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Journal Abstract Search

106 related items for PubMed ID: 16042548

  • 1. Expression of normal sequence pathogenic proteins for neurodegenerative disease contributes to disease risk: 'permissive templating' as a general mechanism underlying neurodegeneration.
    Hardy J.
    Biochem Soc Trans; 2005 Aug; 33(Pt 4):578-81. PubMed ID: 16042548
    [Abstract] [Full Text] [Related]

  • 2. The law of mass action applied to neurodegenerative disease: a hypothesis concerning the etiology and pathogenesis of complex diseases.
    Singleton A, Myers A, Hardy J.
    Hum Mol Genet; 2004 Apr 01; 13 Spec No 1():R123-6. PubMed ID: 14976159
    [Abstract] [Full Text] [Related]

  • 3. [Proteins and mutations: a new vision (molecular) of neurodegenerative diseases].
    Christen Y.
    J Soc Biol; 2002 Apr 01; 196(1):85-94. PubMed ID: 12134639
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  • 4. Neurodegeneration and Alzheimer's disease: the lesson from tauopathies.
    Sorrentino G, Bonavita V.
    Neurol Sci; 2007 Apr 01; 28(2):63-71. PubMed ID: 17464468
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  • 5. [Conformationally altered proteins cause neurodegenerative diseases].
    Ingelsson M, Nilsson L, Basun H, Aquilonius SM, Lannfelt L.
    Lakartidningen; 2007 Apr 01; 102(47):3542-3, 3545-6, 3549 passim. PubMed ID: 16408392
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  • 6. A molecular pathway of neurodegeneration linking alpha-synuclein to ApoE and Abeta peptides.
    Gallardo G, Schlüter OM, Südhof TC.
    Nat Neurosci; 2008 Mar 01; 11(3):301-8. PubMed ID: 18297066
    [Abstract] [Full Text] [Related]

  • 7. Neurofibrillary degeneration in Alzheimer's disease: from molecular mechanisms to identification of drug targets.
    Pei JJ, Sjögren M, Winblad B.
    Curr Opin Psychiatry; 2008 Nov 01; 21(6):555-61. PubMed ID: 18852562
    [Abstract] [Full Text] [Related]

  • 8. [Abeta, tau and alpha-synuclein and glial cells].
    Akiyama H.
    Nihon Shinkei Seishin Yakurigaku Zasshi; 2006 Feb 01; 26(1):23-31. PubMed ID: 16637593
    [Abstract] [Full Text] [Related]

  • 9. Truncated tau from sporadic Alzheimer's disease suffices to drive neurofibrillary degeneration in vivo.
    Zilka N, Filipcik P, Koson P, Fialova L, Skrabana R, Zilkova M, Rolkova G, Kontsekova E, Novak M.
    FEBS Lett; 2006 Jun 26; 580(15):3582-8. PubMed ID: 16753151
    [Abstract] [Full Text] [Related]

  • 10. Transgenic animal models of neurodegenerative diseases and their application to treatment development.
    Rockenstein E, Crews L, Masliah E.
    Adv Drug Deliv Rev; 2007 Sep 30; 59(11):1093-102. PubMed ID: 17869376
    [Abstract] [Full Text] [Related]

  • 11. Accelerated amyloid deposition, neurofibrillary degeneration and neuronal loss in double mutant APP/tau transgenic mice.
    Ribé EM, Pérez M, Puig B, Gich I, Lim F, Cuadrado M, Sesma T, Catena S, Sánchez B, Nieto M, Gómez-Ramos P, Morán MA, Cabodevilla F, Samaranch L, Ortiz L, Pérez A, Ferrer I, Avila J, Gómez-Isla T.
    Neurobiol Dis; 2005 Dec 30; 20(3):814-22. PubMed ID: 16125396
    [Abstract] [Full Text] [Related]

  • 12. Tau gene mutations and their effects.
    Goedert M.
    Mov Disord; 2005 Aug 30; 20 Suppl 12():S45-52. PubMed ID: 16092090
    [Abstract] [Full Text] [Related]

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  • 15. Tauopathy in human and experimental variant Creutzfeldt-Jakob disease.
    Giaccone G, Mangieri M, Capobianco R, Limido L, Hauw JJ, Haïk S, Fociani P, Bugiani O, Tagliavini F.
    Neurobiol Aging; 2008 Dec 30; 29(12):1864-73. PubMed ID: 17560687
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  • 18. Cerebrovascular P-glycoprotein expression is decreased in Creutzfeldt-Jakob disease.
    Vogelgesang S, Glatzel M, Walker LC, Kroemer HK, Aguzzi A, Warzok RW.
    Acta Neuropathol; 2006 May 30; 111(5):436-43. PubMed ID: 16523342
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  • 19. Age-related accumulation of Reelin in amyloid-like deposits.
    Knuesel I, Nyffeler M, Mormède C, Muhia M, Meyer U, Pietropaolo S, Yee BK, Pryce CR, LaFerla FM, Marighetto A, Feldon J.
    Neurobiol Aging; 2009 May 30; 30(5):697-716. PubMed ID: 17904250
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