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Journal Abstract Search

316 related items for PubMed ID: 16198481

  • 1. Permanent effector phenotype of neuroantigen-specific T cells acquired in the central nervous system during experimental allergic encephalomyelitis.
    Hofstetter HH, Toyka KV, Gold R.
    Neurosci Lett; 2006 Jan 02; 391(3):127-30. PubMed ID: 16198481
    [Abstract] [Full Text] [Related]

  • 2. Preferential recruitment of interferon-gamma-expressing TH17 cells in multiple sclerosis.
    Kebir H, Ifergan I, Alvarez JI, Bernard M, Poirier J, Arbour N, Duquette P, Prat A.
    Ann Neurol; 2009 Sep 02; 66(3):390-402. PubMed ID: 19810097
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  • 3. Differential expression of inflammatory cytokines parallels progression of central nervous system pathology in two clinically distinct models of multiple sclerosis.
    Begolka WS, Vanderlugt CL, Rahbe SM, Miller SD.
    J Immunol; 1998 Oct 15; 161(8):4437-46. PubMed ID: 9780223
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  • 4. Presentation of proteolipid protein epitopes and B7-1-dependent activation of encephalitogenic T cells by IFN-gamma-activated SJL/J astrocytes.
    Tan L, Gordon KB, Mueller JP, Matis LA, Miller SD.
    J Immunol; 1998 May 01; 160(9):4271-9. PubMed ID: 9574529
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  • 5. T- and B-cell nonresponsiveness to self-alphaB-crystallin in SJL mice prevents the induction of experimental allergic encephalomyelitis.
    van Stipdonk MJ, Willems AA, Verbeek R, Boog CJ, van Noort JM.
    Cell Immunol; 2000 Sep 15; 204(2):128-34. PubMed ID: 11069720
    [Abstract] [Full Text] [Related]

  • 6. Anti-IL-16 therapy reduces CD4+ T-cell infiltration and improves paralysis and histopathology of relapsing EAE.
    Skundric DS, Dai R, Zakarian VL, Bessert D, Skoff RP, Cruikshank WW, Kurjakovic Z.
    J Neurosci Res; 2005 Mar 01; 79(5):680-93. PubMed ID: 15682385
    [Abstract] [Full Text] [Related]

  • 7. An important role for the chemokine macrophage inflammatory protein-1 alpha in the pathogenesis of the T cell-mediated autoimmune disease, experimental autoimmune encephalomyelitis.
    Karpus WJ, Lukacs NW, McRae BL, Strieter RM, Kunkel SL, Miller SD.
    J Immunol; 1995 Nov 15; 155(10):5003-10. PubMed ID: 7594507
    [Abstract] [Full Text] [Related]

  • 8. CD62L is required for the priming of encephalitogenic T cells but does not play a major role in the effector phase of experimental autoimmune encephalomyelitis.
    Li O, Liu JQ, Zhang H, Zheng P, Liu Y, Bai XF.
    Scand J Immunol; 2006 Aug 15; 64(2):117-24. PubMed ID: 16867156
    [Abstract] [Full Text] [Related]

  • 9. Activation and control of pathogenic T cells in OSP/claudin-11-induced EAE in SJL/J mice are dominated by their focused recognition of a single epitopic residue (OSP58M).
    Kaushansky N, Eisenstein M, Oved JH, Ben-Nun A.
    Int Immunol; 2008 Nov 15; 20(11):1439-49. PubMed ID: 18801757
    [Abstract] [Full Text] [Related]

  • 10. Global inhibition of IL-2 and IFN-gamma secreting T cells precedes recovery from acute monophasic experimental autoimmune encephalomyelitis.
    Jensen MA, Arnason BG, Toscas A, Noronha A.
    J Autoimmun; 1996 Oct 15; 9(5):587-97. PubMed ID: 8933274
    [Abstract] [Full Text] [Related]

  • 11. [Activation of T cells in experimental autoimmune encephalomyelitis and multiple sclerosis].
    Rodríguez-Rodríguez Y, Suárez-Luis I.
    Rev Neurol; 1996 Oct 15; 36(7):649-52. PubMed ID: 12666047
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  • 13. Antigen presentation in the CNS by myeloid dendritic cells drives progression of relapsing experimental autoimmune encephalomyelitis.
    Miller SD, McMahon EJ, Schreiner B, Bailey SL.
    Ann N Y Acad Sci; 2007 Apr 15; 1103():179-91. PubMed ID: 17376826
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  • 15. Epicutaneous (EC) immunization with myelin basic protein (MBP) induces TCRalphabeta+ CD4+ CD8+ double positive suppressor cells that protect from experimental autoimmune encephalomyelitis (EAE).
    Tutaj M, Szczepanik M.
    J Autoimmun; 2007 Jun 15; 28(4):208-15. PubMed ID: 17442539
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  • 19. CD8+ T cells in inflammatory demyelinating disease.
    Weiss HA, Millward JM, Owens T.
    J Neuroimmunol; 2007 Nov 15; 191(1-2):79-85. PubMed ID: 17920696
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