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280 related items for PubMed ID: 16131363

  • 1. Diabetic embryopathy: studies using a rat embryo culture system and an animal model.
    Akazawa S.
    Congenit Anom (Kyoto); 2005 Sep; 45(3):73-9. PubMed ID: 16131363
    [Abstract] [Full Text] [Related]

  • 2. A diabetes-like environment increases malformation rate and diminishes prostaglandin E(2) in rat embryos: reversal by administration of vitamin E and folic acid.
    Wentzel P, Eriksson UJ.
    Birth Defects Res A Clin Mol Teratol; 2005 Jul; 73(7):506-11. PubMed ID: 15959876
    [Abstract] [Full Text] [Related]

  • 3. PPARdelta and its activator PGI2 are reduced in diabetic embryopathy: involvement of PPARdelta activation in lipid metabolic and signalling pathways in rat embryo early organogenesis.
    Higa R, González E, Pustovrh MC, White V, Capobianco E, Martínez N, Jawerbaum A.
    Mol Hum Reprod; 2007 Feb; 13(2):103-10. PubMed ID: 17148578
    [Abstract] [Full Text] [Related]

  • 4. Role of reactive oxygen species (ROS) in the diabetes-induced anomalies in rat embryos in vitro: reduction in antioxidant enzymes and low-molecular-weight antioxidants (LMWA) may be the causative factor for increased anomalies.
    Ornoy A, Zaken V, Kohen R.
    Teratology; 1999 Dec; 60(6):376-86. PubMed ID: 10590399
    [Abstract] [Full Text] [Related]

  • 5. Dietary myo-inositol therapy in hyperglycemia-induced embryopathy.
    Khandelwal M, Reece EA, Wu YK, Borenstein M.
    Teratology; 1998 Feb; 57(2):79-84. PubMed ID: 9562680
    [Abstract] [Full Text] [Related]

  • 6. Altered mitochondrial morphology of rat embryos in diabetic pregnancy.
    Yang X, Borg LA, Eriksson UJ.
    Anat Rec; 1995 Feb; 241(2):255-67. PubMed ID: 7710141
    [Abstract] [Full Text] [Related]

  • 7. N-Acetylcysteine and alpha-cyano-4-hydroxycinnamic acid alter protein kinase C (PKC)-delta and PKC-zeta and diminish dysmorphogenesis in rat embryos cultured with high glucose in vitro.
    Gäreskog M, Wentzel P.
    J Endocrinol; 2007 Jan; 192(1):207-14. PubMed ID: 17210758
    [Abstract] [Full Text] [Related]

  • 8. Maternal diabetes in vivo and high glucose concentration in vitro increases apoptosis in rat embryos.
    Gäreskog M, Cederberg J, Eriksson UJ, Wentzel P.
    Reprod Toxicol; 2007 Jan; 23(1):63-74. PubMed ID: 17034987
    [Abstract] [Full Text] [Related]

  • 9. The role of prostanoids in the development of diabetic embryopathy.
    Wiznitzer A, Furman B, Mazor M, Reece EA.
    Semin Reprod Endocrinol; 1999 Jan; 17(2):175-81. PubMed ID: 10528368
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  • 11. Reduced SOD activity and increased neural tube defects in embryos of the sensitive but not of the resistant Cohen diabetic rats cultured under diabetic conditions.
    Weksler-Zangen S, Yaffe P, Ornoy A.
    Birth Defects Res A Clin Mol Teratol; 2003 Jun; 67(6):429-37. PubMed ID: 12962287
    [Abstract] [Full Text] [Related]

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  • 13. Protective role of arjunolic acid in response to streptozotocin-induced type-I diabetes via the mitochondrial dependent and independent pathways.
    Manna P, Sinha M, Sil PC.
    Toxicology; 2009 Mar 04; 257(1-2):53-63. PubMed ID: 19133311
    [Abstract] [Full Text] [Related]

  • 14. Safflower and olive oil dietary treatments rescue aberrant embryonic arachidonic acid and nitric oxide metabolism and prevent diabetic embryopathy in rats.
    Higa R, White V, Martínez N, Kurtz M, Capobianco E, Jawerbaum A.
    Mol Hum Reprod; 2010 Apr 04; 16(4):286-95. PubMed ID: 20051498
    [Abstract] [Full Text] [Related]

  • 15. Dietary vitamin and lipid therapy rescues aberrant signaling and apoptosis and prevents hyperglycemia-induced diabetic embryopathy in rats.
    Reece EA, Wu YK, Zhao Z, Dhanasekaran D.
    Am J Obstet Gynecol; 2006 Feb 04; 194(2):580-5. PubMed ID: 16458664
    [Abstract] [Full Text] [Related]

  • 16. Activation of oxidative stress signaling that is implicated in apoptosis with a mouse model of diabetic embryopathy.
    Yang P, Zhao Z, Reece EA.
    Am J Obstet Gynecol; 2008 Jan 04; 198(1):130.e1-7. PubMed ID: 18166327
    [Abstract] [Full Text] [Related]

  • 17. Importance of genetic predisposition and maternal environment for the occurrence of congenital malformations in offspring of diabetic rats.
    Eriksson UJ.
    Teratology; 1988 Apr 04; 37(4):365-74. PubMed ID: 3394110
    [Abstract] [Full Text] [Related]

  • 18. Occurrence of oxidative impairments, response of antioxidant defences and associated biochemical perturbations in male reproductive milieu in the Streptozotocin-diabetic rat.
    Shrilatha B, Muralidhara.
    Int J Androl; 2007 Dec 04; 30(6):508-18. PubMed ID: 17573857
    [Abstract] [Full Text] [Related]

  • 19. Glutathione metabolism and oxidative stress in neonatal rat tissues from streptozotocin-induced diabetic mothers.
    Raza H, John A.
    Diabetes Metab Res Rev; 2004 Dec 04; 20(1):72-8. PubMed ID: 14737748
    [Abstract] [Full Text] [Related]

  • 20. Signaling pathways and diabetic embryopathy.
    Dhanasekaran N, Wu YK, Reece EA.
    Semin Reprod Endocrinol; 1999 Dec 04; 17(2):167-74. PubMed ID: 10528367
    [Abstract] [Full Text] [Related]

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