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317 related items for PubMed ID: 1611704

  • 1. Polymicrobial sepsis selectively activates peritoneal but not alveolar macrophages to release inflammatory mediators (interleukins-1 and -6 and tumor necrosis factor).
    Ayala A, Perrin MM, Kisala JM, Ertel W, Chaudry IH.
    Circ Shock; 1992 Mar; 36(3):191-9. PubMed ID: 1611704
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  • 5. Organ-association phenomena during sepsis. TNF and IL-6 in different macrophages.
    Meng XJ, Xu YX, Song XH, Li L, Li H.
    Chin Med J (Engl); 1993 Jun; 106(6):458-62. PubMed ID: 8222898
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  • 6. The importance of systemic cytokines in the pathogenesis of polymicrobial sepsis and dehydroepiandrosterone treatment in a rodent model.
    Hildebrand F, Pape HC, Hoevel P, Krettek C, van Griensven M.
    Shock; 2003 Oct; 20(4):338-46. PubMed ID: 14501948
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  • 7. Polymicrobial sepsis but not low-dose endotoxin infusion causes decreased splenocyte IL-2/IFN-gamma release while increasing IL-4/IL-10 production.
    Ayala A, Deol ZK, Lehman DL, Herdon CD, Chaudry IH.
    J Surg Res; 1994 Jun; 56(6):579-85. PubMed ID: 8015314
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  • 8. Effects of prolactin and metoclopramide on macrophage cytokine gene expression in late sepsis.
    Zhu XH, Zellweger R, Wichmann MW, Ayala A, Chaudry IH.
    Cytokine; 1997 Jun; 9(6):437-46. PubMed ID: 9199878
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  • 9. Interleukin-12 and -18 induce severe liver injury in mice recovered from peritonitis after sublethal endotoxin challenge.
    Ono S, Ueno C, Seki S, Matsumoto A, Mochizuki H.
    Surgery; 2003 Jul; 134(1):92-100. PubMed ID: 12874588
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  • 10. Liu-Shen-Wan, a traditional Chinese medicine, improves survival in sepsis induced by cecal ligation and puncture via reducing TNF-alpha levels, MDA content and enhancing macrophage phagocytosis.
    Ma H, Kou J, Zhu D, Yan Y, Yu B.
    Int Immunopharmacol; 2006 Aug; 6(8):1355-62. PubMed ID: 16782549
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  • 11. Interaction between the innate and adaptive immune systems is required to survive sepsis and control inflammation after injury.
    Shelley O, Murphy T, Paterson H, Mannick JA, Lederer JA.
    Shock; 2003 Aug; 20(2):123-9. PubMed ID: 12865655
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  • 14. Regulation of macrophage TNF alpha, IL-1 beta, and Ia (I-A alpha) mRNA expression during peritonitis is site dependent.
    McMasters KM, Cheadle WG.
    J Surg Res; 1993 May; 54(5):426-30. PubMed ID: 8361168
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  • 15. [The effect of caspase-3 inhibitor on the concentrations of serum inflammatory cytokines in sepsis related acute kidney injury induced by peritoneal cavity infection in mice].
    Liu LX, Hu ZJ, Li Y, Su J, Huo Y, Fan LQ.
    Zhongguo Wei Zhong Bing Ji Jiu Yi Xue; 2010 Dec; 22(12):736-9. PubMed ID: 21190601
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  • 16. Anti-tumor necrosis factor antibody therapy fails to prevent lethality after cecal ligation and puncture or endotoxemia.
    Eskandari MK, Bolgos G, Miller C, Nguyen DT, DeForge LE, Remick DG.
    J Immunol; 1992 May 01; 148(9):2724-30. PubMed ID: 1315357
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  • 17. Alveolar macrophage suppression in sepsis is associated with high mobility group box 1 transmigration.
    Pahuja M, Tran C, Wang H, Yin K.
    Shock; 2008 Jun 01; 29(6):754-60. PubMed ID: 18004227
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  • 18. Cytokine release by macrophages after interacting with CSF-1 and extracellular matrix proteins: characteristics of a mouse model of inflammatory responses in vitro.
    Kremlev SG, Chapoval AI, Evans R.
    Cell Immunol; 1998 Apr 10; 185(1):59-64. PubMed ID: 9636683
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  • 19. Selective macrophage suppression during sepsis.
    Ellaban E, Bolgos G, Remick D.
    Cell Immunol; 2004 Apr 10; 231(1-2):103-11. PubMed ID: 15919375
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  • 20. IL-4-induced activation of the Stat6 pathway contributes to the suppression of cell-mediated immunity and death in sepsis.
    Song GY, Chung CS, Chaudry IH, Ayala A.
    Surgery; 2000 Aug 10; 128(2):133-8. PubMed ID: 10922982
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