroda S, Kokubo Y, Kuzuhara S, Honma T, Mochizuki Y, Mizutani T, Yamada S, Toi S, Sasaki S, Iwata M, Hirano A, Yamamoto T, Kato Y, Sawada T, Kobayashi M.
    Neuropathology; 2007 Feb; 27(1):49-61. PubMed ID: 17319283
    [Abstract] [Full Text] [Related]

  • 7. HtrA2/Omi-immunoreactive intraneuronal inclusions in the anterior horn of patients with sporadic and Cu/Zn superoxide dismutase (SOD1) mutant amyotrophic lateral sclerosis.
    Kawamoto Y, Ito H, Kobayashi Y, Suzuki Y, Akiguchi I, Fujimura H, Sakoda S, Kusaka H, Hirano A, Takahashi R.
    Neuropathol Appl Neurobiol; 2010 Jun; 36(4):331-44. PubMed ID: 20202124
    [Abstract] [Full Text] [Related]

  • 8. FUS-immunoreactive inclusions are a common feature in sporadic and non-SOD1 familial amyotrophic lateral sclerosis.
    Deng HX, Zhai H, Bigio EH, Yan J, Fecto F, Ajroud K, Mishra M, Ajroud-Driss S, Heller S, Sufit R, Siddique N, Mugnaini E, Siddique T.
    Ann Neurol; 2010 Jun; 67(6):739-48. PubMed ID: 20517935
    [Abstract] [Full Text] [Related]

  • 9. TDP-43 is consistently co-localized with ubiquitinated inclusions in sporadic and Guam amyotrophic lateral sclerosis but not in familial amyotrophic lateral sclerosis with and without SOD1 mutations.
    Maekawa S, Leigh PN, King A, Jones E, Steele JC, Bodi I, Shaw CE, Hortobagyi T, Al-Sarraj S.
    Neuropathology; 2009 Dec; 29(6):672-83. PubMed ID: 19496940
    [Abstract] [Full Text] [Related]

  • 10. Colocalization of 14-3-3 proteins with SOD1 in Lewy body-like hyaline inclusions in familial amyotrophic lateral sclerosis cases and the animal model.
    Okamoto Y, Shirakashi Y, Ihara M, Urushitani M, Oono M, Kawamoto Y, Yamashita H, Shimohama S, Kato S, Hirano A, Tomimoto H, Ito H, Takahashi R.
    PLoS One; 2011 Dec; 6(5):e20427. PubMed ID: 21655264
    [Abstract] [Full Text] [Related]

  • 11. S100A6 overexpression within astrocytes associated with impaired axons from both ALS mouse model and human patients.
    Hoyaux D, Boom A, Van den Bosch L, Belot N, Martin JJ, Heizmann CW, Kiss R, Pochet R.
    J Neuropathol Exp Neurol; 2002 Aug; 61(8):736-44. PubMed ID: 12152788
    [Abstract] [Full Text] [Related]

  • 12. Delayed disease onset and extended survival in the SOD1G93A rat model of amyotrophic lateral sclerosis after suppression of mutant SOD1 in the motor cortex.
    Thomsen GM, Gowing G, Latter J, Chen M, Vit JP, Staggenborg K, Avalos P, Alkaslasi M, Ferraiuolo L, Likhite S, Kaspar BK, Svendsen CN.
    J Neurosci; 2014 Nov 19; 34(47):15587-600. PubMed ID: 25411487
    [Abstract] [Full Text] [Related]

  • 13. Recent advances in research on neuropathological aspects of familial amyotrophic lateral sclerosis with superoxide dismutase 1 gene mutations: neuronal Lewy body-like hyaline inclusions and astrocytic hyaline inclusions.
    Kato S, Saito M, Hirano A, Ohama E.
    Histol Histopathol; 1999 Jul 19; 14(3):973-89. PubMed ID: 10425565
    [Abstract] [Full Text] [Related]

  • 14. Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1.
    Jaarsma D, Haasdijk ED, Grashorn JA, Hawkins R, van Duijn W, Verspaget HW, London J, Holstege JC.
    Neurobiol Dis; 2000 Dec 19; 7(6 Pt B):623-43. PubMed ID: 11114261
    [Abstract] [Full Text] [Related]

  • 15. The small heat shock protein B8 (HspB8) promotes autophagic removal of misfolded proteins involved in amyotrophic lateral sclerosis (ALS).
    Crippa V, Sau D, Rusmini P, Boncoraglio A, Onesto E, Bolzoni E, Galbiati M, Fontana E, Marino M, Carra S, Bendotti C, De Biasi S, Poletti A.
    Hum Mol Genet; 2010 Sep 01; 19(17):3440-56. PubMed ID: 20570967
    [Abstract] [Full Text] [Related]

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  • 17. Aberrant localization of FUS and TDP43 is associated with misfolding of SOD1 in amyotrophic lateral sclerosis.
    Pokrishevsky E, Grad LI, Yousefi M, Wang J, Mackenzie IR, Cashman NR.
    PLoS One; 2012 Sep 01; 7(4):e35050. PubMed ID: 22493728
    [Abstract] [Full Text] [Related]

  • 18. Pathological Modification of TDP-43 in Amyotrophic Lateral Sclerosis with SOD1 Mutations.
    Jeon GS, Shim YM, Lee DY, Kim JS, Kang M, Ahn SH, Shin JY, Geum D, Hong YH, Sung JJ.
    Mol Neurobiol; 2019 Mar 01; 56(3):2007-2021. PubMed ID: 29982983
    [Abstract] [Full Text] [Related]

  • 19. Bromocriptine methylate suppresses glial inflammation and moderates disease progression in a mouse model of amyotrophic lateral sclerosis.
    Tanaka K, Kanno T, Yanagisawa Y, Yasutake K, Hadano S, Yoshii F, Ikeda JE.
    Exp Neurol; 2011 Nov 01; 232(1):41-52. PubMed ID: 21867702
    [Abstract] [Full Text] [Related]

  • 20. Pathological TDP-43 distinguishes sporadic amyotrophic lateral sclerosis from amyotrophic lateral sclerosis with SOD1 mutations.
    Mackenzie IR, Bigio EH, Ince PG, Geser F, Neumann M, Cairns NJ, Kwong LK, Forman MS, Ravits J, Stewart H, Eisen A, McClusky L, Kretzschmar HA, Monoranu CM, Highley JR, Kirby J, Siddique T, Shaw PJ, Lee VM, Trojanowski JQ.
    Ann Neurol; 2007 May 01; 61(5):427-34. PubMed ID: 17469116
    [Abstract] [Full Text] [Related]

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